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"etiology": "<p>Возбудитель — палочковидная грамотрицательная бактерия Rickettsia conorii. Облигатный внутриклеточный паразит; размножается в культуре тканей (в желточном мешке куриного эмбриона) и при заражении лабораторных животных (в клетках мезотелия). Патогенен для морских свинок, обезьян, кроликов, сусликов, белых мышей и белых крыс. По антигенным свойствам близок к другим возбудителям группы клещевых пятнистых лихорадок. Может паразитировать в цитоплазме и ядрах клеток хозяина. У больных возбудитель выявляют в крови в первые дни лихорадочного периода, в первичном аффекте и в розеолах кожи.</p>",
"pathogenesis": "<p>Возбудитель проникает через кожу или слизистые оболочки носа и конъюнктивы. На месте внедрения формируется первичный аффект («чёрное пятно»), выявляемый вскоре после укуса клещом (за 5–7 сут до возникновения симптомов заболевания). Через лимфатическую систему риккетсии сначала попадают в регионарные лимфатические узлы (вызывают лимфоаденит), а затем в кровь (поражают эндотелий капилляров и венул). При этом возникают изменения, схожие с выявляемыми при эпидемическом сыпном тифе, однако количество гранулём (узелков) меньше и некротические изменения выражены слабее.</p>",
"diagnostics": "<p>При диагностике данного заболевания учитывают эпидемиологические предпосылки (пребывание в эндемичной местности, сезон, контакт с собаками, укусы клещами и др.). В клинической картине наибольшее значение имеет триада симптомов:</p>\r\n<p>· первичный аффект («чёрное пятно»);<br />· регионарный лимфаденит;<br />· раннее появление обильной полиморфной сыпи по всему телу, включая ладони и подошвы.</p>\r\n<p>Учитывают умеренную выраженность общей интоксикации и отсутствие тифозного статуса.</p>\r\n<p>Лабораторное подтверждение диагноза основано на серологических реакциях: реакции связывания комплемента со специфическим антигеном (параллельно проводят реакцию и с другими риккетсиозными антигенами), РНГА.</p>\r\n<p> </p>",
"treatment": "<p>Как и при других риккетсиозах, наиболее эффективен тетрациклин.</p>\r\n<p>Патогенетическое лечение направлено на устранение интоксикации и геморрагических проявлений.</p>",
"prevention": "<p>Специфической профилактики не разработано.</p>\r\n<p>В эпидемических очагах проводят обработку возможных мест обитания клещей инсектицидами (например, собак, собачьих будок), отлов бродячих собак.</p>",
"clinical_picture": "<p>Инкубационный период относительно короткий (3–7 сут).</p>\r\n<p>Классификация</p>\r\n<p>Выделяют четыре периода:</p>\r\n<p>· инкубационный;<br />· начальный (до появления сыпи);<br />· разгара;<br />· выздоровления.</p>\r\n<p>Особенность лихорадки — наличие первичного аффекта, выявляемого у большинства пациентов до начала заболевания. Первичный аффект сначала представляет собой очаг воспаления кожи с покрытым тёмной корочкой участком некроза диаметром 2–3 мм в центре. Размеры первичного аффекта постепенно увеличиваются до 5–10 мм к началу лихорадочного периода. Корочка отпадает лишь на 5–7-е сут после установления нормальной температуры. Открывшаяся небольшая язвочка постепенно эпителизируется (в течение 8–12 сут), после чего остаётся пигментированное пятно. Локализация первичного аффекта разнообразна (обычно на участках кожи, закрытых одеждой); может быть 2–3 очага. На субъективные ощущения в области первичного аффекта больные не жалуются. Примерно у трети из них возникает регионарный лимфаденит с небольшим увеличением и болезненностью лимфатических узлов. Начало заболевания острое, с быстрым повышением температуры до 38–40 °С. постоянного типа (реже ремиттирующая) сохраняется в течение 3–10 сут и сопровождается ознобом, сильной головной болью, общей слабостью, выраженной миалгией, а также артралгией и бессонницей. Возможна \">Рвота</a>\">\">рвота</a></a>. При осмотре выявляют гиперемию и некоторую одутловатость лица, инъекцию сосудов склер и слизистых оболочек зева.</p>\r\n<p>Для разгара заболевания характерно появление экзантемы (на 2–4-е сутки его течения), выявляемой у всех больных. возникает сначала на груди и животе, затем распространяется на шею, лицо, конечности; почти у всех больных её обнаруживают на ладонях и подошвах. обильные (особенно на конечностях), состоят из пятен и папул, часть элементов подвергается геморрагическому превращению. У многих больных на месте папул возникают везикулы. На ногах наиболее обильна; её элементы ярче и крупнее, чем на других участках кожи.</p>\r\n<p> исчезают через 8–10 сут, оставляя после себя пигментацию кожи, сохраняющуюся иногда до 2–3 мес.</p>\r\n<p>Выявляют брадикардию, небольшое снижение АД. Существенной патологии органов дыхания не развивается. Живот мягкий или (у некоторых больных) умеренно вздут, при пальпации безболезненный. У 50% больных в лихорадочном периоде выявляют задержку стула и очень редко жидкий стул. У некоторых пациентов выражено и, реже, селезёнки. Снижается суточный диурез и возникает протеинурия (особенно в первую неделю). В период реконвалесценции улучшается общее состояние и угасают все симптомы.</p>",
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"code": "A77",
"name": "Пятнистая лихорадка",
"icd_name": "Пятнистая лихорадка [клещевые риккетсиозы]",
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"age_min": 10,
"age_max": 70,
"cause": [
2,
6
