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"lead": "Нарушение здоровья, относящееся к группе болезни желчного пузыря, желчевыводящих путей и поджелудочной железы",
"description": "Острый панкреатит — стремительно развивающееся воспаление поджелудочной железы, обусловленное поражением органа собственными ферментами, заболевание без лечения может угрожать жизни пациента. Заболевание возникает чаще в возрасте 30—60 лет, причем у женщин в 2 раза чаще, чем у мужчин.",
"etiology": "<p>Факторы, способствующие возникновению острого панкреатита:</p>\r\n<ul>\r\n<li>злоупотребление алкоголем (в 70% случаев), вредные пищевые привычки (жирная, острая пища);</li>\r\n<li>заболевания желчевыводящих путей, в том числе желчнокаменная болезнь (в 25-30% случаев);</li>\r\n<li>вирусные (эпидемический паротит, вирус Коксаки) или бактериальные инфекции;</li>\r\n<li>травмы поджелудочной железы;</li>\r\n<li>хирургические вмешательствапо поводу других патологий поджелудочной железы и желчевыводящих путей;</li>\r\n<li>прием эстрогенов, кортикостероидов, тиазидных диуретиков, азатиоприна, других лекарственных средств с выраженным патологическим действием на поджелудочную железу (медикаментозный панкреатит);</li>\r\n<li>врожденные аномалии развития железы, генетическая предрасположенность, муковисцидоз;</li>\r\n<li>воспалительные заболевании органов пищеварения (холецистит, гепатит, гастродуоденит).</li>\r\n</ul>",
"pathogenesis": "<p>В развитии острого воспаления поджелудочной железы основным фактором выступает повреждение клеток преждевременно активированными ферментами самой железы. В нормальных условиях пищеварительные ферменты вырабатываются поджелудочной железой в неактивной форме и активизируются уже в пищеварительном тракте. Под воздействием внешних и внутренних патологических факторов механизм выработки нарушается, ферменты (фосфолипаза А и липаза) активизируются в поджелудочной железе и начинают переваривание ее собственной ткани. Вследствие этого развивается воспаление, отек ткани, поражаются сосуды паренхимы железы.</p>\r\n<p>Патологический процесс при остром панкреатите может распространяться на близлежащие ткани: забрюшинную клетчатку, сальниковую сумку, брюшину, сальник, брыжейку кишечника и связки печении ДПК. Тяжелая форма острого панкреатита способствует резкому повышению уровня различных биологически активных веществ в крови, что ведет к выраженным общим нарушениям жизнедеятельности.</p>\r\n<p> </p>",
"diagnostics": "<p>Лабораторная диагностика:</p>\r\n<p>При исследовании крови в общем анализе отмечаются признаки воспаления (ускорена СОЭ, повышено содержание лейкоцитов), в биохимическом анализе крови обнаруживают повышение активности панкреатических ферментов (амилаза, липаза), возможна гипергликемия и гипокальциемия. Также проводят определение концентрации ферментов в моче. Может отмечаться билирубинемия и повышения активности печеночных ферментов.</p>\r\n<p>При диагностировании острого панкреатита берут биохимический анализ мочи и определяют активность амилазы мочи.</p>\r\n<p>Инструментальная диагностика:</p>\r\n<p>При ультразвуковом исследовании обнаруживают снижение эхогенности паренхимы железы и появление отсутствующего в норме просвета сальниковой сумки за счёт скопления в ней выпота</p>\r\n<p>Рентгеновское исследование брюшной полости обычно малоинформативно, однако иногда позволяет выявить как косвенные признаки воспалительного процесса в поджелудочной железе</p>\r\n<p>КТ имеет преимущество перед ультразвуковым исследованием, так как обеспечивает лучшую специфическую визуализацию ткани поджелудочной железы.</p>\r\n<p>МРТ позволяет оценить уровень тканевого метаболизма, наличие ишемии, некроз панкреатоцитов. Это важно при оценке течения заболевания до развития тяжёлых состояний и осложнений.</p>\r\n<p>Ангиография позволяет установить нарушения кровообращения в поджелудочной железе и окружающих тканях и органах. Эти данные позволяют определиться с прогнозом и тактикой хирургического вмешательства.</p>\r\n<p>ЭГДС относится к дополнительным методам инструментального исследования острого панкреатита.</p>\r\n<p> </p>",
