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},
"code": "E53",
"name": "Недостаточность других витаминов группы B",
"icd_name": "Недостаточность других витаминов группы B",
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3
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"lead": " Патологическое состояние, обусловленное дефицитом этого витамина в организме и проявляющееся нарушениями преимущественно со стороны нервной системы, сердца и кишечника.",
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"etiology": "<p>Гиповитаминоз B1 в развитых странах встречается нередко и наблюдается в основном у больных с хроническими заболеваниями желудочно-кишечного тракта, хроническим алкоголизмом, полиневритами, эндокринными заболеваниями (сахарный диабет, токсический зоб) и др. Недостаточность тиамина часто сочетается с дефицитом других витаминов группы В.</p>",
"pathogenesis": "<p>Витамин B1 хорошо растворяется в воде; кофермент тиаминдифосфат (кокарбоксилаза) играет существенную роль в углеводном обмене. Наряду с этим тиамин участвует в обмене аминокислот, нуклеотидов, ацетилхолина и холинэстеразы. При дефиците витамина В1 в тканях и крови накапливаются недоокисленные продукты: пировиноградная кислота, молочная кислота и др. При этом нарушается деятельность различных отделов нервной системы (особенно периферической нервной системы), скелетных мышц и миокарда, а также функция желудочно-кишечного тракта.</p>",
"diagnostics": "<p>Диагноз гиповитаминоза B1 подтверждается уменьшением содержания тиамина в крови (в сыворотке ниже 5 мкг/л, в эритроцитах ниже 30 мкг/л), резким повышением концентрации пировиноградной кислоты в крови (выше 0,01 г/л) и моче (выше 30 мг/сут), повышением содержания в крови молочной кислоты (выше 200 мг/л). В последнее время с этой целью используют также ряд более сложных современных методов.</p>",
"treatment": "<p>Лечение больных обычно проводится амбулаторно. Рекомендуется диета, богатая тиамином, в сочетании с препаратами тиамина внутрь (50-100 мг/сут) или парентерально (30-60 мг/сут).</p>\r\n<p>Больные с выраженными формами болезни часто нуждаются в стационарном лечении. Парентерально вводят тиамин по 1 мл и кокарбоксилазу. Одновременно назначают другие витамины группы В (рибоксин, пиридоксин, никотиновая кислота) и аскорбиновую кислоту.</p>",
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},
"code": "E52",
"name": "Недостаточность никотиновой кислоты [пеллагра]",
"icd_name": "Недостаточность никотиновой кислоты [пеллагра]",
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3
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"slug": "e52_nedostatochnost_nikotinovoy_kisloty_pellagra",
"lead": " Патологическое состояние, обусловленное дефицитом этого витамина в организме. ",
"description": "При выраженном дефиците развивается пеллагра, в менее тяжелых случаях — гиповитаминоз РР, проявляющийся нарушениями функции нервной системы, кишечника, дистрофическими изменениями кожи.",
"etiology": "<p>• Первичная — недостаток в поступающей пище, например, при преобладании в рационе кукурузы, поскольку витамин РР, хотя и содержится в ней, находится в связанном состоянии и не ассимилируется вЖКТ. Кроме того, белки кукурузы бедны триптофаном, используемого в организме для синтеза эндогенноговитамина РР<br />• Вторичная — обусловлена нарушением всасывания или усвоения витамина РР, а также увеличениемпотребности в нём <br />• Длительная <a href=\"/symptom/diareya/\" title=\"Перейти на страницу симптома Диарея\">диарея</a> <br />• Заболевания печени (чаще всего к недостаточности приводит цирроз)<br />• Алкоголизм<br />• Парентеральное питание без достаточного возмещения витаминов<br />• Злокачественные карциноидные опухоли (увеличивается потребность в триптофане)<br />• Сахарный диабет <br />• Болезнь Хартнапа</p>",
"pathogenesis": "",
"diagnostics": "<p>• Выделение N'-метилникотинамида с мочой менее 4 мг/сут<br />• Снижение содержания никотиновой кислоты<br />• Снижается содержание в крови и моче других витаминов группы В.</p>",
