Disease List – Django REST framework

ВОзвращает список болезней. Можно фильтр/искать

search, age, gender, parent, symptoms, sort_name, sort_by, rand, branches

GET /disease/?format=api&ordering=-published&page=555

HTTP 200 OK
Allow: GET, HEAD, OPTIONS
Content-Type: application/json
Vary: Accept

{
    "count": 11794,
    "next": "http://api.symptomd.ru/disease/?format=api&ordering=-published&page=556",
    "previous": "http://api.symptomd.ru/disease/?format=api&ordering=-published&page=554",
    "results": [
        {
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            "symptoms": [],
            "alternative_names": [],
            "complications": [],
            "medicine_branches": [],
            "who": [
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                    "danger": 0.0,
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                    "diseased_1": 1424,
                    "diseased_1_4": 7296,
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                    "diseased_10_14": 356,
                    "diseased_15_19": 1424,
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                    "diseased_25_29": 1780,
                    "diseased_30_34": 2669,
                    "diseased_35_39": 1958,
                    "diseased_40_44": 2491,
                    "diseased_45_49": 6229,
                    "diseased_50_54": 7652,
                    "diseased_55_59": 15839,
                    "diseased_60_64": 15305,
                    "diseased_65_69": 19754,
                    "diseased_70_74": 31499,
                    "diseased_75_79": 37906,
                    "diseased_80_84": 41643,
                    "diseased_85_89": 17440,
                    "diseased_90_94": 5339,
                    "diseased_95": 0,
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                },
                {
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                    "deaths": 0,
                    "danger": 0.0,
                    "mortality": 0.0,
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                    "hospital_days": 69,
                    "diseased_1": 2076,
                    "diseased_1_4": 8872,
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                    "diseased_10_14": 2265,
                    "diseased_15_19": 3398,
                    "diseased_20_24": 5852,
                    "diseased_25_29": 5097,
                    "diseased_30_34": 7739,
                    "diseased_35_39": 8306,
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                    "diseased_45_49": 7928,
                    "diseased_50_54": 13780,
                    "diseased_55_59": 24351,
                    "diseased_60_64": 33600,
                    "diseased_65_69": 33411,
                    "diseased_70_74": 39074,
                    "diseased_75_79": 33223,
                    "diseased_80_84": 28692,
                    "diseased_85_89": 12459,
                    "diseased_90_94": 2265,
                    "diseased_95": 189,
                    "disease": 6453
                }
            ],
            "thumbnail_100x100": null,
            "thumbnail_300x300": null,
            "thumbnail_600x600": null,
            "symptom_count": 0,
            "symptom_matched": 0,
            "relevance": 1.39932474630173,
            "who_popularity": 509821.0,
            "group_population": 364333584,
            "who_danger": 0.0,
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            },
            "code": "H27.1",
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            "treatment": "",
            "prevention": "",
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            "image_alt": null,
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            },
            "code": "H27.8",
            "name": "Другие уточненные болезни хрусталика",
            "icd_name": "Другие уточненные болезни хрусталика",
            "gender": 0,
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            "slug": "h27.8_drugie_utochnennye_bolezni_hrustalika",
            "lead": "",
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            "etiology": "",
            "pathogenesis": "",
            "diagnostics": "",
            "treatment": "",
