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},
"code": "A87",
"name": "Вирусный менингит",
"icd_name": "Вирусный менингит",
"gender": 0,
"age_min": 0,
"age_max": 84,
"cause": [
2,
4,
5,
7
],
"periodicity": 2,
"slug": "a87_virusnyy_meningit",
"lead": "Нарушение здоровья, относящееся к группе вирусные инфекции центральной нервной системы",
"description": "Вирусный менингит — воспаление серозных оболочек головного мозга, реже спинного, вызываемое инфекцией вирусной природы. Причиной возникновения в 80% являются энтеровируc. Вирусный менингит сопровождается серозным воспалительным процессом. Для серозного воспаления характерно образование серозного выпота, который пропитывает оболочки головного мозга, приводя к их утолщению. Отек мозговых оболочек вызывает нарушение оттока цереброспинальной жидкости и приводит к повышению внутричерепного давления.",
"etiology": "<p> В 80% случаев вирусный менингит обусловлен энтеровирусной инфекцией, реже причиной становится вирус эпидемического паротита, вирус Эпштейна-Барр (возбудитель инфекционного мононуклеоза), аренавирусы, цитомегаловирус, герпетическая инфекция, аденовирусы, ВИЧ.</p>",
"pathogenesis": "<p></p>\r\n<p>Возбудителями вирусных менингитов могут выступать различные вирусы, которые попадают в мозговые оболочки гематогенным, лимфогенным или периневральным путем. В зависимости от типа вируса вирусный менингит может возникнуть при контактном или воздушно-капельном заражении. К патогенетическим механизмам клинических проявлений менингита относятся воспаление и отек мозговых оболочек и нередко прилегающих тканей мозга, нарушение циркуляции в мозговых и оболочечных сосудах, гиперсекреция цереброспинальной жидкости и задержка ее всасывания, что приводит к развитию водянки мозга и повышению внутричерепного давления; перераздражение оболочек головного мозга, а также общее воздействие интоксикации.</p>",
"diagnostics": "<p>Заподозрить менингит позволяют характерные жалобы пациента, острое начало заболевания и наличие менингеальных симптомов. Для установления вирусного характера воспаления мозговых оболочек проводят люмбальную пункцию с исследованием цереброспинальной жидкости, ПЦР-исследования и выделение возбудителя.</p>\r\n<p>Анализ цереброспинальной жидкости при вирусном менингите показывает незначительное увеличение белка, нормальное содержание глюкозы и лейкоцитоз. В первые 1-2 сутки вирусный менингит может сопровождаться нейтрофильным лейкоцитозом ликвора, что более характерно для бактериального воспаления. Однако отсутствие возбудителя при микроскопии различно окрашенных мазков цереброспинальной жидкости свидетельствует в пользу вирусной этиологии заболевания. Для ее подтверждения необходимо повторное исследование ликвора спустя 12ч, при котором в случае вирусного менингита наблюдается уменьшение числа нейтрофилов и нарастание количества лимфоцитов.</p>\r\n<p>Анализ цереброспинальной жидкости позволяет дифференцировать вирусный менингит от других видов серозного воспаления мозговых оболочек (например, туберкулезного).</p>\r\n<p>Основным современным методом диагностики возбудителя при вирусном менингите является ПЦР- исследование цереброспинальной жидкости, позволяющее обнаружить геном вируса (ДНК или РНК).</p>\r\n<p>Серологические реакции для диагностики вирусного менингита показательны только в случае сравнения их результатов в начале заболевания и спустя 2-3 недели. Из-за большой длительности такой диагностики она может носить лишь ретроспективный характер.</p>\r\n<p>Пациентам, имеющим вирусный менингит, дополнительно проводят клинический анализ крови, биохимические пробы печени, определение электролитного состава крови, содержание в ней глюкозы, креатинина, липазы и амилазы. При атипичном течении вирусного менингита и сомнениях при его диагностике возможно проведение электромиографии, ЭЭГ, МРТ и КТ головного мозга.</p>",
"treatment": "<p>В отношении вирусного менингита в большинстве случаев проводится симптоматическая терапия. Пациенту рекомендован покой, постельный режим, нахождение в затемненном помещении. Для купирования головной боли назначают анальгетики. Но зачастую она значительно уменьшается после снижения внутричерепного давления в результате диагностической люмбальной пункции. Температура тела выше 38°C является показанием к приему жаропонижающих препаратов (парацетамол, ибупрофен и др.).</p>\r\n<p>Специфическая и неспецифическая противовирусная терапия вирусного менингита необходима у пациентов с ослабленным состоянием иммунной системы и у грудных детей. В таких случаях проводится внутривенное введение иммуноглобулина. Если вирусный менингит вызван вирусом герпеса или вирусом Эпштейн-Барр, то возможно применение ацикловира.</p>",
