ВОзвращает список болезней. Можно фильтр/искать

search, age, gender, parent, symptoms, sort_name, sort_by, rand, branches
GET /disease/?format=api&ordering=-code&page=550
HTTP 200 OK
Allow: GET, HEAD, OPTIONS
Content-Type: application/json
Vary: Accept

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            "code": "B78",
            "name": "Стронгилоидоз",
            "icd_name": "Стронгилоидоз",
            "gender": 1,
            "age_min": 0,
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                5,
                7
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            "slug": "b78_strongiloidoz",
            "lead": "Гельминтоз, в ранней стадии которого наблюдаются преимущественные симптомы аллергического характера со стороны кожи и легких, в поздней стадии преобладают поражения органов пищеварительной системы. ",
            "description": "Инфекционное заболевание.",
            "etiology": "<p>Возбудитель стронгилоидоза — круглый гельминт Strongyloides stercoralis — кишечная угрица. Самец длиной 0,7 мм, шириной 0,04—0,06 мм. На загнутом хвостовом конце тела имеется две спикулы и рулек. Самка длиной 2,2 мм, шириной 0,03—0,7 мм. Яйца прозрачные, овальной формы, размером 0,05 х 0,03 мм. Развитие паразитов протекает со сменой свободноживущих и паразитических поколений. Половозрелые самки паразитического поколения локализуются в организме человека в толще слизистой оболочки, главным образом в либеркюновых железах двенадцатиперстной кишки, при интенсивной инвазии проникают в желудок, слизистую тонкого кишечника, панкреатические и желчные ходы. Оплодотворенные самки откладывают яйца, из которых выходят личинки, достигающие в длину 0,2—0,3 мм.</p>\r\n<full></full>\r\n<p>Личинки выделяются с фекалиями во внешнюю среду, где либо превращаются в филяриевидные личинки (гомогения), либо в свободноживущих половозрелых самцов и самок (гетерогония), способных откладывать яйца. Филяриевидные личинки способны инвазировать человека, проникая через кожные покровы или через рот с водой и пищей. При перкутантном заражении личинки совершают миграцию, подобно личинкам аскарид и анкилостомид, и достигают кишечника через дыхательные пути, глотку, пищевод. Источник инвазии — больной стронгилоидозом человек. Заражение происходит из зараженной почвы. Особенно благоприятные условия имеются в странах с жарким и влажным климатом, а также в сырых с высокой температурой шахтах и туннелях. Распространен, в основном, в странах тропического и субтропического пояса. Спорадические случаи возможны на территории России.</p>",
            "pathogenesis": "",
            "diagnostics": "<p>Паразитологический диагноз устанавливается по обнаружению личинок (реже яиц) в дуоденальном содержимом и в кале, обработанных по методу Бермана, в миграционной стадии иногда удается выявить личинки и половозрелых паразитов в мокроте. Исследованию на стронгилоидоз подлежат все больные с высокой эозинофилией крови.</p>",
            "treatment": "<p>Больные стронгилоидозом подлежат лечению в стационаре. Наиболее эффективными препаратами является минтезол (тиабендазол) и ивермектин (мектизан). Минтезол назначают из расчета 25-50 мг/кг массы тела в сутки в 3 приема 2 дня подряд. Ивермектин (мектизан) - по 200 мг/кг/сут в течение 2 дней. Могут быть использованы вермокс, альбендазол. Больным назначают десенсибилизирующие средства, при интоксикации проводят инфузионную терапию. Контроль эффективности лечения через 2 нед и затем ежемесячно в течение З меc.</p>",
            "prevention": "<p>Профилактические мероприятия при стронгилоидозе направлены на выявление и оздоровление инвазированных и на охрану окружающей среды от загрязнения фекалиями. <br /><br />Обязательному обследованию на стронгилоидоз подлежат больные, страдающие заболеваниями органов пищеварения, гепатобилиарной системы, особенно при наличии у них эозинофилии или крапивницы, т. е. при наличии характерных для инвазии клинических показаний.</p>\r\n<full></full>\r\n<p><br /><br />Учитывая особенности эпидемиологии стронгилоидоза, наличие групп профессий повышенного риска заражения, обследованию методом Бермана подлежат землекопы, шахтеры, рабочие очистных сооружений, строители дорог, парниковых хозяйств, овощных баз и др., а также лица, находящиеся в больницах, интернатах, колониях для психических больных. <br /><br />Выявленные инвазированные подлежат дегельминтизации лучше в условиях стационара или полустационара с последующим диспансерным наблюдением на протяжении одного года: с обследованием специальным методом Бермана один раз в месяц в течение 6 месяцев и один раз в квартал следующие полгода. С диспансерного учета снимают через год после трехкратного обследования с интервалом 2-3 дня при получении отрицательных результатов анализов. <br /><br />Важным и решающим методом профилактики стронгилоидоза является охрана внешней среды от фекальных загрязнении и обеззараживание загрязненной почвы. Это, прежде всего меры по санитарному благоустройству населенных мест, организация плановой очистки дворовых туалетов, запрещению удобрение почвы огородов необезвреженными фекалиями.</p>",
