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},
"code": "P23",
"name": "Врожденная пневмония",
"icd_name": "Врожденная пневмония",
"gender": 0,
"age_min": 0,
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"cause": [
4,
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"periodicity": 2,
"slug": "p23_vrozhdennaya_pnevmoniya",
"lead": "Нарушение здоровья, относящееся к группе дыхательные и сердечно-сосудистые нарушения, характерные для перинатального периода",
"description": " \r\nВрожденная пневмония — это остро протекающее инфекционно-воспалительное заболевание легких, которое проявляется в первые 72 часа жизни. ",
"etiology": "<p>Воспаление легких у новорожденных вызывают различного рода болезнетворные микроорганизмы, грибы, вирусы, простейшие. К ним относятся возбудители: стафилококк, стрептококк, пневмококк, вирус простого герпеса, цитомегаловирус, микоплазмы, хламидии, грибы рода Кандида и множество других. Пути попадания инфекции различны. </p>\r\n<p>Классифицируют бронхогенный (заражение происходит через легкие во время прохождения по родовым путям) и гематогенный (инфицирование ребенка матерью через кровь во время вынашивания плода) способ ее проникновения в детский организм. </p>\r\n<p>К группе риска относятся младенцы, период дородового развития которых происходил с такими осложнениями, как: </p>\r\n<ul>\r\n<li>внутриутробная гипоксия (недостаток получения кислорода плодом); </li>\r\n<li>застойные явления в легких при гипотрофии, то есть недоразвитие дыхательных путей; </li>\r\n<li>пороки сердца, легких; </li>\r\n<li>нарушение иммунитета, передающееся по наследству; </li>\r\n<li>недостаток или избыток витаминов в организме плода; </li>\r\n<li>хронические болезни матери и перенесенные инфекции в период созревания ребенка; </li>\r\n<li>патологические кровотечения во время беременности; </li>\r\n<li>слишком раннее отхождение околоплодных вод; </li>\r\n<li>повреждения головного мозга и верхних дыхательных путей при родах. </li>\r\n</ul>",
"pathogenesis": "<p>Возбудитель, проникнув в легкие через кровь или дыхательные пути, вызывает здесь воспалительный процесс (как правило, двусторонний). Это сопровождается отеком тканей, патологической продукцией секрета, очаговым спаданием альвеол. В результате — легкие не в состоянии качественно исполнять свою функцию, а организм новорожденных страдает от недостаточности кислорода, избытка углекислого газа, нарушения обменных процессов. При отсутствии адекватного лечения возможны опасные последствия: тяжелые дыхательные расстройства, отек легких, нарушение сердечной деятельности, полиорганная недостаточность. </p>",
"diagnostics": "<p>В общем анализе крови выявляют повышение (или снижение) числа лейкоцитов, сдвиг формулы влево, появление незрелых форм. </p>\r\n<p>Исследование мочи поможет определить кетоацидоз, поражение почек. </p>\r\n<p>На рентгенографии грудной клетки выявляют характерные воспалительные инфильтраты, усиление легочного рисунка, расширение прикорневой зоны. </p>\r\n<p>Бактериологические и серологические исследования крови (ликвора) дают возможность определить возбудителя и его чувствительность к лекарственным средствам. </p>",
"treatment": "<p>Уход. Необходимо поддерживать оптимальную температуру окружающей среды, недопустимо переохлаждение и перегревание ребенка. Правильный уход предусматривает свободное пеленание, частые перемены положения ребенка, регулярную обработку кожи и слизистых оболочек. </p>\r\n<p>Оксигенотерапия применяется для борьбы с дыхательной недостаточностью и в зависимости от ее степени тяжести осуществляется путем ингаляций, помещения ребенка в кислородную палатку, </p>\r\n<p>Вскармливание. Способ кормления (грудь, соска, зонд) и количество пищи определяется возрастом ребенка и его состоянием. </p>\r\n<p>В качестве этиотропной (направленной на возбудителя) терапии назначаются антибактериальные препараты с учетом чувствительности к ним возбудителя. </p>\r\n<p>Инфузионная терапия с целью детоксикации. </p>\r\n<p>Кислородотерапия. </p>\r\n<p>Пассивная иммунотерапия. При тяжелом течении пневмонии показаны специфические иммуноглобулины. </p>\r\n<p>Местная терапия: - СВЧ на грудную клетку 5-7 сеансов, далее электрофорез с эуфиллином, новокаином, препаратами кальция 7-10 сеансов; - отвлекающая терапия - горчичное обертывание грудной клетки; - регулярные санации трахеобронхиального дерева или бронхоскопии с промыванием бронхов антисептическими растворами. </p>",