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"slug": "a77_pyatnistaya_lihoradka",
"lead": "Пятнистая лихорадка передается при укусе насекомых (клещей), характеризуется доброкачественным течением, наличием первичного аффекта и распространённой макулопапулёзной сыпью.",
"description": "",
"etiology": "<p>Возбудитель — палочковидная грамотрицательная бактерия Rickettsia conorii. Облигатный внутриклеточный паразит; размножается в культуре тканей (в желточном мешке куриного эмбриона) и при заражении лабораторных животных (в клетках мезотелия). Патогенен для морских свинок, обезьян, кроликов, сусликов, белых мышей и белых крыс. По антигенным свойствам близок к другим возбудителям группы клещевых пятнистых лихорадок. Может паразитировать в цитоплазме и ядрах клеток хозяина. У больных возбудитель выявляют в крови в первые дни лихорадочного периода, в первичном аффекте и в розеолах кожи.</p>",
"pathogenesis": "<p>Возбудитель проникает через кожу или слизистые оболочки носа и конъюнктивы. На месте внедрения формируется первичный аффект («чёрное пятно»), выявляемый вскоре после укуса клещом (за 5–7 сут до возникновения симптомов заболевания). Через лимфатическую систему риккетсии сначала попадают в регионарные лимфатические узлы (вызывают лимфоаденит), а затем в кровь (поражают эндотелий капилляров и венул). При этом возникают изменения, схожие с выявляемыми при эпидемическом сыпном тифе, однако количество гранулём (узелков) меньше и некротические изменения выражены слабее.</p>",
"diagnostics": "<p>При диагностике данного заболевания учитывают эпидемиологические предпосылки (пребывание в эндемичной местности, сезон, контакт с собаками, укусы клещами и др.). В клинической картине наибольшее значение имеет триада симптомов:</p>\r\n<p>· первичный аффект («чёрное пятно»);<br />· регионарный лимфаденит;<br />· раннее появление обильной полиморфной сыпи по всему телу, включая ладони и подошвы.</p>\r\n<p>Учитывают умеренную выраженность общей интоксикации и отсутствие тифозного статуса.</p>\r\n<p>Лабораторное подтверждение диагноза основано на серологических реакциях: реакции связывания комплемента со специфическим антигеном (параллельно проводят реакцию и с другими риккетсиозными антигенами), РНГА.</p>\r\n<p> </p>",
"treatment": "<p>Как и при других риккетсиозах, наиболее эффективен тетрациклин.</p>\r\n<p>Патогенетическое лечение направлено на устранение интоксикации и геморрагических проявлений.</p>",
"prevention": "<p>Специфической профилактики не разработано.</p>\r\n<p>В эпидемических очагах проводят обработку возможных мест обитания клещей инсектицидами (например, собак, собачьих будок), отлов бродячих собак.</p>",
"clinical_picture": "<p>Инкубационный период относительно короткий (3–7 сут).</p>\r\n<p>Классификация</p>\r\n<p>Выделяют четыре периода:</p>\r\n<p>· инкубационный;<br />· начальный (до появления сыпи);<br />· разгара;<br />· выздоровления.</p>\r\n<p>Особенность лихорадки — наличие первичного аффекта, выявляемого у большинства пациентов до начала заболевания. Первичный аффект сначала представляет собой очаг воспаления кожи с покрытым тёмной корочкой участком некроза диаметром 2–3 мм в центре. Размеры первичного аффекта постепенно увеличиваются до 5–10 мм к началу лихорадочного периода. Корочка отпадает лишь на 5–7-е сут после установления нормальной температуры. Открывшаяся небольшая язвочка постепенно эпителизируется (в течение 8–12 сут), после чего остаётся пигментированное пятно. Локализация первичного аффекта разнообразна (обычно на участках кожи, закрытых одеждой); может быть 2–3 очага. На субъективные ощущения в области первичного аффекта больные не жалуются. Примерно у трети из них возникает регионарный лимфаденит с небольшим увеличением и болезненностью лимфатических узлов. Начало заболевания острое, с быстрым повышением температуры до 38–40 °С. постоянного типа (реже ремиттирующая) сохраняется в течение 3–10 сут и сопровождается ознобом, сильной головной болью, общей слабостью, выраженной миалгией, а также артралгией и бессонницей. Возможна \">Рвота</a>\">\">рвота</a></a>. При осмотре выявляют гиперемию и некоторую одутловатость лица, инъекцию сосудов склер и слизистых оболочек зева.</p>\r\n<p>Для разгара заболевания характерно появление экзантемы (на 2–4-е сутки его течения), выявляемой у всех больных. возникает сначала на груди и животе, затем распространяется на шею, лицо, конечности; почти у всех больных её обнаруживают на ладонях и подошвах. обильные (особенно на конечностях), состоят из пятен и папул, часть элементов подвергается геморрагическому превращению. У многих больных на месте папул возникают везикулы. На ногах наиболее обильна; её элементы ярче и крупнее, чем на других участках кожи.</p>\r\n<p> исчезают через 8–10 сут, оставляя после себя пигментацию кожи, сохраняющуюся иногда до 2–3 мес.</p>\r\n<p>Выявляют брадикардию, небольшое снижение АД. Существенной патологии органов дыхания не развивается. Живот мягкий или (у некоторых больных) умеренно вздут, при пальпации безболезненный. У 50% больных в лихорадочном периоде выявляют задержку стула и очень редко жидкий стул. У некоторых пациентов выражено и, реже, селезёнки. Снижается суточный диурез и возникает протеинурия (особенно в первую неделю). В период реконвалесценции улучшается общее состояние и угасают все симптомы.</p>",
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