"treatment": "<p>Тактика лечения больных с острым панкреатитом определяется клинико-морфологической формой заболевания в соответствии с фазой развития воспалительного и некротического процесса и степенью тяжести состояния больного. Оптимальным видом лечения ОП в доинфекционной фазе является интенсивная консервативная терапия, а при появлений инфекционных (септических) осложнении – оперативное лечение.</p>\r\n<p>Немедикаментозное лечение: Голод в течение 2-3 суток, постоянное назогастралъное зондирование и аспирация, местная гипотермия (холод на живот). </p>\r\n<p> Для лечения нетяжёлого панкреатита достаточно проведения базисного лечебного комплекса: - анальгетики; - спазмолитики; - инфузионная</p>\r\n<p>Базисную терапию целесообразно усиливать антисекреторной и антиферментной.</p>\r\n<p>При отсутствии эффекта от проводимой базисной терапии в течение 6 часов и наличии хотя бы одного из признаков тяжёлого панкреатита следует констатировать тяжёлый панкреатит и перевести больного в отделение реанимации и интенсивной терапии и проводить лечение, соответствующее тяжёлому острому панкреатиту.</p>\r\n<p>Протокол интенсивной терапии тяжёлого панкреатита</p>\r\n<p>Основной вид лечения - интенсивная консервативная терапия. Приведенный выше базисный лечебный комплекс при тяжёлом ОП является недостаточно эффективным и должен быть дополнен специализированным лечебным комплексом. Эффективность последнего максимальна при раннем начале лечения (первые 12 часов от начала заболевания). Хирургическое вмешательство в виде лапаротомии показано лишь при развитии осложнений хирургического профиля, которые невозможно устранить эндоскопическими методами (деструктивный холецистит, желудочно-кишечное кровотечение, острая кишечная непроходимость и др.).</p>\r\n<p>Специализированное лечение. 1) Антисекреторная терапия (оптимальный срок - первые трое суток заболевания); 2) Антикоагулянты; 3) Возмещение плазмопотери (коррекция водно-электролитных, белковых потерь и др.; 4) Антиферментная, антиоксидантная и антигипоксантная терапия. 5) Детоксикация – при тяжёлом ОП показаны экстракорпоральные методы детоксикации, из которых наиболее эффективным является серийный лечебный плазмаферез. 6) Антибиотикотерапия широкого спектра действия (цефалоспорины III-IV поколений или фторхинолоны II-III поколений в сочетании с метронидазолом).</p>\r\n<p>Хирургическое лечение показано в случае выявления:</p>\r\n<p>камней в желчных протоках; скоплений жидкости в железе или вокруг нее; участков панкреатического некроза, кист, абсцессов.</p>\r\n<p>К операциям, проводимым при остром панкреатите с образованием кист или абсцессов, относятся: эндоскопическое дренирование, марсупиализация кисты, цистогастростомия и др. При образовании участков некроза, в зависимости от их размера, проводят некрэктомию или резекцию поджелудочной железы. Наличие камней является показанием к операциям на протоке поджелудочной железы.</p>\r\n<p>К хирургическому вмешательству могут прибегать и в случае сомнений в диагностики и вероятности пропустить другое хирургическое заболевание, требующее хирургического лечения.</p>",
"prevention": "<p>Первичной профилактикой панкреатита является рациональное здоровое питание, исключение алкоголя, острой, жирной обильной пищи, отказ от курения. Острый панкреатит может развиться не только у лиц, регулярно злоупотребляющих алкоголем, но и как следствие однократного приема спиртосодержащих напитков под жирную, жареную и острую закуску в больших количествах.</p>",