"prevention": "<p>Профилактика врожденной пневмонии заключается в устранении предрасполагающих факторов к развитию пневмонии, соблюдении санэпидрежима в родильном доме, в отделениях больниц для новорожденных. </p>",
"clinical_picture": "<p>Клиническая картина заболевания выглядит следующим образом: </p>\r\n<ul>\r\n<li>гипотония (понижение кровяного давления); </li>\r\n<li>асфиксия (затрудненное дыхание, удушье); </li>\r\n<li>бледный с серым оттенком цвет кожи; </li>\r\n<li>рвота, частое срыгивание; </li>\r\n<li>отечность конечностей; </li>\r\n<li>наличие хрипов при прослушивании легких; </li>\r\n<li>вялость, слабость, сонливость, отказ от кормления, заторможенность; </li>\r\n<li>появление кашля и одышки; </li>\r\n<li>нередко сердечно-сосудистая недостаточность, вздутие живота; </li>\r\n<li>повышение у доношенных детей температуры тела до 40°С; </li>\r\n<li>понижение у недоношенных детей температуры тела до 35°С; </li>\r\n<li>при взятии анализа крови определяется повышенный лейкоцитоз; </li>\r\n<li>резкое снижение веса в первые дни жизни.</li>\r\n</ul>",
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},
"code": "P36",
"name": "Бактериальный сепсис новорожденного",
"icd_name": "Бактериальный сепсис новорожденного",
"gender": 0,
"age_min": 0,
"age_max": 39,
"cause": [
4,
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],
"periodicity": 2,
"slug": "p36_bakterialnyy_sepsis_novorozhdennogo",
"lead": "Нарушение здоровья, относящееся к группе инфекционные болезни, специфичные для перинатального периода",
"description": "Сепсис новорожденных – это тяжелое инфекционное заболевание, возникающее у детей первого месяца жизни. При нем образуются: гнойное воспаление одного или нескольких органов, полиорганная недостаточность (нарушение функций всех органов), циркуляция бактерий в крови (бактериемия). Сепсис — одна из основных причин смерти новорожденных.",
"etiology": "<p>Неонатальный сепсис вызывается условнопатогенной бактериальной микрофлорой. Наиболее частой причиной инфицирования в период внутриутробного развития плода и чаще в интранатальный период является S. Agalacticae — представитель стрептококков группы А. Реже сепсис вызывает E. Coli и другая кишечная грамотрицательная флора. </p>\r\n<p>Большую роль играют острые и хронические инфекционные заболевания у матери, различные акушерские вмешательства, длительный безводный период,эндометрит, наличие других гнойно-воспалительных очагов у матери (гнойный мастит и др. ).</p>\r\n<p>Предрасполагающими факторами являются внутриутробная гипоксия, внутричерепная родовая травма, незрелость новорожденного, повреждение кожи новорожденного во время акушерских операций и такие манипуляции, как интубация, катетеризация подключичных и пупочных вен и др. </p>",
"pathogenesis": "<p>Входными воротами чаще бывают раневая поверхность на коже, слизистых оболочках, пупочная рана и пупочные сосуды, а также неповрежденная кожа и слизистые оболочки верхних дыхательных путей, желудочно-кишечного тракта. При внутриутробном сепсисе очаг инфекции обычно локализуется в плацента или каком-либо органе беременной. Нередко входные ворота и первичный септический очаг определить не удается. </p>\r\n<p>Возбудитель проникает в кровеносную систему, вызывая бактериемию, токсинемию и антигенную стимуляцию продуктами жизнедеятельности и распада. В ответ на инфекцию организм отвечает системной воспалительной реакцией, проявляющейся выбросом провоспалительных цитокинов и активацией нейтрофилов, системы комплемента, свертывания крови и агрегацией тромбоцитов. Чрезмерный системный ответ, наряду с токсическим и антигенным воздействием возбудителя, приводит к повреждающему действию на органы и системы организма. Нарушается микроциркуляция, проницаемость и функции эндотелия капилляров, развивается синдром внутрисосудистого свертывания. В дальнейшем возникает полиорганная недостаточность. </p>",