"prevention": "<p>Специфическая профилактика (вакцины) для вирусного менингита не разработана. В повседневности необходимо ограничивать контакт с больными респираторными инфекциями, гриппом, эпидемическим паротитом и другими вирусными инфекциями. Обеззараживание питьевой воды, мытье фруктов и овощей обеззараженной водой, запрет купания в водоемах важные пункты профилактики менингита.</p>\r\n<p>Важна ранняя диагностика и последующая изоляция больных на две недели. В очаге возникновения проводят дезинфекцию. Если заболевший ребенок посещал детский сад, заведение закрывают на двухнедельный карантин.</p>\r\n<p>Людям, контактировавшим с больными, в качестве профилактики внутривенно вводят иммуноглобулин, закапывают интерферон в течение недели.</p>",
"clinical_picture": "<p>Как правило, начало болезни острое, температура тела повышается до фебрильных цифр (40 С). Развивается головная боль, тошнота, многократная рвота. Наблюдается выраженная общая слабость, сопровождающаяся сонливостью, иногда оглушенностью. В более тяжелых случаях появляется спутанность сознания вплоть до его потери. Может возникать миалгия — боль в мышцах тела. Часто болезнь сопровождается диспепсическими расстройствами по типу гастроэнтерита: спастические боли в животе, жидкий стул. Аппетит отсутствует. Кожные покровы шеи и лица красные, горячие на ощупь. Сосуды склер инъецированы (красные глаза).</p>\r\n<p>При осмотре ротовой полости обнаруживается покраснение стенок глотки, дужек неба, слизистой оболочка мягкого неба, миндалин. При ощупывании затылочные, шейные, подчелюстные лимфатические узлы могут быть увеличены, чаще если возбудитель болезни вирус эпидемического паротита, цитомегаловирус, ВЭБ.</p>\r\n<p>Менингеальные симптомы (признаки, позволяющие поставить диагноз менингит) проявляются болью в затылке и по ходу позвоночника, ригидностью мышц затылка (неспособность согнуть голову вперед), характерной позой больного (поза «легавой» собаки) – в положении на боку, голова запрокинута, ноги поджаты к груди. Часто развиваются симптомы Кернига (неспособность разогнуть согнутую в колене ногу) и Брудзинского (верхний, средний, нижний). У детей может наблюдаться симптом Лесажа (подвешивания) – при поднимании за подмышечные ямки ребенок подтягивает ноги к животу.</p>\r\n<p>Вирусный менингит отличается относительно коротким течением. Уже на 3-5 сутки температура тела падает до нормальных цифр, хотя в некоторых случаях наблюдается вторая волна лихорадки. Весь период заболевания длится от 7 до 14 дней, в среднем около 10 дней.</p>",
"image": null,
"image_alt": null,
"standard_type": 3,
"danger": 79,
"published": 2,
"parent": null,
"block_rubric": 9,
"standards": [
5032
]
},
{
"id": 6085,
"symptoms": [
{
"id": 3234,
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{
"name": "Мышцы не расслабляются"
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{
"name": "Мышцы постоянно напряжены"
},
{
"name": "Мышцы сводит"
},
{
"name": "Постоянный тонус мышц"
},
{
"name": "Скованность шеи"
},
{
"name": "Ригидность мышц"
}
],
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"code": "A84",
"name": "Клещевой вирусный энцефалит",
"icd_name": "Клещевой вирусный энцефалит",
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"slug": "a84_kleschevoy_virusnyy_encefalit",
"lead": "Нарушение здоровья, относящееся к группе вирусные инфекции центральной нервной системы",
"description": "<p>Клещевой энцефалит – это вирусное, природно-очаговое (характерное только для определенных территорий) заболевание с преимущественным поражением центральной нервной системы.</p>",
"etiology": "<p>Вирус клещевого энцефалита — нейротропный, РНК-содержащий. Относится к семейству Flaviviridae экологической группы арбовирусов. Возбудитель способен длительно сохранять вирулентные свойства при низких температурах, но нестоек к высоким температурам (при кипячении погибает через 2-3 мин), дезинфицирующим средствам и ультрафиолетовому излучению. </p>\r\n<p>Основным резервуаром, поддерживающим существование возбудителя, являются иксодовые клещи — Ixodes persulcatus (преимущественно в азиатском регионе России) и Ixodes ricinus (преимущественно в европейском регионе). </p>\r\n<p>Кроме человека, вирус может поражать диких и домашних животных, в том числе коров и коз. Поэтому заражение может происходить как напрямую – при укусе человека клещом или случайном раздавливании насекомого при попытках его извлечь, так и при употреблении молока и молочных продуктов, не прошедших термическую обработку и полученных от больных животных.</p>",