"clinical_picture": "<p>Характерные симптомы острого панкреатита.</p>\r\n<ul>\r\n<li>Болевой синдром. Боль может локализоваться в эпигастрии, левом подреберье, нередко носит опоясывающий характер, может отдавать под левую лопатку. Боль носит выраженный постоянный характер, в положении лежа на спине усиливается. Усиление боли происходит и после приема пищи, особенно – жирной, острой, жареной, алкоголя.</li>\r\n<li>Тошнота, рвота. Рвота может быть неукротимой, содержит желчь, не приносит облегчения. Как правило, из-за интоксикации и рвоты наступает нарушение водно-электролитного баланса, обезвоживание, которое играет важную роль в патогенезезаболевания.</li>\r\n<li>Повышение температуры тела.</li>\r\n<li>Умеренно выраженная желтушность склер. Редко – легкая желтуха кожных покровов.</li>\r\n</ul>\r\n<p>Кроме того, острый панкреатит может сопровождаться диспепсическими симптомами (вздутие живота, метеоризм, изжога), кожными проявлениями (синюшные пятна на левой боковой стенке живота, иногда с желтоватым оттенком, кровоизлияния в области пупка).</p>",
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"periodicity": 1,
"slug": "k81_holecistit",
"lead": "Нарушение здоровья, относящееся к группе болезни желчного пузыря, желчевыводящих путей и поджелудочной железы",
"description": "Острый холецистит - воспаление желчного пузыря, сопровождающееся местной и системной воспалительной реакцией. \r\n\r\nХронический холецистит - воспаление желчного пузыря, сопровождающееся нарушением его моторной функции и в некоторых случаях – образованием конкрементов (камней). ",
"etiology": "<p>Ведущая роль в развитии острого холецистита принадлежит инфекции (кишечная палочка, стафилококк, стрептококк, протей, энтерококк, брюшнотифозная палочка). Исследуют патогенетическую роль ферментов поджелудочной железы, поступающих в желчные протоки и желчный пузырь и провоцирующих острый ферментативный холецистит. </p>\r\n<p>Острый холецистит является самым частым осложнением желчнокаменной болезни (ЖКБ). Примерно в 90% наблюдений острый холецистит развивается на фоне ЖКБ, а в 10% - при отсутствии камней в желчном пузыре и поэтому обозначается как острый бескаменный холецистит (часто развивается у тяжелобольных пожилого возраста и сопровождается большим числом осложнений и более высокой летальностью). </p>\r\n<p>Развитию хронического холецистита способствуют следующие факторы: </p>\r\n<ul>\r\n<li>врожденное нарушение строения желчного пузыря, снижение его тонуса гиподинамия, опущение тех или иных органов брюшной полости, беременность (факторы, способствующие механически обусловленному застою желчи); </li>\r\n<li>нарушение диеты (переедание, ожирение, регулярное употребление острой , жирной пищи, алкоголизм); </li>\r\n<li>дискинезии желчевыводящих путей по гипотипу; </li>\r\n<li>кишечные паразиты (лямблии, амебы, аскариды, описторхи); </li>\r\n<li>желчекаменная болезнь. </li>\r\n</ul>",
"pathogenesis": "<p>Основной причиной возникновения острого воспаления и некробиотических процессов в стенке пузыря является повышение внутрипузырного давления. Как правило (90- 95%), оно возникает из-за нарушения оттока желчи из желчного пузыря в результате закупорки камнем, комочком слизи или желчным шламмом желчевыводящего протока. </p>\r\n<p>Возникновение желчной гипертензии является главным фактором в уменьшении кровоснабжения слизистой желчного пузыря. Снижение кровоснабжения стенки (особенно в пожилом и старческом возрасте при наличии склеротических изменений) способствует нарушению слизистого барьера, проникновению и росту микроорганизмов, что в свою очередь увеличивает воспалительную экссудацию в просвет органа. Очевидно, что процесс экссудации сам по себе ведет к дальнейшему