"diagnostics": "<p>Клинический анализ крови: повышение количества лейкоцитов (клетки крови, отвечающие за иммунитет) либо их резкое снижение. </p>\r\n<p>Повышение уровня С-реактивного белка и прокальцитонина в крови (показатели острого воспалительного процесса в организме). </p>\r\n<p>Посев крови для выделения возбудителя и определения чувствительности к антибиотикам (какие антибиотики действуют на данного возбудителя в данном случае). </p>\r\n<p>Посев спинномозговой жидкости либо мочи, либо гнойного отделяемого с кожи (в зависимости от входных ворот инфекции (например, гноя с пуповинного остатка, если первично возникло его поражение). Взятый материал сеют на питательную среду и через определенное время анализируют, какие возбудители размножились. </p>",
"treatment": "<p>Лечение включает: </p>\r\n<ul>\r\n<li>Нахождение ребенка в кувезе (специальном аппарате для выхаживания новорожденных, в котором поддерживается оптимальная температура и влажность воздуха). </li>\r\n<li>Вскармливание предпочтительно грудное. В тяжелых случаях — парентеральтное (введение питательных растворов внутривенно). </li>\r\n<li>Антибактериальная терапия (действует непосредственно на причину болезни, то есть на бактерии) является кардинальным направлением в лечении сепсиса. Антибиотики сначала назначаются исходя из предположения о наиболее вероятных возбудителях инфекции у данного больного. После получения результатов посева крови с определением чувствительности данных возбудителей к антибиотикам возможна коррекция терапии. </li>\r\n<li>Инфузионная терапия (внутривенное введение растворов). </li>\r\n<li>Иммунокорригирующая терапия — внутривенное введение иммуноглобулинов, интерферонов. </li>\r\n<li>Переливание крови и плазмы крови (проводят для восполнения клеточного состава крови, факторов свертывания крови, плазма – для плазмофереза, то есть очищения жидкого компонента крови). </li>\r\n<li>С целью местного лечения гнойных очагов производится вскрытие фурункулов и абсцессов, перевязки с антибактериальными и ферментными препаратами; назначается УВЧ, СВЧ, электрофорез. </li>\r\n</ul>",
"prevention": "<p>Профилактика сепсиса новорожденных включает выявление и санацию инфекций мочеполовой сферы у беременной, соблюдение противоэпидемических мероприятий медперсоналом родильных домов и отделений новорожденных, тщательный гигиенических уход за новорожденным, естественное вскармливание. Вопрос о сроках вакцинации новорожденных с сепсисом против туберкулеза и против гепатита В , проводимой в первые дни жизни, решается в индивидуальном порядке.</p>",
"clinical_picture": "<p>Предвестниками сепсиса новорожденных могут выступать вялость или беспокойство, снижение аппетита, обильные частые срыгивания, плохая прибавка в весе. </p>\r\n<p>В период разгара развивается температурная реакция (повышение или снижение температуры тела), отечный синдром или обезвоживание, снижение массы тела. Характерным признаком сепсиса новорожденных является грязновато-серый (землистый) оттенок кожных покровов, желтушность, мраморность кожи, сыпь. </p>\r\n<p>Вследствие токсического поражения различных органов возникают увеличение печени и селезенки, поражение легких (учащение дыхания, синюшность, дыхательная недостаточность), поражение сердца (тахикардия или брадикардия, артериальная гипотензия), диспепсия, мочевой синдром (снижение диуреза, острая почечная