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"code": "I47",
"name": "Пароксизмальная тахикардия",
"icd_name": "Пароксизмальная тахикардия",
"gender": 0,
"age_min": 0,
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"periodicity": 1,
"slug": "i47_paroksizmalnaya_tahikardiya",
"lead": "Нарушение здоровья, относящееся к группе другие болезни сердца",
"description": "вид аритмии, для которого характерны приступы сердцебиения (пароксизмы) с частотой сердечных сокращений от 140 до 220 и более в минуту, возникающие под влиянием эктопических импульсов, которые приводят к замещению нормального синусового ритма. Обычно приступ начинается внезапно и также внезапно заканчивается. Длительность приступа от нескольких секунд до нескольких часов и суток.",
"etiology": "<p>Частой причиной, вызывающей приступы являются стрессовые реакции — психические или физические. Известно, что стрессовые реакции сопровождаются увеличением содержания адреналина и норадреналина в крови, к которым повышена чувствительность эктопических водителей ритма. этот процесс является одним из патогенетических механизмов пароксизмальной тахикардии.</p>\r\n<p>Возникновение пароксизмальных тахикардий, особенно суправентрикулярных ее форм, связывают также с состоянием нервной системы. Например, хорошо известны случаи пароксизмальной тахикардии с синдромом Вольфа-Паркинсона-Уайта при отсутствии заболеваний сердца. Приступы тахикардии возникали после контузии, а также примерно в одной трети случаев при неврастении и вегетативной дистонии.</p>\r\n<p>Пароксизмальная тахикардия может быть вызвана нервно-рефлекторным путем. В клинике нередко удается установить связь между возникновением приступов пароксизмальной тахикардии и заболеваниями пищеварительного аппарата, диафрагмы, желчного пузыря и почек. Гораздо реже рефлекторные раздражения исходят из других органов — легких и плевры, средостения, позвоночного столба, поджелудочной железы, половых органов.</p>\r\n<p>Желудочковая форма пароксизмальной тахикардии наблюдается чаще при тяжелых органических поражениях сердца. Развитию ишемии в различных областях не только миокарда, но также в специализированной ткани проводниковой системы может способствовать коронарный атеросклероз. Это приводит к возникновению эктопического очага возбуждения в миокарде с высокой степенью автоматизма.<br />У 20 % больных, перенесших инфаркт миокарда выявляется желудочковая тахикардия. Патология может наблюдаться и при других формах ишемической болезни сердца (стенокардия, хроническая коронарная недостаточность, атеросклеротический и постинфарктный кардиосклероз), гипертонической болезни, миокардите, врожденных и приобретенных пороках сердца, тяжелых инфекциях. При тиреотоксикозе и аллергических заболеваниях эта аритмия обнаруживается реже.</p>\r\n<p>Факторы, провоцирующие возникновение аритмии:<br />- эмоциональный стресс;<br />- прием алкоголя;<br />- курение;<br />- гипертонический криз;<br />- прием некоторых медицинских препаратов (сердечных гликозидов).</p>",
"pathogenesis": "<p>Патогенез пароксизмальной тахикардии не до конца изучен. Наибольшее признание получили две теории механизма ее развития — экстрасистолическая и теория «кругового движения».<br /><br />Предполагается, что, как и при экстрасистолии, причиной развития приступа пароксизмальной тахикардии может служить локальное усиление волны возбуждения, приобретающее более устойчивый характер. С этих позиций тахикардия рассматривается как приступ частой непрерывной экстрасистолии.