            "clinical_picture": "<p>В ранней миграционной стадии (до 10 суток) стронгилоидоза возникает <a title=\"Перейти на страницу симптома Высокая температура 38-42° \" href=\"../../../symptom/vysokaya_temperatura_38-42/\">лихорадка</a>, кожный <a title=\"Перейти на страницу симптома Зуд\" href=\"../../../symptom/zud/\">зуд</a>, крапивница или папулезные <a title=\"Перейти на страницу симптома Сыпь\" href=\"../../../symptom/syp/\">высыпания</a>, местные отеки, в легких появляются эозинофильные инфильтраты. В поздней фазе инвазии, когда гельминты достигают половой зрелости, иногда могут наблюдаться тяжелые, летально заканчивающиеся клинические прояв-ления. В легких случаях течения заболевания отмечаются <a title=\"Перейти на страницу симптома Тошнота\" href=\"../../../symptom/toshnota/\">тошнота</a>, тупые боли в эпигастрии. Стул, как правило, не изменен, иногда запоры или чередование запоров с легкими поносами. Иногда ведущим синдромом является ал-лергический. При выраженных проявлениях <a title=\"Перейти на страницу симптома Тошнота\" href=\"../../../symptom/toshnota/\">тошнота</a> нередко сопровождается рвотой, возникают острые боли в эпигастрии или по всему животу.</p>\r\n<full></full>\r\n<p>Периодически появляются поносы до 5—7 раз в сутки. Печень у некоторых больных увеличена и уплотнена. В периферической крови у большинства боль-ных выявляется эозинофилия до 70—80%, при длительных инвазиях возникает вторичная анемия, обычно умеренно выраженная. При тяжелых формах стронгилоидоза поносы становятся постоянными, стул приобретает гнилостный запах и содержит много остатков непереваренной пищи. Наступает обезвоживание организма, тяжелая вторичная анемия, <a title=\"Перейти на страницу симптома Кахексия\" href=\"../../../symptom/kaheksiya/\">кахексия</a>. Со стороны нервной системы отмечаются <a title=\"Перейти на страницу симптома Головная боль\" href=\"../../../symptom/golovnaya_bol/\">головная боль</a>, головокружения, повышенная умственная <a title=\"Перейти на страницу симптома Утомляемость\" href=\"../../../symptom/utomlyaemost/\">утомляемость</a>, неврастенические и психастенические синдромы. Наблюдаются симптомы дуоденита, энтероколита, реже ангиохолита и гепатита. При отсутствии лечения гельминтоз приобретает длительное хроническое течение.</p>",
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            "code": "B77",
            "name": "Аскаридоз",
            "icd_name": "Аскаридоз",
            "gender": 0,
            "age_min": 0,
            "age_max": 100,
            "cause": [
                2,
                7
            ],
            "periodicity": 1,
            "slug": "b77_askaridoz",
            "lead": "Аскаридоз - это паразитарное заболевание (гельминтоз), возбудителем которого является аскарида человеческая (Ascaris lumbricoides). Заражение человека происходит при проглатывании зрелых яиц аскарид. ",
            "description": "Аскаридоз – это заразное заболевание, которое относится к кишечным гельминтозам и вызвано соответствующим гельминтом аскаридой. Встречается аскаридоз повсеместно, заразиться им может любой человек при употреблении в пищу овощей или фруктов, загрязненных почвой с яйцами аскарид. Нет этого заболевания только на широтах с вечной мерзлотой и пустынях. ",
            "etiology": "<p>Наибольшее количество случаев заболевания аскаридозом регистрируется в тропическом и субтропическом климате. Единственным источником гельминтоза является больной человек или носитель. Заражение человека аскаридозом происходит алиментарным путем передачи яиц возбудителя с жизнеспособными личинками в них с продуктами питания, плохо очищенными от земли. Основные факторы передачи – немытые овощи (особенно зелень, салаты) или фрукты. Реже заражение происходит при употреблении загрязненной воды. Группой риска в отношении заражения аскаридозом являются люди, которые по роду своей профессиональной деятельности постоянно контактируют с землей (фермеры, работники сельскохозяйственных предприятий). Также часто заражаются дети, что связано с их низким уровнем гигиенической культуры и употреблением в пищу немытых овощей или фруктов. В случае заболевания человек непосредственно для окружающих остается не заразным, так как яйца выделяются не инвазивными. Для того чтобы они могли привести к заболеванию, необходимо их дозревание в почве в течение 15-20 дней (в зависимости от температуры и влажности). При неблагоприятных условиях, яйца могут сохранять свою жизнеспособность в почве до 10-15 лет</p>",