"treatment": "<p>В среднетяжёлых и тяжёлых случаях необходима госпитализация.<br />• Диета. Включение в рацион продуктов, богатых витамином РР (мясо, яйца) и триптофаном (молоко).<br />• Назначение никотиновой кислоты (или никотинамида) от 300 до 1000 мг/сут внутрь в начале лечения привыраженном снижении содержания витамина РР в плазме крови, затем по 300-500 мг.<br />• При заболеваниях ЖКТ никотиновую кислоту (или никоти-намид) вводят парентерально (50-100 мг в/м 2-5раз/сут или 25-100мг <br />• Одновременно назначают по 5 мг тиамина, рибофлавина и пиридоксина.</p>",
"prevention": "<p>• Питание с достаточным содержанием в пищевом рационе продуктов, богатых никотиновой кислотой<br />• При эндогенной форме — своевременная диагностика и лечение заболеваний, нарушающих всасывание иусвоение витамина РР, профилактическое введение препаратов никотиновой кислоты; дополнительноевведение витамина РР лицам с увеличенной в нём потребностью.</p>",
"clinical_picture": "<p>При недостаточности витамина РР возникают нарушения функций кожи, слизистых оболочек, ЖКТ и ЦНС. Характерно наличие трёх Д (дерматит, <a title=\"Перейти на страницу симптома Диарея\" href=\"../../../symptom/diareya/\">диарея</a>, <a title=\"Перейти на страницу симптома Деменция\" href=\"../../../symptom/demenciya/\">деменция</a>) и поражение слизистых оболочек.<br />• Стадия прегиповитаминоза — неспецифические нарушения (<a title=\"Перейти на страницу симптома Слабость\" href=\"../../../symptom/slabost/\">слабость</a>, <a title=\"Перейти на страницу симптома Утомляемость\" href=\"../../../symptom/utomlyaemost/\">утомляемость</a>, <a title=\"Перейти на страницу симптома Снижение аппетита\" href=\"../../../symptom/snizhenie_appetita/\">снижение аппетита</a> ит.д.).<br />• Стадии гипо- и авитаминоза<br />• Дерматит. Поражение кожи может быть четырёх типов. Чаще всего оно симметрично и возникает научастках кожи, подвергающихся солнечному облучению или травматизации (давлению)<br />• Остро возникают кожные проявления в виде пеллагрической эритемы с последующим образованиемпузырьков, пузырей, корочек. Часто присоединяется вторичная инфекция <br />• Поражение кожных складок — <a title=\"Перейти на страницу симптома Покраснение кожи\" href=\"../../../symptom/pokrasnenie_kozhi/\">покраснение</a>, мацерация, эрозирование и присоединение вторичнойинфекции</p>\r\n<full></full>\r\n<p><br />• Хронические гипертрофические изменения -кожа утолщена, теряет эластичность, становится складчатой. Отмечают выраженную пигментацию на открытых участках тела и в местах, подвергающихся давлению. Очаги поражения имеют чёткую границу и окружены бордюром восстанавливающегося эпителия<br />• Хронические атрофические изменения кожи с потерей эластичности и шелушением. Характерны длядлительно текущих форм пеллагры. <br />• Поражение слизистых оболочек первично затрагивает ротовую полость, хотя возможно вовлечениеслизистых оболочек влагалища и уретры<br />• Для острой недостаточности характерны <a title=\"Перейти на страницу симптома Глоссит\" href=\"../../../symptom/glossit/\">глоссит</a> и стоматит, причём язык приобретает ярко-краснуюокраску. Первоначально в процесс вовлекаются кончик и края языка, а также участки слизистой оболочки, окружающие выходное отверстие стенбнова протока<br />• Постепенно вовлекается вся слизистая оболочка, язык становится болезненным и отёчным, возникаетгиперсаливация<br />• На поздних стадиях заболевания возникает характерная картина лакового языка (ярко-красный язык сгладкой поверхностью вследствие атрофии сосочков)<br />• Возможно появление язв на слизистой оболочке дна полости рта, нижней губы, а также напротив моляров. <br />• <a title=\"Перейти на страницу симптома Диарея\" href=\"../../../symptom/diareya/\">Диарея</a> — самый характерный признак нарушения функций пищеварительной системы. Поражение ЖКТвозникает на поздних стадиях заболевания. Характеризуется появлением чувства жжения слизистой оболочкиполости рта, глотки, пищевода; возникает желудочная <a title=\"Перейти на страницу симптома Диспепсия\" href=\"../../../symptom/dispepsiya/\">диспепсия</a> в виде тошноты, реже рвоты, чтообусловлено атрофическими изменениями слизистой оболочки желудка и возникновением ахилии. Возможнадиарея, чередующийся с запорами. О тяжёлом течении свидетельствует <a title=\"Перейти на страницу симптома Диарея\" href=\"../../../symptom/diareya/\">диарея</a> с прожилками крови врезультате образования эрозий и язв слизистой оболочки кишечника.