</p>\r\n<p>Возникновение такого рода потологии может возникать вследствие наличия дополнительных путей проведения импульса. Особенно значимыми являются пучок Кента (между предсердием и желудочком) и пучок Джеймса (соединяющий синоатриальный и атриовентрикулярный узлы). При наличии дополнительных пучков происходит как бы «сброс» электрического сигнала ранее, чем это должно происходить в норме, в результате желудочки сокращаются преждевременно. но в большинстве случае этот сигнал возвращается обратно, циркулируя между основным и дополнительным пучком. Это приведет к возникновению наджелудочковой тахикардии. В целом такое состояние называется синдромом предвозбуждения желудочков. Различают два синдрома – Вольфа – Паркинсона – Уайта и Клерка – Леви – Кристеско (синдром укороченного PQ). Таким образом, эти два синдрома могут приводить к развитию суправентрикулярной тахикардии.</p>\r\n<p>Биохимической основой пароксизмальной тахикардии является, прежде всего, нарушение электролитного обмена. При органическом поражении миокарда это нарушение связано, вероятно, с различным содержанием электролитов в пораженной и непораженной части мышцы сердца.</p>",
"diagnostics": "<p>Пароксизмальная тахикардия выявляется по типичности приступа с внезапным началом и окончанием, а также путем исследования частоты сердечных сокращений. Так, при желудочковой форме тахикардии ЧСС обычно не превышает 180 ударов в минуту. При суправентрикулярной тахикардии ЧСС достигает 220-250 уд. в минуту, купирование такого приступа возможно с помощью вагусного маневра.</p>\r\n<p>При регистрации ЭКГ во время приступа определяются характерные изменения формы и полярности зубца Р, а также его расположения относительно желудочкового комплекса QRS, позволяющие различить форму пароксизмальной тахикардии. Для предсердной формы типично расположение зубца Р перед комплексом QRS. При пароксизме, исходящем из предсердно-желудочкового соединения, регистрируется отрицательный зубец Р, расположенный позади комплекса QRS или сливающийся с ним. Для желудочковой формы характерна деформация и расширение комплекса QRS, напоминающего желудочковые экстрасистолы; может регистрироваться обычный, неизмененный зубец Р.</p>\r\n<p>При невозможности зарегистрировать приступ на ЭКГ проводится суточное мониторирование ЭКГ, регистрирующего короткие эпизоды тахикардии, не ощущаемые пациентами. В ряде случаев при пароксизмальной тахикардии проводится запись эндокардиальной электрокардиограммы путем внутрисердечного введения электродов. Для исключения органической патологии проводят УЗИ сердца, МРТ или МСКТ сердца.</p>",
"treatment": "<p>Вопрос о тактике лечения пациентов с пароксизмальной тахикардией решается с учетом формы аритмии, ее этиологии, частоты и длительности приступов, наличия или отсутствия осложнений во время пароксизмов (сердечной или сердечно-сосудистой недостаточности). <br />Большинство случаев желудочковой пароксизмальной тахикардии требуют экстренной госпитализации в отделение кардиологии.<br />До приезда бригады скорой медицинской помощи можно облегчить состояние больного. Для этого в начале приступа необходимо усадить больного, расстегнуть стесняющую одежду, обеспечить доступ свежего воздуха, принять нитроглицерин.</p>\r\n<p><br /><strong>Неотложная помощь при пароксизме включает:</strong></p>\r\n<p><br />Вагусные пробы – приемы, оказывающие механическое воздействие на блуждающий нерв:<br />- проба Вальсальвы (попытка энергичного выдоха при закрытых носовой щели и ротовой полости);<br />- проба Ашнера (равномерное и умеренное надавливание на верхний внутренний угол глазного яблока);<br />- проба Чермака-Геринга (надавливание на область одного или обоих каротидных синусов в области сонной артерии); <br />- попытка вызвать рвотный рефлекс путем раздражения корня языка; обтирание холодной водой и др.<br /> <br />Вагусные пробы направлены на стимуляцию блуждающего нерва, что способствует урежению ритма сердца. Они носят вспомогательный характер, доступны самим пациентам и их родным, но не всегда устраняют аритмию, поэтому введение медикаментов — обязательное условие лечения пароксизма ПТ.<br />Пробы проводят только до момента восстановления ритма, иначе создаются условия для брадикардии и остановки сердца. Массаж каротидного синуса противопоказан пожилым людям с диагностированным атеросклерозом сонных артерий.