недостаточность), кровотечения, надпочечниковая недостаточность. Чаще всего септицемия осложняется присоединением пневмонии (воспаления легких), которая выступает как самостоятельное интеркуррентное заболевание. </p>\r\n<p>Септикопиемия или сепсис новорожденных с гнойными очагами характеризуется возникновением на фоне интоксикации вторичных очагов отсева в мозговых оболочках, легких, костях, печени, других органах. Чаще всего септикопиемия протекает с развитием гнойного менингита, абсцедирующей пневмонии, остеомиелита; реже – с формированием абсцесса печени, гнойного артрита, поражения глаз, желудка или кишечника. </p>\r\n<p>В восстановительном периоде происходит опорожнение гнойных очагов, стихание интоксикации, постепенное восстановление нарушенных функций. </p>\r\n<p>Молниеносная форма сепсиса новорожденных протекает с развитием септического шока: стремительным ухудшением состояния ребенка, снижением температуры тела, брадикардией, кровоточивостью, отеком легких, острой почечной недостаточностью. Летальный исход при молниеносном течении сепсиса новорожденных наступает в течение 3-5 суток. </p>",
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"code": "P38",
"name": "Омфалит новорожденного с небольшим кровотечением или без него",
"icd_name": "Омфалит новорожденного с небольшим кровотечением или без него",
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"slug": "p38_omfalit_novorozhdennogo_s_nebolshim_krovotecheniem_ili_bez_nego",
"lead": "Нарушение здоровья, относящееся к группе инфекционные болезни, специфичные для перинатального периода",
"description": "Омфалит – инфекция пуповинного остатка и пупочной ранки, приводящая к воспалению кожи и подкожной клетчатки, нарушению процессов эпителизации.",
"etiology": "<p>У новорождённого развитие омфалита чаще всего происходит из-за попадания инфекции в незажившую пупочную рану. Это может произойти при перевязке пуповины или несоблюдении правил при обработке пупка мамой в период новорождённости. Также некоторые внешние факторы могут провоцировать возникновение омфалита у новорождённого: </p>\r\n<ul>\r\n<li>Наличие внутриутробной инфекции в детском организме; </li>\r\n<li>Нарушение правил гигиены при уходе за заживающей пупочной раной; </li>\r\n<li>Неправильная обработка, использование запрещённых средств, купание в загрязнённой воде; </li>\r\n<li>Применение необработанных и непродезинфицированных предметов ухода; </li>\r\n<li>Новорожденные с гипоксией и аномалиями развития, в первую очередь это аномалии самого пупка; </li>\r\n<li>Наличии пеленочного дерматита или других инфекционных заболеваний кожи у новорожденного (пиодермии, фолликулита). </li>\r\n</ul>\r\n<p>Возбудителями омфалита чаще всего оказываются стафилококки, стрептококки, примерно в 30% случаев - грамотрицательные микроорганизмы (кишечная палочка, клебсиелла и др.). </p>",
"pathogenesis": "<p>По причинам возникновения омфалит может быть первичным (при инфицировании пупочной ранки) или вторичным (в случае присоединения инфекции на фоне имеющихся врожденных аномалий - свищей). Вторичный омфалит у ребенка развивается в более поздние сроки и протекает более длительно. </p>\r\n<p>По характеру и степени воспалительных изменений в области пупка различают катаральный или простой омфалит («мокнущий пупок»), флегмонозный и гангренозный (некротический) омфалит </p>\r\n<p>Возбудитель (преимущественно стафилококк) проникает в ткани, прилежащие к пупку, через культю пуповины или через ранку после её отпадения. Инфекция может распространяться и фиксироваться в пупочных сосудах (чаще в артериях, реже в венах), вызывая продуктивное, гнойное или некротическое воспаление. Распространение воспаления приводит к развитию флегмоны в области пупка. При вовлечении в процесс пупочной вены возникает флебит (воспаление стенки сосуда), который может распространиться по воротной вене во внутрипечёночные её разветвления. Нередко при этом по ходу вен образуются гнойные очаги, иногда уже после заживления пупочной раны </p>",