"pathogenesis": "<p>Вирус попадает в организм человека двумя путями: через укус клеща и алиментарно. Алиментарное заражение происходит при употреблении сырого молока, а также молочных продуктов, приготовленных из молока зараженных коров и коз. При укусе клеща вирус сразу попадает в кровь. Однако при обоих способах заражения вирус проникает в нервную систему гематогенно и по периневральным пространствам. Вирус обнаруживается в ткани мозга через 2-3 дня после укуса, а максимальная концентрация его отмечается уже к 4-му дню. </p>",
"diagnostics": "<p>Серологический метод. Материалом являются парные сыворотки больного. Определение диагностического нарастания титра антител в реакциях РТГА(реакция торможения гемагглютинации) и ИФА (иммуноферментный анализ). </p>\r\n<p>Молекулярно-биологический метод. Материалом является клещ. Клеща исследуют на наличие антигена вируса клещевого энцефалита, реже с помощью ПЦР (полимеразно-цепная реакция) выявляют вирусную РНК (клеща). Для исследований на наличие антигена используют живой материал, ПЦР диагностика возможна по фрагментам клеща. </p>\r\n<p>Вирусологический метод. Выделение вируса из крови и спинномозговой жидкости путём введения материала в мозг новорожденным белым мышам. </p>",
"treatment": "<p>Лечение включает специфическую (направленную на возбудителя), патогенетическую (блокирующую механизмы развития заболевания) и симптоматическую терапию. Больному назначается строгий постельный режим. Схема специфического лечения зависит от времени, прошедшего с момента появления первых симптомов. В самом начале заболевания (первая неделя) высокую эффективность показало назначение больным противоклещевого иммуноглобулина. Его вводят в течение 3 дней. Также при ранней диагностике хорошие результаты дает применение противовирусных препаратов: рибонуклеазы, рибавирина, интерферона, экстракт побегов картофеля. </p>\r\n<p>Все эти препараты неэффективны на поздних стадиях заболевания, когда вирус уже поразил центральную нервную систему. В этом случае лечение направлено не на борьбу с возбудителем болезни, а на патологические механизмы, угрожающие жизни пациента. Для этого используют подачу кислорода через маску, ИВЛ при нарушении дыхания, мочегонные средства для снижения внутричерепного давления, препараты, увеличивающие устойчивость мозга к кислородному голоданию, нейролептики, глюкокортикоиды. </p>",
"prevention": "<p>В качестве специфической профилактики применяют вакцинацию, которая является самой надежной превентивной мерой. Обязательной вакцинации подлежат все лица, проживающие в эндемичных районах или въезжающие в них. Население в эндемичных районах составляет примерно половину всего населения России. </p>\r\n<p>Неспецифические меры профилактики сводятся к предупреждению присасывания клещей, а также к их раннему удалению. </p>\r\n<p>Избегать посещения мест обитания клещей (лесные биотопы с высокой травой, кустарником) в апреле-июле. Энцефалитные клещи нападают, цепляясь за проходящих мимо теплокровных животных и людей, как репейник. Местом для поджидания жертвы они избирают запачканые потовыми следами теплокровных травинки и веточки в тенистых травянистых местах. С учетом этого в походах следует держаться подальше от троп животных, и домашнего скота. На дорожках и широких тропах держаться середины троп,избегая контакта со свешивающейся на тропу растительностью. </p>\r\n<p>Применять репелленты, содержащие ДЭТА или перметрин. </p>\r\n<p>Следует надевать одежду с длинными рукавами и штанинами без дыр и отверстий, штанины желательно заправлять в длинные носки, рубашку в брюки. Волосы следует прятать под головной убор. Чтобы клещей было легче заметить, предпочтительно надевать светлую одежду. </p>\r\n<p>Во время пребывания в лесу рекомендуется регулярно осматривать одежду и контролировать открытые участки кожи (шею, запястья рук). При соблюдении правил носки одежды, указанной пунктом выше, клещи, не снятые с одежды, неизбежно попадают на шею, где их легко обнаружить. </p>\r\n<p>По возвращении из леса производится осмотр одежды и тела. Поскольку некоторые участки тела недоступны самоосмотру, следует прибегнуть к посторонней помощи для осмотра спины и волосистой части головы. </p>\r\n<p>Поскольку личиночные формы клещей очень мелки, их можно не заметить на одежде. Во избежание их присасывания одежду рекомендуется простирать в горячей воде. </p>\r\n<p>При обнаружении присосавшегося клеща его следует немедленно удалить. Чем раньше клещ удален, тем меньше вероятность заражения. Удалять клеща можно маникюрным пинцетом или нитью, обвязав её вокруг головы паразита. Клещ удаляется раскачивающе-выкручивающими движениями. Избегайте раздавливания клеща! Ранку можно обработать любым дезинфицирующим раствором (хлоргексидин, раствор йода, спирт, и т. п.). </p>",