            "pathogenesis": "<p>Инвазионные яйца аскарид попадают через рот в желудок, затем в тонкую кишку, где в просвете развиваются личинки, проникающие в венозные сосуды слизистой оболочки, затем стоком крови в воротную вену. Начинается ранняя, миграционная, фаза патогенеза, в основе которой лежит сенсибилизация продуктами метаболизма личиночных стадий аскарид и травматизации тканей во время миграции. По системе воротной вены личинки попадают в печень, активно проникают в печеночные дольки, в печеночную вену, затем в нижнюю полую вену, в правую половину сердца, в капиллярную сеть легких. Личинки активно выходят в просвет альвеол, поднимаются в бронхиолы, бронхи, трахею и глотку. С бронхиальным секретом попадают в ротовую полость, заглатываются со слюной, возвращаются в тонкую кишку, где развиваются до половозрелых особей. Весь цикл развития аскарид от момента заражения человека до появления в фекалиях яиц новой генерации составляет 2,5–3 мес. По ходу миграции личинок образуются воспалительные клеточные инфильтраты с большим содержанием эозинофилов (в печени, легких), которые могут иметь клинические проявления или развиваться бессимптомно. В этот период нередко наблюдают увеличение количества эозинофилов в периферической крови.<br />Поздняя фаза патогенеза – кишечная (хроническая) – обусловлена паразитированием зрелых особей гельминтов в просвете тонкой кишки и характеризуется механическим повреждением слизистой оболочки, а также токсическим действием продуктов обмена гельминта на различные органы и ткани и, прежде всего, на нервную систему. Паразитирование аскарид вызывает иммуносупрессию, что проявляется неблагоприятным влиянием инвазии на течение инфекционных болезней (дизентерии, вирусных гепатитов, брюшного тифа и др.).</p>",
            "diagnostics": "<p style=\"padding-top: 5px; padding-bottom: 5px; border: 0px; outline: 0px; vertical-align: baseline;\">Основной анализ на аскаридоз – это копрологическое исследование. Однако яйца глистов в кале можно выявить лишь в кишечной фазе болезни. Фекалии собирают в чистую посуду с широким горлом и закрывают крышкой. Максимальный срок хранения материала для анализа составляет не более 18 часов. Если кал собирают вечером, его оставляют в холодильнике до утра. Для постановки более точного диагноза рекомендуют собирать испражнения несколько раз в течение 2 недель.</p><p style=\"padding-top: 5px; padding-bottom: 5px; border: 0px; outline: 0px; vertical-align: baseline;\">В начальной стадии болезни личинки аскарид выявляются в мокроте и на рентгене легких в виде инфильтратов. Также взрослые особи обнаруживаются во время оперативных вмешательств. В общем анализе крови увеличивается количество лейкоцитов и эозинофилов.</p><p style=\"padding-top: 5px; padding-bottom: 5px; border: 0px; outline: 0px; vertical-align: baseline;\">Диагностика аскаридоза может осуществляться на основании серологических методов даже в ранней стадии недуга. Используют реакцию непрямой гемагглютинации, ускорения оседания эритроцитов, латекс-агглютинации. Однако эти методики не нашли особого применения на практике.</p><p><span style=\"color: rgb(69, 67, 67); font-family: Arial; font-size: 15px; line-height: 18px; text-align: justify;\"><br></span></p>",
            "treatment": "<p style=\"padding-top: 5px; padding-bottom: 5px; border: 0px; outline: 0px; vertical-align: baseline;\">Лечение аскаридоза основывается на использовании антигельминтных средств. Дополнительно назначают белковую диету, ферментные средства, поливитамины, препараты железа при наличии анемии, десенсибилизирующие лекарства. В тяжелых случаях применяют также дезинтоксикационную терапию, противорвотные средства. При наличии осложнений показано оперативное вмешательство.</p><p style=\"padding-top: 5px; padding-bottom: 5px; border: 0px; outline: 0px; vertical-align: baseline;\">Болезнь аскаридоз эффективно лечится с использованием кислорода, который вводится через зонд в желудок натощак. Процедуру повторяют 2-3 раза.</p><p style=\"padding-top: 5px; padding-bottom: 5px; border: 0px; outline: 0px; vertical-align: baseline;\">Результат терапии оценивают через 3 недели путем исследования фекалий.</p><p style=\"padding-top: 5px; padding-bottom: 5px; border: 0px; outline: 0px; vertical-align: baseline;\">Диета при аскаридозе основывается на лечебном столе № 5. Следует ограничить употребление жиров, алкоголя, увеличить количество белковой пищи.<span style=\"color: rgb(88, 88, 88); font-family: inherit; font-size: 14px; line-height: 18px; border-style: initial; border-color: initial; border-image-source: initial; border-image-slice: initial; border-image-width: initial; border-image-outset: initial; border-image-repeat: initial; font-style: inherit;\"> </span>Питание при аскаридозе требует строгого соблюдения правил гигиены: необходимо мыть овощи и фрукты, отказаться от употребления сырой воды.</p>",