увеличению внутрипузырного давления, еще большему нарушению микроциркуляции, усилению повреждения слизистой. Таким образом формируется патофизиологический порочный «замкнутый круг». </p>\r\n<p>У 10-15% больных ишемия стенки желчного пузыря, развивающаяся на фоне внутрипузырной гипертензии, приводит к некрозу, перфорации, местному или диффузному перитониту. </p>\r\n<p>Патогенез хронического холецистита связан с нарушением моторной функции желчного пузыря. Нормальная циркуляция желчи нарушается, происходит ее застой и загустевание. Позднее присоединяется инфекция. Возникает воспалительный процесс. При хроническом холецистите воспаление развивается медленее, протекает вяло. Может постепенно переходить со стенок желчного пузыря на желчевыводящие пути. При длительном течении могут формироваться спайки, деформации пузыря, сращения с находящимися рядом органами (кишечником), формирование свищей. </p>",
"diagnostics": "<p>Для уточнения диагноза холецистита используются следующие методы диагностики: </p>\r\n<p>Общий и биохимический анализ крови - позволяют выявить признаки воспаления и другие, характерные для заболеваний печени и желчного пузыря признаки. </p>\r\n<p>Ультразвуковое исследование — УЗИ органов брюшной полости, в частности желчного пузыря позволяет определить форму и размеры желчного пузыря, состояние желчных протоков, выявить утолщение и деформацию стенок желчного пузыря, камни в желчном пузыре. </p>\r\n<p>Компьютерная томография с контрастом позволяют выявить признаки воспаления желчного пузыря, закупорку желчных протоков, оценить функциональность желчного пузыря. </p>\r\n<p>Для определения моторики и формы желчного пузыря может применяться холецистография, холеграфия. Артериография выявляет утолщение стенки желчного пузыря и разрастание сосудистой сети в области двенадцатиперстной кишки и прилегающих отделах печени. </p>",
"treatment": "<p>Основой консервативного лечения являются: отказ от приема пищи и воды через рот, внутривенное введение жидкости и антибактериальная терапия, особенно при наличии признаков обезвоживания и лихорадке. </p>\r\n<p>Для купирования болевого синдрома возможно применение НПВП, спазмолитиков или опиоидных анальгетиков. Проводится дезинтоксикационная терапия. </p>\r\n<p>Флегманозный и гангренозный холецистит подлежат хирургическому лечению. Острый холецистит подлежит хирургическому лечению в ранние сроки – до 72 часов от начала заболевания. Операция выполняется по мере завершения диагностического процесса и готовности пациента в зависимости от степени тяжести его состояния. Ранняя операция холецистэктомия (удаление желчного пузыря) может быть успешно выполнена с помощью стандартной методики из лапаротомного доступа, методом минихолецистэктомии или видеолапароскопической холецистэктомии. </p>\r\n<p>Ранний дренаж желчного пузыря – холецистостомия - чрескожная или минилапаротомная – позволяют избежать выполнения операции в сложных технических условиях. Она абсолютно показана при тяжелом течении острого холецистита у соматически осложненных пациентов. </p>\r\n<p>Так же существует методика медикаментозного разрушения камней с помощью препаратов солей урсодезоксихолиевой и хенодезоксихолиевой кислот </p>\r\n<p>При хроническом калькулёзном холецистите показано хирургическое удаление желчного пузыря – источника образования конкрементов. В отличие от лечения острого калькулёзного холецистита, операция по удалению желчного пузыря (холецистотомия лапароскопическая или открытая) при хроническом холецистите не является экстренной мерой, назначается планово. Применяются те же хирургические методики, как и при остром холецистите – лапароскопическая операция удаления желчного пузыря, холецистэктомия из минидоступа. Для ослабленных и пожилых пациентов – чрескожная холецистостомия для формирования альтернативного пути оттока желчи. При хроническом холецистите в случае противопоказаний к оперативному вмешательству можно попробовать методику нехирургического дробления камней с помощью экстракорпоральной ударно-волновой цистолитотрипсии, однако стоит помнить, что разрушение камней не ведет к излечению и довольно часто происходит их повторное формирование. </p>",