"clinical_picture": "<p>Выделяют пять клинических форм клещевого энцефалита: </p>\r\n<ol>\r\n<li>лихорадочную (стертую); </li>\r\n<li>менингеальную; </li>\r\n<li>менингоэнцефалитическую; </li>\r\n<li>полиомиелитическую; </li>\r\n<li>полирадикулоневритическую. </li>\r\n</ol>\r\n<p>Лихорадочная форма клещевого энцефалита характеризуется благоприятным течением с быстрым выздоровлением. Продолжительность лихорадки - 3-5 дней. Основными клиническими признаками ее являются токсико-инфекционные проявления: головная боль, слабость, тошнота - при слабо выраженной неврологической симптоматике. Показатели ликвора без отклонений от нормы. </p>\r\n<p>Менингеальная форма является наиболее частой формой клещевого энцефалита. Больные жалуются на сильную головную боль, усиливающуюся при малейшем движении головы, головокружение, тошноту, однократную или, многократную рвоту, боли в глазах, светобоязнь. Они вялы и заторможены. Определяется ригидность мышц затылка, симптомы Кернига и Брудзинского. Менингеальные симптомы держатся на протяжении всего лихорадочного периода. Иногда они определяются и при нормальной температуре. Продолжительность лихорадки в среднем 7-14 дней. В спиномозговой жидкости изменения, характерные для вирусного мнгингита. </p>\r\n<p>Менингоэнцефалитическая форма клещевого энцефалита отличается более тяжелым течением. Нередко наблюдаются бред, галлюцинации, психомоторное возбуждение с утратой ориентировки в месте и во времени. Могут развиваться эпилептические припадки. </p>\r\n<p>При диффузном менингоэнцефалите выражены общемозговые нарушения (глубокие расстройства сознания, эпиприпадки вплоть до эпилептического статуса) и рассеянные очаги органического поражения мозга в виде псевдобульбарных расстройств (нарушение дыхания в виде урежения или учащения частоты, появление патологического дыхания), неравномерности глубоких рефлексов, асимметричных патологических рефлексов, центральных парезов мимической мускулатуры и мышц языка. </p>\r\n<p>При очаговом менингоэнцефалите быстро развиваются парезы, эпилептические припадки, реже - подкорковые и мозжечковые синдромы. В редких случаях может развиваться синдром желудочного кровотечения с кровавой рвотой. Характерны очаговые поражения черепных нервов. </p>\r\n<p>Полиомиелитическая форма клещевого энцефалита наблюдается почти у трети больных. Характеризуется продромальным периодом (1-2 дня), в течение которого отмечаются общая слабость и повышенная утомляемость. Затем выявляются периодически возникающие подергивания мышц. Внезапно может развиться слабость в какой-либо конечности или появление чувства онемения в ней (в дальнейшем в этих конечностях нередко развиваются выраженные двигательные нарушения). В последующем на фоне фебрильной лихорадки и общемозговых симптомов развиваются вялые парезы шейно-плечевой (шейно-грудной) локализации, которые могут нарастать в течение нескольких дней, а иногда до 2 нед. Наблюдаются следующие симптомы: \"свисающая на грудь голова\", \"горделивая осанка\", \"согбенная сутуловатая поза\". вялые парезы рук и спастические - ног, комбинации амиотрофий и гиперфлексии в пределах одной паретической конечности. В первые дни болезни у больных этой формой клещевого энцефалита часто резко выражен болевой синдром. Наиболее характерная локализация болей в области мышц шеи, особенно по задней поверхности, в области надплечий и рук. В конце 2-3-й недели болезни развивается атрофия пораженных мышц. </p>\r\n<p>Полирадикулоневритическая форма клещевого энцефалита характеризуется поражением периферических нервов и корешков. У больных появляются боли по ходу нервных стволов, парестезии (чувство \"ползания мурашек\", покалывание). Появляются расстройства чувствительности в дистальных отделах конечностей. Могут возникнуть вялые параличи, которые начинаются с ног и распространяются на мускулатуру туловища и рук. Восхождение может начинаться и с мышц плечевого пояса, захватывать шейные мышцы. </p>",
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}
]
}