            "prevention": "<p>Профилактические мероприятия в предотвращении аскаридоза направленный в первую очередь на то, чтобы избежать инфицирование человека.<br><br>Профилактические меры включают в себя:<br></p><ol style=\"margin-top: 7px; margin-bottom: 7px; margin-left: 30px; border: 0px; font-stretch: inherit;\"><li style=\"border: 0px; font-stretch: inherit;\">Активное выявление лиц с возможным носительством паразитов. Данные мероприятия проводят преимущественно в дошкольных учреждениях, путем периодических медицинских осмотров детей с забором мазков из промежностной области для лабораторного исследования на наличие яиц гельминтов.</li><li style=\"border: 0px; font-stretch: inherit;\">Обучение детей соблюдать элементарные гигиенические правила:</li></ol><ul style=\"margin-top: 7px; margin-bottom: 7px; margin-left: 30px; border: 0px; font-stretch: inherit;\"><li style=\"border: 0px; font-stretch: inherit;\">После туалета и перед приемом пищи всегда мыть руки с мылом</li><li style=\"border: 0px; font-stretch: inherit;\">Перед тем как съесть сырой фрукт или овощ обязательно его обмыть горячей кипяченой водой</li><li style=\"border: 0px; font-stretch: inherit;\">Постригать ногти на пальцах рук по мере их вырастания</li><li style=\"border: 0px; font-stretch: inherit;\">Каждый день менять нательное белье</li></ul><ol style=\"margin-top: 7px; margin-bottom: 7px; margin-left: 30px; border: 0px; font-stretch: inherit;\"><li value=\"3\" style=\"border: 0px; font-stretch: inherit;\">Профилактический прием противогельминтных препаратов лицам, живущим в очаге распространения паразитов. </li></ol><ol style=\"margin-top: 7px; margin-bottom: 7px; margin-left: 30px; border: 0px; font-stretch: inherit;\"><li value=\"4\" style=\"border: 0px; font-stretch: inherit;\">Мероприятия, проводимые в очаге, направленные на уничтожение возможных источников распространения яиц гельминтов. <span style=\"line-height: 1.42857;\">Обработка всех поверхностей полов, столов, тумбочек и др. дезинфицирующими средствами. Примером может служить любое моющее средство, содержащее слабый раствор хлорной извести.</span></li></ol><p>Смена нательного белья каждый день. Использованное нательное, постельное белье кипятят в течение 5-10 минут, затем стирают с использованием детергентов.<br></p>",
            "clinical_picture": "<p align=\"justify\">Аскаридами заражаются около 65% всех хозяев домашних животных. Заболевание аскаридозом можно распознать не сразу после заражения, так как паразиты, превращаясь из личинок во взрослые особи, мигрируют в организме несколько месяцев, и только потом останавливаются в кишечнике. Симптомы аскаридоза проявляются сначала легким дискомфортом, затем более серьезными нарушениями.</p>\r\n<p align=\"justify\">Когда личинки проходят стадию миграции через легкие, может появляться , боль в области грудины, едва заметные перебои дыхания. Попадая далее в пищеварительный тракт, они вызывают ухудшение пищеварения, нарушение всасывания пищи. Вне зависимости от приемов пищи появляется чувство тошноты, нарушения стула, аппетит пропадает. Общее самочувствие тоже ухудшается, зараженный человек чувствует , у него внезапно проявляются симптомы простуды, а вес тела уменьшается.</p>\r\n<full></full>\r\n<p align=\"justify\">В связи с тем, что жизнедеятельность аскарид сопровождается продуцированием токсических веществ, на коже появляется , , покраснения и другие аллергические признаки. На более поздних стадиях заболевания закупоривается просвет в полости кишечника, в желчных протоках, из-за чего развивается опасная для жизни кишечная непроходимость. Нередко аскаридоз сопровождается холециститом, холангитом, обострением аппендицита.</p>\r\n<p align=\"justify\">Взрослые особи аскарид имеют довольно упругие тела и ведут подвижный образ жизнедеятельности. Это приводит к нарушенной рефлекторной функции желудка или кишечника, а также в некоторых случаях – к травмированию стенок кишечника. Со временем при отсутствии лечения возникает необратимая патология – перфорация стенок кишечника.</p>\r\n<p align=\"justify\">Из-за того, что яйца аскариды попадают в органы дыхания и затрудняют работу дыхательных путей, возможны серьезные нарушения, требующие хирургического вмешательства. У зараженного человека появляется , астматические симптомы, , а при рентгенологическом обследовании обнаруживается повышение эозинофилов. Последствиями таких нарушений могут быть острая пневмония, бронхит, разрыв капилляров в легких, сопровождающийся кровоизлиянием и воспалительными процессами.</p>\r\n<p align=\"justify\">При аскаридозе может наблюдаться и усиленное , особенно это заметно у маленьких детей по утрам, до приема пищи. Симптомы аскариды у малышей проявляются капризностью, жалобами на , астеническими симптомами, нарушениями работы нервной системы, раздражительностью. Присущими аскаридозу симптомами является \">, нарушение способности запоминать, ухудшение сна и , спазмы и , ухудшение способности мыслить и рассеянность. </p>\r\n<p align=\"justify\">В области живота или кишечника можно ощутить непонятные движения чего-то. При пальпации хорошо прощупывается клубок или округлость, образованную паразитами.</p>",
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            },
            "code": "B76",
            "name": "Анкилостомидоз",