"diagnostics": "<p>Диагностика заболевания не представляет трудностей и основывается на характерных местных признаках заболевания (наличие отделяемого из пупочной ранки, покраснение кожи вокруг нее). </p>\r\n<p>При развитии гнойной, флегмонозной или некротической форм возможны воспалительные изменения в общем анализе крови (повышение уровня лейкоцитов и СОЭ). </p>\r\n<p>При неэффективности назначенного лечения проводится исследование – посев отделяемого из пупка с определением чувствительности микроорганизмов к антибиотикам (в зависимости от его результатов подбирается другой антибиотик). </p>\r\n<p>Дополнительные исследования проводятся при возникновении осложнений: </p>\r\n<ul>\r\n<li>УЗИ брюшной полости (при подозрении на перитонит (воспаление брюшной полости)); </li>\r\n<li>УЗИ мягких тканей (при подозрении на флегмону брюшной стенки – гнойное воспаление подкожной клетчатки). </li>\r\n</ul>",
"treatment": "<p> Местное лечение: </p>\r\n<ul>\r\n<li>Удаление пупочного отделяемого и обработки пупочной ранки антисептическими растворами (перекисью водорода, бриллиантовым зеленым); </li>\r\n<li>в случае гнойной формы местно назначаются антибактериальные мази, а также применяется дренирование раны (установка в рану специальной трубки для оттока гноя наружу с ее помощью); </li>\r\n<li>иссечение некротизированных тканей; </li>\r\n<li>применяется также физиолечение (УФО – ультрафиолетовое облучение, УВЧ – ультравысокочастоная терапия). </li>\r\n</ul>\r\n<p>Системное лечение: </p>\r\n<ul>\r\n<li>антибиотики широкого спектра действия с учетом чувствительности флоры; </li>\r\n<li>витаминотерапия; </li>\r\n<li>дезинтоксикационная терапия; </li>\r\n<li>средства, повышающие иммунитет. </li>\r\n</ul>",
"prevention": "<p>Профилактика омфалита предполагает соблюдение асептики при обработке пуповины, ежедневном уходе за пупочной ранкой, соблюдение гигиены ухаживающим персоналом. Категорически недопустимо насильственно срывать корочки с пупочной ранки, закрывать ее повязкой или подгузником, заклеивать лейкопластырем, поскольку это провоцирует мокнутие и инфицирование. В случае покраснения пупочной ранки, появления отечности и отделяемого следует незамедлительно проконсультироваться у педиатра. <br class=\"SCXW172751371\" /> </p>",
"clinical_picture": "<p>При катаральном омфалите пупочная ранка не затягивается, из нее начинает выделяться скудный секрет серозного (прозрачного), кровянистого или серозно-гнойного характера. Ранка периодически затягивается корочками, однако после их отторжения дефект не эпителизируется. Пупочное кольцо гиперемировано (красное) и отечно. При длительном мокнутии (в течение 2-х и более недель) может происходить избыточное разрастание молодой соединительной ткани (грануляций) с формированием на дне пупочной ранки грибовидного выпячивания – фунгуса пупка, который еще более затрудняет заживление. Общее состояние новорожденного (аппетит, физиологические отправления, сон, прибавка в массе) при простой форме омфалита обычно не нарушено; иногда отмечается небольшое повышение температуры. </p>\r\n<p>Флегмонозный омфалит характеризуется распространением воспаления на окружающие ткани и обычно является продолжением «мокнущего пупка». Кожа вокруг пупка гиперемирована, подкожная клетчатка отечна и возвышается над поверхностью живота. Рисунок венозной сети на передней брюшной стенке усилен, наличие красных полос свидетельствует о присоединении воспаления лимфатических сосудов. </p>\r\n<p>Кроме мокнутия пупочной ранки, отмечается пиорея – истечение гнойного отделяемого и выделение гноя при надавливании на околопупочную область. Возможно образование на дне пупочной ямки язвочки, покрытой гнойным налетом. При флегмонозном омфалите состояние младенца ухудшается: температура тела повышается до 38°С, выражены признаки интоксикации (вялость, плохой аппетит, срыгивания, диспепсия), замедляется нарастание массы тела. У недоношенных детей локальные изменения при омфалите могут быть выражены минимально, зато на первый план обычно выходят общие проявления, молниеносно развиваются осложнения. </p>\r\n<p>Некротический омфалит встречается редко, обычно у ослабленных детей (с иммунодефицитом, гипотрофией и т.