<br />• <a title=\"Перейти на страницу симптома Деменция\" href=\"../../../symptom/demenciya/\">Деменция</a> — самый яркий признак поражения ЦНС; может сопровождаться органическим психозом и/илиэнцефало-патическим синдромом. На ранних стадиях появляется <a title=\"Перейти на страницу симптома Раздражительность и озлобление\" href=\"../../../symptom/razdrazhitelnost_i_ozloblenie/\">раздражительность</a>, возможны признакиполиневрита<br />• Органический <a title=\"Перейти на страницу симптома Психоз\" href=\"../../../symptom/psihoz/\">психоз</a> проявляется галлюцинаторно-параноидной, аффективной симптоматикой, психомоторным возбуждением<br />• Энцефалопатический синдром, характеризующийся затемнением сознания, гипертонусом мышцконечностей, появлением неконтролируемых сосательного и хватательного рефлексов.<br />• На поздних стадиях нарушаются функции эндокринной системы, возникает гипопротеинемия.</p>",
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},
"code": "E51",
"name": "Недостаточность тиамина",
"icd_name": "Недостаточность тиамина",
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0
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"lead": "Заболевание протекающее с накоплением в крови и тканях пировиноградной кислоты, нарушением водного, углеводного, белкового и жирового обменов, сопровождающееся расстройством функций нервной системы, ослаблением сердечной деятельности.",
"description": "",
"etiology": "<p>Возможные причины развития:</p>\r\n<ul>\r\n<li>недостаточное поступление тиамина в организм с пищей;</li>\r\n<li>заболевания толстого и тонкого кишечника;</li>\r\n<li>аномальная активность щитовидной железы.</li>\r\n</ul>\r\n<p>В группе риска находятся алкоголики, больные малярией и ВИЧ-инфекцией.</p>",
"pathogenesis": "",
"diagnostics": "<p>Специалист назначает анализ крови и мочи. Для уточнения диагноза используют определение транскетолазной реакции.</p>",
"treatment": "<p>Как правило, терапия проводится амбулаторно. Во всех случаях больному назначается диета. Все продукты должны быть богаты витамином В1. Помимо этого, витамин вводится внутрь или принимается перорально.</p>\r\n<p>Пациентов с более тяжелыми формами болезни направляют в стационар. В этом случае микроэлемент вводят в организм парентерально по 1 мг наряду с кокарбоксилазой. Одновременно назначают другие витамины группы В.</p>",
"prevention": "<p>С целью профилактики медики рекомендуют потреблять пищу, богатую микроэлементами, особенно витамином В1.</p>",
"clinical_picture": "<p>К ранним проявлениям заболевания относят:</p>\r\n<ul>\r\n<li>неприятные ощущения в животе;</li>\r\n<li>ухудшение памяти;</li>\r\n<li>проблемы со сном;</li>\r\n<li>плохой аппетит;</li>\r\n<li>уменьшение веса;</li>\r\n<li><a href=\"/symptom/utomlyaemost/\" title=\"Перейти на страницу симптома Утомляемость\">утомляемость</a>;</li>\r\n<li><a href=\"/symptom/razdrazhitelnost_i_ozloblenie/\" title=\"Перейти на страницу симптома Раздражительность и озлобление\">раздражительность</a>.</li>\r\n</ul>\r\n<p>Необходимо своевременно обращаться к врачу при возникновении подобной симптоматики. При несвоевременном обращении к специалисту развивается тяжелый дефицит витамина В1. Впоследствии наблюдаются нарушения работы мозга, сердца, нервной системы. Во всех ситуациях изменяются процессы обмена в эритроцитах, а уровень микроэлемента в моче резко снижается.</p>\r\n<p>Наблюдается <a href=\"/symptom/onemenie_kozhi/\" title=\"Перейти на страницу симптома Онемение кожи\">покалывание</a> в пальцах нижних конечностей, жжение в стопах, которое усиливается в ночное время суток, также возникают судороги в мышцах икр и появляются боли в стопах. Во многих случаях развития болезни вибрационная чувствительность пальцев ног снижена.</p>",
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},
"code": "E50",