            "icd_name": "Анкилостомидоз",
            "gender": 2,
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            "lead": "Называются гельминтозы анкилостомоз и некатороз, обладающие большим сходством биологии возбудителей, патогенеза и клиники и часто встречающиеся совместно. ",
            "description": "",
            "etiology": "<p>Возбудители анкилостомидоза — круглые гельминты семейства Ancylostomatidae: Ancylostomaduodenale (Dubini, 1843), A. braziliense (de Faria,1910), Necator americanus (Stiles, 1902). Анкилостомидозы широко распространены в тропической и субтропической зонах, в областях с жарким и влажным климатом, между 450 с. ш. и 30° ю. ш., особенно в Южной и Центральной Америке, Азии и Африке. На территории России некатороз регистрируется на черноморском побережье Краснодарского края. Развитие личинок анкилостомид во внешней среде возможно при температуре от 14 до 40° (оптимальная — 27—30°) и высокой влажности почвы, лишь небольшая часть личинок способна перезимовать в глубоких слоях почвы при незначительных понижениях температуры. В субтропических зонах почва полностью очищается от личинок, поэтому заражение носит сезонный характер, в тропических районах оно может носить круглогодичный характер, усиливаясь в сезон дождей. Источником инвазии A. duodenale и N. americanus являются больные люди, A braziliense — преимущественно собаки и кошки. Заражение анкилостомозом происходит через загрязненные руки, овощи, фрукты, зелень, а некаторозом — при ходьбе босиком, лежании на земле.</p>",
            "pathogenesis": "<p>Анкилостома и некатор локализуются в тонком кишечнике, главным образом в двенадцатиперстной и тощей кишках. Личинки анкилостом попадают в организм хозяина преимущественно через рот и развиваются в кишечнике без миграции. Личинки некатора обычно внедряются активно через кожу, проникают в кровеносные капилляры, мигрируют по большому и малому кругам кровообращения. Достигнув легких, через воздухоносные пути, гортань и глотку они попадают в пищевод и кишечник, где через 4—5 недель развива-ются во взрослых гельминтов. Паразитируя в кишечнике, гельминты питаются в основном кровью, нанося слизистой оболочке кишки мелкие ранения хитино-вым вооружением ротовой капсулы. В головном и шейном отделах имеются железы, которые выделяют особые антикоагулянты, которые обусловливают длительное <a href=\"/symptom/krovotechenie/\" title=\"Перейти на страницу симптома Кровотечение\">кровотечение</a>. Интенсивная инвазия, особенно в детском и молодом возрасте, может привести к задержке в физическом и умственном развитии, истощению и кахексии. Нередко в этих случаях инвазия заканчивается летальным исходом. Продолжительность жизни гельминтов, вероятно, 3—5 лет, возможно, дольше. Большинство анкилостомид погибает через 1—2 года после проникновения в тело человека.</p>",
            "diagnostics": "<p align=\"justify\">В различные фазы анкилостомидоза пациент может обращаться за медицинской помощью к отоларингологу, пульмонологу, гастроэнтерологу или терапевту. При диагностике анкилостомидоза учитываются клинические и эпидемиологические данные. В периферической крови отмечается гипохромная железодефицитная анемия, увеличение СОЭ, эозинофилия, гипоальбуминемия. При исследовании общего анализа мокроты обнаруживается большое количество эозинофилов. В фазу миграции на рентгенограммах легких могут выявляться воспалительные эозинофильные инфильтраты; на ЭКГ – признаки миокардиодистрофии. Данные рентгенографии пассажа бария указывают на гипотонию кишечника, нарушение моторики с явлениями застоя каловых масс.</p>\r\n<p align=\"justify\">Решающими для подтверждения анкилостомидоза являются результаты анализа кала на яйца гельминтов или исследования содержимого</p>\r\n<full></full>\r\n<p align=\"justify\">12-перстной кишки, получаемого при дуоденальном зондировании. Кроме этого, используются серологические методы диагностики (реакции гемагглютинации и латекс-агглютинации). Поскольку яйца анкилостомид имеют одинаковое строение, идентификация гельминтов (анкилостом и некаторов) возможна лишь после проведения дегельминтизации и отхождения с калом взрослых особей.</p>",
            "treatment": "<p>Этиотропная терапия анкилостомидоза проводится противогельминтными препаратами, назначаемыми врачом инфекционистом (паразитологом). Для проведения дегельминтизации используются следующие лекарственные средства: пирантел, нафтамон, левамизол, вермокс. Через 3-4 недели после антигельминтной терапии проводится контроль эффективности лечения – 3-хкратное исследование кала с интервалом в 1 месяц. Симптоматическое и патогенетическое лечение анкилостомидоза предполагает назначение препаратов железа, витамина B12, фолиевой кислоты, антигистаминных средств и др.</p>",