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                    "lead": "Нарушения зрения  — снижение способности видеть до такой степени, что вызывает проблемы, не устраняемые обычными способами, такими как очки или лекарства.[1][2][3]\r\n\r\nЭто может быть из-за болезни, травмы, врождённых или дегенеративных заболеваний. Эта потеря зрения, как правило, определяется как:\r\n\r\nОстрота зрения с коррекцией менее 20/60, или значительный дефект центрального поля,\r\nЗначительный одноимённый или разноимённый двусторонний визуальный дефект периферического поля, включающий дефект поля или общее сжатия или сужение поля, или\r\nПониженный пик контрастной чувствительности с любым из указанных выше условий.[1][2][3][4]\r\nГлазные нарушения, которые могут привести к нарушениям зрения могут включать в себя дегенерацию сетчатки, альбинизм, катаракты, глаукомы, мышечные проблемы, которые приводят к нарушениям зрения, расстройства роговицы, диабетическую ретинопатию, врождённые пороки и инфекции. Нарушения зрения также могут быть вызваны расстройствами мозга и нервной системы, в этом случае их, как правило, называют кортикальными нарушениями зрения (CVI). Путеводитель оценки постоянного ухудшения американской медицинской ассоциации попытался представить «стандартизированный, объективный подход к оценке медицинских нарушений». Глава Визуальная система «устанавливает критерии для оценки постоянного ухудшения зрительной системы, как это влияет на способность человека выполнять действия в повседневной жизни».[5] Путеводитель оценил, что потеря одного глаза составляет 25 % нарушений зрительной системы и 24 % от полного обесценивания человека;[5][6]. Полная потеря зрения на оба глаза, как считается, является 100 % нарушением зрения и 85 % от полного обесценивания человека[5]\r\n\r\nВизуальные нарушения наносят значительный экономический удар даже в развитых странах.[7] Значительную часть глобального нарушения зрения можно предупредить.",
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            "code": "H28.0*",
            "name": "Диабетическая катаракта (E10-E14+ с общим четвертым знаком .3)",
            "icd_name": "Диабетическая катаракта (E10-E14+ с общим четвертым знаком .3)",
            "gender": 0,
            "age_min": 10,
            "age_max": 100,
            "cause": [
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            "periodicity": 1,
            "slug": "h28.0*_diabeticheskaya_katarakta_e10-e14_s_obschim_chetvertym_znakom_3",
            "lead": "заболевание, возникающее на фоне сахарного диабета, вызванное нарушением метаболизма или сочетанием с возрастными изменениями зрительного аппарата",
            "description": "",
            "etiology": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Ведущий этиологический фактор диабетической катаракты &ndash; повышение уровня глюкозы в крови при СД 1 и 2 типа. При инсулинозависимом диабете, клиническую картину болезни выявляют в более молодом возрасте, это обусловлено хронической гипергликемией на фоне абсолютной или относительной недостаточности инсулина. При инсулиннезависимом диабете нарушается взаимодействие клеток с гормоном, подобные изменения более характерны для пациентов средней возрастной группы.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Риск развития катаракты напрямую зависит от диабетического &laquo;стажа&raquo;. Чем дольше пациент страдает СД, тем выше вероятность формирования помутнений хрусталика. Резкий переход с пероральных таблетированных форм гипогликемических препаратов на инсулин для подкожного введения может стать триггером, который запускает цепь патологических изменений. Необходимо отметить, что при своевременной адекватной компенсации дисфункции углеводного обмена подобных расстройств удается избежать.</span></p>\r\n<p>&nbsp;</p>",
            "pathogenesis": "<p>&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Прозрачный хрусталик в сложной оптической системе глаза выполняет функции преломляющей свет линзы, которая фокусирует изображение (в перевернутом виде) на сетчатке; оттуда оно передается в аналитические и интерпретирующие зоны мозга, где и воссоздается целостный зрительный образ. При сахарном диабете состав крови отличается нестабильностью и периодическими повышениями концентрации глюкозы; это неизбежно сказывается на &laquo;питании&raquo; глазных структур: скапливаются вредоносные соединения, которые лишают хрусталик естественной прозрачности и эластичности, т.е. запускают процесс формирования катаракты. Как следствие &mdash; характерные нарушения зрения.</span></p>",