"prevention": "<p>Первичной профилактикой холецистита является соблюдение здорового образа жизни, ограничение в приеме алкоголя, отсутствие вредных пищевых привычек (переедание, пристрастие к острой и жирной пище), физически активная жизнь. При наличии врожденных аномалий внутренних органов – своевременное выявление и коррекция застойных явлений в желчном пузыре. Избегание стрессов и своевременное лечение желчекаменной болезни и паразитарных заражений кишечника и печени. </p>\r\n<p>Для профилактики обострений больным необходимо строго следовать диете и принципам дробного питания, избегать гиподинамии, стрессов и переохлаждения, тяжелой физической нагрузки. Больные хроническим холециститом состоят на диспансерном учете и дважды в год должны проходить обследование. Им показано регулярное санаторно-курортное лечение. </p>",
"clinical_picture": "<p>Катаральный холецистит проявляется постоянными сильными болями в правом подреберье, отдающими в правую половину тела — лопатку, плечо, правую поверхность шеи. В начале заболевания боли могут быть приступообразными. Часто появляется рвота содержимым желудка. В рвотных массах почти всегда обнаруживается желчь. Температура — субфебрильная, пульс — в норме. </p>\r\n<p>При флегмонозном холецистите боли интенсивнее, усиливаются при смене положения тела, дыхании, кашле, натуживании. Рвота — многократна, температура — высокая (38-39°С). Язык сухой, обложенный. Пульс учащается до 100 более ударов за минуту. Живот вздут, болезнен при выщупывании. </p>\r\n<p>При гангренозном холецистите на первое место выходит общая симптоматика. Боли несколько стихают, однако это — лишь кажущееся благополучие. Причина уменьшения болевых ощущений — гибель нервных рецепторов под действием инфекции, проникающей во всю толщу стенки желчного пузыря. Состояние больного резко утяжеляется, он становится заторможенным, вялым. Нарастают явления общего перитонита — воспаления брюшины, возникающего вследствие распространения инфекции из желчного пузыря в брюшную полость. Температура высокая, отмечается выраженная тахикардия, живот резко вздут, болезнен при пальпации во всех отделах, напряжен. Лицо осунувшееся, глаза запавшие, наблюдается желтушность кожных покровов. </p>\r\n<p>Хронический холецистит развивается в течение продолжительного времени, периоды ремиссии чередуются с обострениями. Основным симптомом является болевой. Боль умеренно выражена, локализуется в правом подреберье, имеет тупой ноющий характер, может продолжаться до нескольких дней (недель). Иррадиация может происходить в спину под правую лопатку, правую половину поясничной области, правое плечо. Для хронического холецистита характерно усиления болевого симптома после приема острой или жирной пищи, газированных напитков, алкоголя. Обострению хронического холецистита чаще всего предшествуют подобные нарушения в диете, а так же переохлаждение и стрессы. </p>\r\n<p>Болевой симптом при калькулёзном хроническом холецистите может протекать по типу желчной колики (боль острая, сильная, схваткообразная). Помимо болевого симптома у больных нередко отмечают тошноту (вплоть до рвоты), отрыжку, привкус гречи во рту. В период обострения может отмечаться повышение температуры тела до субфебрильных значений. </p>",
"image": null,
"image_alt": null,
"standard_type": 3,
"danger": 1,
"published": 2,
"parent": null,
"block_rubric": 118,
"standards": []
}
]
}