            "prevention": "<p>Профилактические мероприятия заключаются в соблюдении норм личной гигиены - мытье рук после посещения туалета, защите кожных покровов при контакте с землей, тщательной обработке фруктов и овощей, кипячении воды перед употреблением и т. д. Важное значение имеет санитарная обработка участков почвы и объектов внешней среды, предположительно загрязненных личинками анкилостомид. Группы повышенного риска по заболеваемости анкилостомидозом должны проходить ежегодное медицинское обследование.</p>",
            "clinical_picture": "<p>При проникновении через кожу возникает <a title=\"Перейти на страницу симптома Зуд\" href=\"../../../symptom/zud/\">зуд</a> и жжение кожи, различного рода <a title=\"Перейти на страницу симптома Сыпь\" href=\"../../../symptom/syp/\">высыпания</a> (эритематозные, папулезные, везикулезные, пустулезные), которые могут сохраняться в течение нескольких месяцев. Массивная инвазия приводит к отекам конечностей. При миграции личинок через дыхательные пути возможно развитие катаральных явлений, появляются <a title=\"Перейти на страницу симптома Одышка\" href=\"../../../symptom/odyshka/\">одышка</a>, <a title=\"Перейти на страницу симптома Хрипы\" href=\"../../../symptom/hripy/\">хрипы</a>, возможно развитие бронхита, плеврита, пневмонии. На более поздней стадии, при попадании в желудочно-кишечный тракт, развивается дуоденит с изжогой, отсутствием или усилением аппетита, иногда извращением вкуса (желанием есть, например, глину), тошнотой, рвотой, болями в подложечной и печеночной области, нередко сопровождающиеся диареей.</p>\r\n<full></full>\r\n<p><br /><br />Наиболее характерное проявление заболевания – гипохромная железодефицитная анемия. Анкилостома питается кровью. Зубцами она прикрепляется к стенкам слизистой оболочки кишки. На месте фиксации гельминта возникают <a title=\"Перейти на страницу симптома Изменения структуры кожи\" href=\"../../../symptom/izmeneniya_struktury_kozhi/\">эрозии</a> и язвы до 2 см в диаметре, при этом могут проявиться длительные кишечные кровотечения, которые и вызывают развитие железодефицитной анемии. Снижается белок крови (гипоальбуминемия). <br /><br />Поражается центральная нервная система, появляется <a title=\"Перейти на страницу симптома Вялость\" href=\"../../../symptom/vyalost/\">вялость</a>, отставание в умственном и физическом развитии.</p>",
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                        {
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                    "id": 2953,
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                            "name": "Нарушение стула"
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                            "name": "Понос"
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                    "id": 2602,
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                        {
                            "name": "Живот болит"
                        },
                        {
                            "name": "Боли в животе"
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                        {
                            "name": "Боль в левой части живота"
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                        {
                            "name": "Боль выше пупка"
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                        {
                            "name": "Боль ниже пупка"
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                        {
                            "name": "Острые боли в животе"
                        },
                        {
                            "name": "Боль в области живота "
                        },
                        {
                            "name": "Боль в области пупка"
                        },
                        {
                            "name": "Боль в правой жасти живота"
                        },
                        {
                            "name": "Пупок тянет"
                        },
                        {
                            "name": "Болит пупок"
                        },
                        {
                            "name": "Болит живот снизу"
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                        {
                            "name": "Болит живот сверху"
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                        {
                            "name": "Боль в правой части живота"
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                        {
                            "name": "Боль в животе у ребенка"
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                        {
                            "name": "У ребенка болит живот"
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                        {
                            "name": "Острая боль в животе"
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                        {
                            "name": "Резь в животе"