д.). При этом расплавление клетчатки распространяется в глубину. В области пупка кожа приобретает темно-багровый, синюшный оттенок. При некротическом омфалите воспаление практически всегда переходит на пупочные сосуды. В некоторых случаях могут некротизироваться все слои передней брюшной стенки с развитием контактного перитонита. Гангренозный омфалит имеет наиболее тяжелое течение: температура тела может снижаться до 36°С, ребенок истощен, заторможен, не реагирует на окружающие раздражители. </p>\r\n<p> </p>",
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"description": "Ядерная желтуха – патологическое состояние, которое может возникнуть у новорожденных в результате билирубиновой интоксикации. Билирубин появляется в базальных ганглиях и ядрах ствола мозга, что приводит к нарушениям мозговых функций. ",
"etiology": "<p>Причинами ядерной желтухи могут стать следующие факторы: </p>\r\n<ul>\r\n<li>травмы головного мозга новорожденного; </li>\r\n<li>рождение раньше срока; </li>\r\n<li>недостаток ферментов, связывающих и выводящих билирубин; </li>\r\n<li>критическое нарушение обменных процессов у новорожденного; </li>\r\n<li>подкожные кровоизлияния у младенца; </li>\r\n<li>несовместимость резус-факторов матери и ребенка; </li>\r\n<li>серьезные патологии эндокринной системы матери; </li>\r\n<li>последствия приема сильнодействующих препаратов в период беременности; </li>\r\n<li>внутриутробные заболевания инфекционного характера; </li>\r\n<li>заболевания крови (матери и ребенка). </li>\r\n</ul>",
"pathogenesis": "<p>Заболевание развивается в связи с неспособностью организма новорожденного справиться с высокой концентрацией билирубина в крови. В норме реакции распада преобладают в первые дни жизни ребенка. Именно с этим связана физиологическая желтуха новорожденных, но в норме новорожденный справляется, и симптомы исчезают в течение нескольких дней или недель. Ядерная желтуха новорожденных характеризуется чрезмерной гипербилирубинемией, вследствие чего желчный пигмент начинает проходить через гематоэнцефалический барьер, окрашивая базальные ядра, ядра мозжечка и черепно-мозговых нервов в желтушный цвет. </p>\r\n<p>Для развития ядерной желтухи новорожденных необходимо множество условий. В частности, гематоэнцефалический барьер пропускает только свободный билирубин, то есть тот, который не связан с альбуминами и не подвергнутый конъюгации в печени. Значит, одним из факторов, способствующих развитию данного состояния, является гипопротеинемия (снижение белков-переносчиков). По этой причине симптомы чаще наблюдаются у детей с отеками, обширной кефалогематомой и т. д. Гипоксическое, инфекционное или травматическое повреждение мозга, имевшее место внутриутробно или в родах, значительно повышает проницаемость гематоэнцефалического барьера. То же касается недоношенности, когда мозговые структуры к моменту рождения остаются незрелыми. </p>\r\n<p>Гемолитическая болезнь новорожденных всегда является фактором повышенного риска ядерной желтухи новорожденных, поскольку в этом случае концентрация несвязанного билирубина быстрее всего достигает предельно допустимых значений и начинает превышать их, способствуя проникновению билирубина в вещество мозга. В таких случаях состояние развивается стремительно, часто с летальным исходом или глубокой инвалидизацией. </p>",