            "diagnostics": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Обследование пациента, страдающего диабетической катарактой, должно быть комплексным. Помимо переднего сегмента глаз проводят детальный осмотр сетчатки, поскольку при СД прослеживается высокий риск сопутствующего поражения внутренней оболочки глаза. Обязательно выполнение таких лабораторных исследований, как анализ крови на гликированный гемоглобин, глюкозотолерантный тест и определение сахара крови. В большинстве случаев консультация офтальмолога включает следующие инструментальные диагностические процедуры:</span></p>\r\n<ul>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Исследование зрительной функции. При проведении визометрии выявляется снижение остроты зрения вдаль. При выполнении работы на расстоянии 30-40 см дискомфорт отсутствует. С возрастом прогрессируют пресбиопические изменения, в тоже время болезнь приводит к кратковременному улучшению зрения вблизи.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Осмотр глаза. В ходе биомикроскопии визуализируются точечные и хлопьевидные помутнения, расположенные в поверхностных отделах передней и задней капсулы. Реже в проходящем свете можно обнаружить небольшие дефекты, которые локализируются глубоко в строме.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Ретиноскопия. Прогрессирование заболевания становится причиной формирования миопического типа клинической рефракции. Ретиноскопию можно заменить скиаскопией с применением скиаскопических линеек. Дополнительно проводится компьютерная рефрактометрия.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Осмотр глазного дна. Офтальмоскопия &ndash; это рутинная процедура в практической офтальмологии. Исследование выполняется с целью исключения диабетической ретинопатии и поражения зрительного нерва. При тотальной катаракте выполнение офтальмоскопии резко затруднено из-за снижения прозрачности оптических сред.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Ультразвуковое исследование. УЗИ глаза (А-сканирование) позволяет измерить переднезадний размер глазного яблока (ПЗР), чтобы определить, чем обусловлена миопизация. При диабетической катаракте ПЗР соответствует норме, при выраженных помутнениях хрусталик увеличен в размере.</span></li>\r\n</ul>\r\n<p>&nbsp;</p>\r\n<p><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">&nbsp;</span></p>",
            "treatment": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">При выявлении начальных изменений целью лечения является достижение толерантных значений глюкозы крови и компенсация СД. Нормализация углеводного обмена возможна при соблюдении диеты, применении пероральных антигипергликемических препаратов и инъекций инсулина. Своевременное назначение консервативной терапии дает возможность положительно повлиять на динамику развития катаракты, обеспечить ее частичную или полную резорбцию. При зрелой стадии нормализация уровня сахара крови не менее важна, однако добиться даже частичного восстановления прозрачности хрусталика при грубых помутнениях невозможно.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Для профилактики прогрессирования патологии назначают инстилляции рибофлавина, аскорбиновой и никотиновой кислот. При незрелой форме используют препараты на основе цитохрома-С, комбинации неорганических солей и витаминов. Доказана эффективность внедрения в офтальмологическую практику лекарственных средств, активным компонентом, которых является синтетическое вещество, предупреждающее окисление сульфгидрильных радикалов растворимых белков, входящих в состав гексагональных клеток.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Хирургическое лечение предусматривает удаление хрусталика микрохирургическим путем (ультразвуковая факоэмульсификация) с последующей имплантацией интраокулярной линзы (ИОЛ) в капсулу. Оперативное вмешательство выполняется при выраженной зрительной дисфункции. Катаракту на начальной стадии целесообразно удалять в том случае, если ее наличие затрудняет проведение витреоретинальной операции или лазерной коагуляции внутренней оболочки при диабетической ретинопатии.</span></p>",
            "prevention": "<p><span id=\"docs-internal-guid-d1a17a1a-7fff-6233-f445-4947f1652c64\"><span style=\"font-size: 7pt; font-family: Verdana; background-color: transparent; font-variant-numeric: normal; font-variant-east-asian: normal; vertical-align: baseline; white-space: pre-wrap;\">Специфическая профилактика не разработана. Неспецифические превентивные меры сводятся к контролю уровня глюкозы крови, соблюдению специальной диеты, профилактическому осмотру у офтальмолога 1 раз в год с обязательным выполнением биомикроскопии и офтальмоскопии.</span></span></p>",
            "clinical_picture": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Клиническая картина зависит от того, как давно имеется сахарный диабет у пациента, насколько он компенсирован. На начальных стадиях развития диабета зрение может быть сохранено в полном объеме, либо же отмечается незначительное ухудшение показателей. Некоторые пациенты замечают улучшение зрения при рассмотрении предметов на близком расстоянии.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">С того момента, как объем пораженных хрусталиковых масс растет, больные предъявляют жалобы на точки, мушки, ощущение пыли перед глазами. Повышается чувствительность к свету, может формироваться светобоязнь. Если посмотреть на источник яркого света, то перед глазами начинают мерцать радужные круги. Прогрессируют нарушения четкости зрения из-за наличия многочисленных помех, особенно тревожным симптомом является появление желтоватого оттенка видимого изображения.