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                        {
                            "name": "Боль под ложечкой"
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                            "name": "Боль после еды"
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            },
            "code": "B75",
            "name": "Трихинеллез",
            "icd_name": "Трихинеллез",
            "gender": 0,
            "age_min": 0,
            "age_max": 100,
            "cause": [
                "2",
                "7"
            ],
            "periodicity": 1,
            "slug": "b75_trihinellez",
            "lead": "острая инфекция, вызываемая круглыми червями",
            "description": "<p><span id=\"docs-internal-guid-18192d37-7fff-f7aa-19fb-363e00ccff7e\"><span style=\"font-size: 7pt; font-family: Verdana; background-color: transparent; font-variant-numeric: normal; font-variant-east-asian: normal; vertical-align: baseline; white-space: pre-wrap;\">Трихинеллез ― это поражение внутренних органов и центральной нервной системы трихинеллой, круглыми червями, паразитирующими в тонкой кишке.</span></span></p>",
            "etiology": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Заражение людей происходит при употреблении в пищу сырого или недостаточно проваренного мяса пораженных трихинеллезом животных, чаще всего мяса, сала, окорока, бекона, корейки, грудинки, колбасы, изготовленных из инвазированной свинины, а также пораженного трихинеллами мяса диких животных (медведя, дикого кабана, барсука). Для того чтобы получить тяжелое заболевание, достаточно съесть 10-15 г трихинеллезного мяса.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Заболеваемость трихинеллезом обычно носит групповой характер. Заболевают члены одной семьи, лица, участвующие в одном праздничном застолье, охотничьей трапезе, использовавшие в питание мясо одного и того же трихинеллезного животного, не прошедшего предварительного ветеринарного контроля.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Домашние свиньи заражаются трихинеллами при поедании не обезвреженных мясных отходов, тушек диких животных, грызунов. Дикие животные - при хищничестве и через падаль.</span></p>\r\n<p>&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Трихинеллы отличаются большой устойчивостью к различным методам их обеззараживания. Не убивает их копчение и засолка. Остаются они живыми даже в жареном и варёном мясе. Известны случаи заражения людей при употреблении сала (с прожилками мяса), ветчины, варёной колбасы, сосисок, пельменей, шашлыков.</span></p>\r\n<p><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">&nbsp;</span></p>",
            "pathogenesis": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Люди заражаются при употреблении в пищу сырого, непрожаренного или термически недостаточно обработанного мяса зараженных животных, обычно свиней, дикого кабана или медведя. В тонкой кишке личинки покидают капсулы и проникают в подслизистый слой; через 6&ndash;8 дней они превращаются во взрослых самцов и самок. Особи женского пола приблизительно 2,2 мм длиной, а особи мужского пола приблизительно 1,2 мм длиной.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Зрелые особи женского пола продуцируют живых личинок в течение 4&ndash;6 недель, а затем умирают или выводятся. Личинки мигрируют через кровоток и лимфатическую систему, откуда попадают в поперечно-полосатую мускулатуру, где в конечном счёте и живут. Личинки полностью инкапсулируются в течение 1&ndash;2 месяцев и остаются жизнеспособными в течение нескольких лет как внутриклеточные паразиты. Мертвые личинки в конечном счете рассасываются или петрифицируются.</span></p>",
            "diagnostics": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Наиболее распространенными диагностическими методами подтверждения трихинеллеза являются следующие инструментальные и лабораторные исследования:</span></p>\r\n<ul>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Физикальное обследование. При объективном осмотре обращают на себя внимание отеки лица, параорбитальной клетчатки, гиперемия конъюнктив, макулопапулезная сыпь на теле. Пациенты с трудом совершают произвольные движения, иногда образуются мышечные контрактуры. В легких при аускультации могут выслушиваться сухие диффузные хрипы. У половины больных наблюдается гепатомегалия.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Лабораторные исследования. Одним из ведущих признаков трихинеллеза является лейкоцитоз и эозинофилия (20-60%) в общем анализе крови с максимальными значениями на 2-4 неделе болезни. Биохимические параметры отличает повышение активности АЛТ и АСТ, гипергаммаглобулинемия, снижение общего белка и электролитов гипоальбуминемия. В мокроте могут быть обнаружены кристаллы Шарко-Лейдена.