"diagnostics": "<p>Подозрение на ядерную желтуху возникает у врача еще на этапе визуального обследования новорожденного. Для подтверждения диагноза малышу назначаются лабораторные исследования и комплексное обследование внутренних систем организма. </p>\r\n<p>В некоторых случаях возникает необходимость в дополнительной консультации с профильными специалистами. </p>\r\n<p>Диагностика ядерной желтухи включает в себя следующие процедуры: </p>\r\n<ul>\r\n<li>определение уровня билирубина - при ядерной желтухе более 256 мкмоль/л у доношенных и более 171 мкмоль/л у недоношенных детей. </li>\r\n<li>пробы Кумбса - определение наличия антител к эритроцитам; </li>\r\n<li>биохимический анализ крови и мочи; </li>\r\n<li>общий анализ крови и мочи; </li>\r\n<li>УЗИ печени и селезенки; </li>\r\n<li>УЗИ головного мозга. </li>\r\n</ul>",
"treatment": "<p>Одним из этапов лечения ядерной желтухи новорожденных является фототерапия. Доказано, что излучение видимого спектра, особенно голубого цвета, способствует переходу токсичного непрямого билирубина в его изомер, который может выводиться печенью и почками без связывания с альбуминами крови. </p>\r\n<p>Если на фоне фототерапии уровень билирубина не снижается, показано переливание плазмы крови. Перелитые вместе с плазмой белки способствуют связыванию избыточного билирубина с последующим выведением его из организма. </p>\r\n<p>При неэффективности перечисленных методов лечения или стремительном прогрессировании симптоматики проводится обменно-заменное переливание крови. </p>\r\n<p>Применяются медикаменты, которые способствуют более быстрому выведению билирубина из тела младенца (препараты с желчегонным действием), энтеросорбенты. </p>\r\n<p>По показаниям проводится противосудорожная терапия.</p>",
"prevention": "<p>Профилактика состояния возможна в первые дни жизни. Профилактическая фототерапия проводится всем новорожденным с уровнем билирубина в крови выше 200 мкмоль/л, а также всем недоношенным новорожденным, детям, перенесшим внутриутробные инфекции, гипоксию плода, родовые травмы и др.</p>",
"clinical_picture": "<p>Гипербилирубинемия в первые дни и недели жизни заметна по желтой окраске кожных покровов и склер. В данном случае этот признак выражен в значительной степени. Кожа малыша имеет ярко-желтый оттенок уже в первый день жизни, в отличие от физиологической желтухи, которая развивается спустя 5-7 дней после рождения. Состояние чаще возникает остро в течение первой недели жизни. Выделяют две стадии: асфиксическую и спастическую. Первая стадия ядерной желтухи новорожденных характеризуется появлением приступов апноэ (ребенок не дышит) разной продолжительности. Мышечный тонус снижен, отмечаются отдельные подергивания. Ребенок вялый, плохо сосет. </p>\r\n<p>Спастическая стадия ядерной желтухи новорожденных представлена выраженным гипертонусом мышц разгибателей вплоть до опистотонуса. Голова ребенка непроизвольно резко запрокидывается назад, глаза закатываются вверх (симптом «заходящего солнца»). Дыхание нарушено, отмечаются частые приступы апноэ. Могут наблюдаться тонические судороги и пронзительный крик, связанный с напряжением мышц и отчасти с болевым синдромом, сопровождающим гипертонус. В тяжелых случаях ядерной желтухи новорожденных мышечное напряжение распространяется на конечности с их резким выпрямлением и пронацией сжатых кулачков. </p>\r\n<p>В течение нескольких недель состояние ребенка улучшается. Симптомы ослабевают, судороги и гипертонус отмечаются реже. Однако к концу первого года жизни в 80% случаев развивается мышечная ригидность, судороги вновь учащаются. Прогрессируют симптомы поражения черепно-мозговых нервов, что клинически проявляется снижением зрения и слуха. Часто развивается косоглазие и тугоухость. Массивное поражение нейронов неизбежно отражается на умственном развитии. У детей, перенесших ядерную желтуху новорожденных, впоследствии нередко диагностируется синдром дефицита внимания с гиперактивностью (СДВГ), а также умственная отсталость. </p>",
"image": null,
"image_alt": null,
"standard_type": 3,
"danger": 1,
"published": 2,
"parent": null,
"block_rubric": 171,
"standards": []
}
]
}