</span></p>\r\n<p>&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Катаракта на стадии зрелой имеет выраженные жалобы. Человек из-за выраженного снижения остроты зрения постепенно утрачивают навык быстро ориентироваться в знакомом пространстве, в тяжелых случаях диабетики не могут самостоятельно себя обслуживать. Визуально отмечается изменение цвета зрачка: невооруженным глазом видно, что линза помутнела и стала белесоватого цвета. Очки или контактные линзы не помогают вернуть утраченное зрение, не компенсируют изменения. Катаракта неумолимо прогрессирует и может со временем привести к полной слепоте</span></p>",
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                    "lead": "Нарушения зрения  — снижение способности видеть до такой степени, что вызывает проблемы, не устраняемые обычными способами, такими как очки или лекарства.[1][2][3]\r\n\r\nЭто может быть из-за болезни, травмы, врождённых или дегенеративных заболеваний. Эта потеря зрения, как правило, определяется как:\r\n\r\nОстрота зрения с коррекцией менее 20/60, или значительный дефект центрального поля,\r\nЗначительный одноимённый или разноимённый двусторонний визуальный дефект периферического поля, включающий дефект поля или общее сжатия или сужение поля, или\r\nПониженный пик контрастной чувствительности с любым из указанных выше условий.[1][2][3][4]\r\nГлазные нарушения, которые могут привести к нарушениям зрения могут включать в себя дегенерацию сетчатки, альбинизм, катаракты, глаукомы, мышечные проблемы, которые приводят к нарушениям зрения, расстройства роговицы, диабетическую ретинопатию, врождённые пороки и инфекции. Нарушения зрения также могут быть вызваны расстройствами мозга и нервной системы, в этом случае их, как правило, называют кортикальными нарушениями зрения (CVI). Путеводитель оценки постоянного ухудшения американской медицинской ассоциации попытался представить «стандартизированный, объективный подход к оценке медицинских нарушений». Глава Визуальная система «устанавливает критерии для оценки постоянного ухудшения зрительной системы, как это влияет на способность человека выполнять действия в повседневной жизни».[5] Путеводитель оценил, что потеря одного глаза составляет 25 % нарушений зрительной системы и 24 % от полного обесценивания человека;[5][6]. Полная потеря зрения на оба глаза, как считается, является 100 % нарушением зрения и 85 % от полного обесценивания человека[5]\r\n\r\nВизуальные нарушения наносят значительный экономический удар даже в развитых странах.[7] Значительную часть глобального нарушения зрения можно предупредить.",
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                    "lead": "Нарушения зрения  — снижение способности видеть до такой степени, что вызывает проблемы, не устраняемые обычными способами, такими как очки или лекарства.[1][2][3]\r\n\r\nЭто может быть из-за болезни, травмы, врождённых или дегенеративных заболеваний. Эта потеря зрения, как правило, определяется как:\r\n\r\nОстрота зрения с коррекцией менее 20/60, или значительный дефект центрального поля,\r\nЗначительный одноимённый или разноимённый двусторонний визуальный дефект периферического поля, включающий дефект поля или общее сжатия или сужение поля, или\r\nПониженный пик контрастной чувствительности с любым из указанных выше условий.[1][2][3][4]\r\nГлазные нарушения, которые могут привести к нарушениям зрения могут включать в себя дегенерацию сетчатки, альбинизм, катаракты, глаукомы, мышечные проблемы, которые приводят к нарушениям зрения, расстройства роговицы, диабетическую ретинопатию, врождённые пороки и инфекции. Нарушения зрения также могут быть вызваны расстройствами мозга и нервной системы, в этом случае их, как правило, называют кортикальными нарушениями зрения (CVI). Путеводитель оценки постоянного ухудшения американской медицинской ассоциации попытался представить «стандартизированный, объективный подход к оценке медицинских нарушений». Глава Визуальная система «устанавливает критерии для оценки постоянного ухудшения зрительной системы, как это влияет на способность человека выполнять действия в повседневной жизни».[5] Путеводитель оценил, что потеря одного глаза составляет 25 % нарушений зрительной системы и 24 % от полного обесценивания человека;[5][6]. Полная потеря зрения на оба глаза, как считается, является 100 % нарушением зрения и 85 % от полного обесценивания человека[5]\r\n\r\nВизуальные нарушения наносят значительный экономический удар даже в развитых странах.[7] Значительную часть глобального нарушения зрения можно предупредить.",
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