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Выявление инфекционных агентов. Серологическая диагностика (ИФА) становится информативной с 3-й недели болезни, требуется проведение исследований в парных сыворотках с интервалом 10-14 дней. Диагностическим титром считается 1:100 и более. В редких случаях не ранее 9-10 суток заболевания производят микроскопию биоптатов мышечной ткани, в которых выявляются возбудители.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Инструментальные методики. Ультразвуковое исследование суставов, мягких тканей и органов брюшной полости рекомендовано с целью дифференциальной диагностики. Проведение ЭКГ, ЭХО-КС пациентам с трихинеллезом необходимо для исключения миокардита. Рентгенография органов грудной клетки назначается по показаниям, в случае поражения легких могут определяться &laquo;летучие&raquo; инфильтраты.</span></li>\r\n</ul>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p>&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>",
            "treatment": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Пациенты со среднетяжелой и тяжелой формами болезни нуждаются в стационарном лечении. При нарастании признаков дыхательной недостаточности, манифестации миокардита больные находятся в отделении интенсивной терапии. Постельный режим рекомендован до 3-4 дней отсутствия повышенной температуры тела при купированном болевом синдроме. Питание пациентов с трихинеллезом не имеет специфических ограничений, рекомендуется придерживаться щадящей диеты с достаточным количеством белка. Водный режим подлежит коррекции, связанной с отечным синдромом, требуется исключение алкоголя, газированных напитков, кофе, чая.</span></p>\r\n<p>&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">В схему терапии трихинеллеза обязательно включают этиотропные препараты (альбендазол, мебендазол, тиабендазол). Симптоматическое лечение направлено на купирование отечного (фуросемид, дихлотиазид), и аллергического (индометацин, дезлоратадин) компонентов заболевания, дезинтоксикацию (хлосоль, глюкозо-солевые растворы), десенсибилизацию (хлорид, глюконат кальция). По показаниям используются гормональные средства (преднизолон, дексаметазон), осуществляется коррекция гипопротеинемии (альбумин, свежезамороженная плазма). Исследуется возможность применения ритуксимаба как альтернативы глюкокортикостероидам.</span></p>",
            "prevention": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">В целях профилактики придерживайтесь следующих простых правил:</span></p>\r\n<ul>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Проводить визуальный осмотр мяса.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Никогда не употребляйте сырое или недоготовленное мясо.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Откажитесь от употребления мяса диких животных и птиц.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">По возможности откажитесь от употребления сала, шпика, фарша, домашних колбас и бекона.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Выбирайте мясные продукты только в проверенных магазинах, но даже в этом случае подвергайте их тщательной термической обработке.</span></li>\r\n<li>Всегда хорошо промывайте ножи и разделочные доски с посудой, которые соприкасались с сырым мясом.</li>\r\n</ul>",
            "clinical_picture": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Симптоматика болезни выглядит следующим образом:</span></p>\r\n<ul>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Сразу после заражения трихинеллезом у человека возникает небольшая боль в кишечнике, которая сопровождается диареей несколько дней. Но при приеме лекарственных препаратов, все нарушения в работе организма прекращаются и человек попросту забывает о проблеме.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Через 2-3 недели после заражения проявляются общие симптомы болезни: головная боль, в конъюнктивных мешках возможно кровоизлияние, глаза принимают покрасневший вид, возможна слабая лихорадка. Часто у людей на какое-то время возникает светобоязнь, но она быстро проходит.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">При сильных инфекциях, симптомы проявляют себя значительно ярче: возможно резкое повышение температуры до 40 градусов, появляется сильный кашель и боли в легких, нарушается слух и зрение, возможны проблемы с сердцем. Возможно развитие миокардита, шока, гепатита, пневмонии и других осложнений.</span></li>\r\n</ul>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p>&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Однако у большинства людей тяжелые признаки трихинеллеза не проявляются, все проблемы исчезают через какое-то время, особенно если человек использует для лечения различные препараты, на что обычно уходит 2-3 месяца с появления первых проблем со здоровьем. Но далее начинают возникать боли в мышцах, так как личинки трихинелл попадают в мышечные ткани и образуют небольшие капсулы. Боль может появляться при глотании пищи или воды, что связано с увеличением шейных лимфоузлов и миндалин.</span></p>",
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}