"name": "Недостаточность витамина A",
"icd_name": "Недостаточность витамина A",
"gender": 0,
"age_min": 0,
"age_max": 100,
"cause": [
0
],
"periodicity": 1,
"slug": "e50_nedostatochnost_vitamina_a",
"lead": "Это патологическое состояние, возникающее вследствие дефицита в организме этого витамина и проявляющееся ухудшением зрения - в первую очередь гемералопией («куриная» слепота), конъюнктивитом, ксерофтальмией, гиперкератозом кожи. ",
"description": "",
"etiology": "<p>Недостаточность витамина А может быть обусловлена нарушением абсорбции витамина в кишечнике при его хронических заболеваниях, целиакии, болезнях печени и поджелудочной железы, железодефицитной анемии, длительном употреблении минерального масла. При потреблении с пищей небольших количеств жира также снижается абсорбция витамина А.</p>\r\n<p>Причиной недостаточности витамина могут быть и чрезмерные потери его организмом. Так, экскреция витамина А увеличивается при раке, болезнях мочевыводящих путей и хронических инфекционных заболеваниях. Низкое потребление белка приводит к недостаточности белка-переносчика и способствует снижению уровня витамина в плазме.</p>",
"pathogenesis": "",
"diagnostics": "<p>При гиповитаминозе А понижено содержание в крови ретинола или каротина (ниже 200 мкг/л), нарушена темновая адаптация, развивается гемералопия. При биомикроскопии глаза на слизистой конъюнктивы могут выявляться белесые и желтоватые мелкие бляшки.</p>",
"treatment": "<p>азначают диету, богатую витамином А и провитаминами, а также препараты ретинола. Растворы ретинола применяют внутрь и в виде внутримышечных инъекций.</p>\r\n<p>Суточная лечебная доза 25 000-100 000 ME ретинола (при приеме внутрь дозу делят на 2-3 приема, больше одной инъекции в сутки обычно не делают), продолжительность курса лечения 2-4 нед. Ретинол содержится в поливитаминных препаратах.</p>",
"prevention": "<p>Профилактика гиповитаминоза А состоит в рациональном питании и назначении внутрь препаратов ретинола (5000-10 000 ME) в течение не менее 2-3 нед. До и после проведенного профилактического курса целесообразно определить содержание в сыворотке крови ретинола или каротина.</p>",
"clinical_picture": "<p>Наблюдаются сухость в полости рта и носа, <a href=\"/symptom/izmeneniya_struktury_kozhi/\" title=\"Перейти на страницу симптома Изменения структуры кожи\">сухость кожи</a>, ее <a href=\"/symptom/shelushenie_kozhi/\" title=\"Перейти на страницу симптома Шелушение кожи\">шелушение</a>, повышенное <a href=\"/symptom/vypadenie_volos/\" title=\"Перейти на страницу симптома Выпадение волос\">выпадение волос</a>. Ухудшается сумеречное зрение («куриная» <a href=\"/symptom/slepota/\" title=\"Перейти на страницу симптома Слепота \">слепота</a>). Одновременно возможны <a href=\"/symptom/svetoboyazn/\" title=\"Перейти на страницу симптома Светобоязнь\">светобоязнь</a> (<a href=\"/symptom/glaznaya_bol/\" title=\"Перейти на страницу симптома Глазная боль\">боль в глазах</a> при ярком освещении) и ксерофтальмия (сухость конъюнктивы с утратой присущего ей блеска, снижение секреции слезных желез, появление бляшек Искерского - Бито, разбросанных по склере). В наиболее тяжелых случаях воспалительные процессы в роговице могут осложняться ее некротическим размягчением (кератомаляция), что обычно заканчивается слепотой.</p>\r\n<p>Сочетание гемералопии с ксерофтальмией позволяет с высокой достоверностью думать о недостаточности витамина А.</p>",
"image": null,
"image_alt": null,
"standard_type": 1,
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"parent": null,
"block_rubric": 50,
"standards": []
},
{
"id": 6251,
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{
"id": 2779,
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{
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{
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{
"name": "резкая потеря веса"
},
{
"name": "сильное похудание"
},
{
"name": "Снижение массы тела"
},
{
"name": "похудение"
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"slug": "anormalnaya_poterya_vesa",
"lead": "Если вес снизился аномально или резко, обратитесь к врачу",
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},
{
"id": 2950,
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{
"name": "горячая на ощупь"
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{
"name": "отек на коже"
},
{
"name": "припухлость"
},
{
"name": "уплотнение"
},
{
"name": "Локализованный отек"
}
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"pathogenesis": "",
"diagnostics": "",
"treatment": "<p>При легкой и умеренной белково-энергетической недостаточности следует устранить возможные причины этого состояния. Суточный приход белков и энергии увеличивают (в соответствии с идеальным весом) для ликвидации их дефицита. Всем больным назначают поливитамины. Проводят также лечение и профилактику дефицита минеральных веществ (в том числе микроэлементов) для предупреждения угрожающих жизни гипокалиемии, гипомагниемии, гипофосфатемии и т. д. Если больной способен принимать пищу и глотать, достаточно самостоятельного питания. При снижении аппетита или в отсутствие зубов дополнительно назначают жидкие питательные смеси для самостоятельного или зондового питания.</p>\r\n<full></full>\r\n<p>При тяжелой белково-энергетической недостаточности требуется более срочное вмешательство. Лечение таких больных затруднено по нескольким причинам:</p>\r\n<ul>\r\n<li>Заболевания, вызвавшие белково-энергетическую недостаточность, протекают тяжело, их труднее лечить. Иногда азотистый баланс можно восстановить только после излечения инфекции и устранения лихорадки.</li>\r\n<li>Белково-энергетическая недостаточность сама по себе может препятствовать излечению вызвавшего ее тяжелого заболевания. В таких случаях необходимо как можно раньше начать зондовое или парентеральное питание.</li>\r\n<li>Поступление пищи через ЖКТ способствует поносу из-за атрофии слизистой и дефицита ферментов кишечника и поджелудочной железы. При этом может быть показано полное парентеральное питание.</li>\r\n<li>Следует устранить сопутствующий дефицит других компонентов пищи (витаминов, незаменимых минеральных веществ, микроэлементов).</li>\r\n</ul>\r\n<p>У взрослых восстановление питательного статуса наступает медленно и не всегда полностью; у детей выздоровление происходит в течение 3-4 мес. Во всех случаях необходимы образовательные и реабилитационные программы, а также меры психологической и социальной поддержки.</p>",
"prevention": "",
"clinical_picture": "<p>Легкая и умеренная белково-энергетическая недостаточность. У детей отсутствует прибавка веса и роста. У взрослых отмечается снижение веса, хотя при отеках или ожирении оно может быть не столь заметным. Уменьшаются толщина кожной складки над трехглавой мышцей плеча и мышечная масса в области плеча.</p>\r\n<p>В отсутствие болезней почек чувствительным показателем белковой недостаточности служит отношение суточной экскреции креатинина к росту (показатель измеряют еженедельно). Снижаются уровни альбумина, трансферрина и транстиретина (преальбумина) в крови. Уровень Т3 снижается, а реверсивного Т3 - повышается. Замедляется метаболизм. Возможны лимфопения и нарушение толерантности к глюкозе . Размеры сердца уменьшены .</p>\r\n<p>Тяжелая белково-энергетическая недостаточность. Тяжелая белково- энергетическая недостаточность сопровождается более выраженными изменениями клинических и лабораторных показателей. При физикальном исследовании обнаруживают западение межреберий, атрофию височных мышц и атрофию мышц конечностей. Подкожная клетчатка атрофирована или отсутствует. Характерны <a href=\"/symptom/apatiya/\" title=\"Перейти на страницу симптома Апатия\">апатия</a>, быстрая <a href=\"/symptom/utomlyaemost/\" title=\"Перейти на страницу симптома Утомляемость\">утомляемость</a>, чувство холода, депигментация кожи и депигментация волос, заострившиеся черты лица; кожа сухая, покрыта трещинами. В запущенных случаях образуются пролежни, кожа изъязвляется. АД снижено, также как и температура тела, пульс ослаблен. Нарушаются функции всех органов и систем.</p>",
"image": null,
"image_alt": null,
"standard_type": 1,
"danger": 52,
"published": 1,
"parent": null,
"block_rubric": 49,
"standards": []
}
]
}