<br />Медикаментозное лечение. Для купировани приступов аритмии применяются препараты антиаритмики. В стенке клеток миокарда и проводящей системы сердца имеется большое число ионных каналов. Через них идет движение ионов калия, натрия, хлора и других внутрь клетки и из нее. Движение заряженных частиц формирует потенциал действия, то есть электрический сигнал. Механизм действия антиаритмических препаратов заключается в блокаде тех или иных ионных каналов. В результате прекращается течение ионов,и подавляется выработка патологических импульсов, вызывающих аритмию.<br />В ряде случаев при невозможности купировать приступы с помощью лекарственных препаратов используется электрическая кардиоверсия (выполняется при помощи дефибриллятора).</p>\r\n<p>К хирургическому лечению прибегают при особо тяжелом течении пароксизмальной тахикардии и неэффективности противорецидивной терапии. Операция заключается в разрушении дополнительных путей проведения импульса или эктопических очагов самостоятельных сокращений сердца или вживлении электрокардиостимулятора (прибор, поддерживающий или навязывающий оптимальную частоту сердечных сокращений).</p>",
"prevention": "<p>Предупреждение приступов пароксизмальной тахикардии должно проводиться с учетом ее формы, частоты и причины возникновения. При редких приступах (один за несколько месяцев или лет) больному рекомендуется вести здоровый образ жизни (без курения и алкоголя) с исключением физических и психических нагрузок. При частых приступах для их профилактики применяются антиаритмические и седативные лекарственных средства.</p>",
"clinical_picture": "<p>Пароксизмальная тахикардия возникает внезапно, возможно — под влиянием провоцирующих факторов или же среди полного благополучия. Больной замечает четкое время начала пароксизма и хорошо ощущает его завершение. На начало приступа указывает толчок в области сердца, переходящий в усиленное сердцебие. Частота сердечных сокращений во время пароксизма достигает 140-220 и более в минуту при сохраненном правильном ритме. В редких случаях аритмии предшествует аура — начинает кружиться голова, ощущается шум в ушах, сжимающие боли в сердце.</p>\r\n<p><br />Симптомы приступа пароксизмальной тахикардии:</p>\r\n<ul>\r\n<li>Головокружение, обмороки при продолжительном пароксизме;</li>\r\n<li>Слабость, шум в голове;</li>\r\n<li>Одышка;</li>\r\n<li>Сжимающее чувство в сердце;</li>\r\n<li>Неврологические проявления — нарушение речи, чувствительности, гемипарезы, афазия;</li>\r\n<li>Вегетативные расстройства — потливость, тошнота, метеоризм, незначительное увеличение температуры, избыточное выделение мочи низкой плотности.</li>\r\n</ul>\r\n<p>Когда приступ заканчивается, больной испытывает значительное облегчение, становится легко дышать, учащенное сердцебиение прекращается толчком либо чувством замирания в груди.</p>",
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"code": "I71",
"name": "Аневризма и расслоение аорты",
"icd_name": "Аневризма и расслоение аорты",
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"slug": "i71_anevrizma_i_rassloenie_aorty",
"lead": "Нарушение здоровья, относящееся к группе болезни артерий, артериол и капилляров",
"description": "Расслаивающаяся аневризма аорты – дефект внутренней оболочки аневризматически расширенной аорты, сопровождающийся образованием гематомы, продольно расслаивающей сосудистую стенку с формированием ложного канала. Расслаивающаяся аневризма аорты проявляется внезапной интенсивной болью, мигрирующей по ходу расслоения, подъемом артериального давления, признаками ишемии сердца, головного и спинного мозга, почек, внутреннего кровотечения.",
"etiology": "<p>К факторам, способствующим развитию аневризмы аорты и ее расслоению, относятся: </p>\r\n<ul>\r\n<li>атеросклероз со значительным изменением vasa vasorum (сосудов сосудов) аорты, ишемию стенки аорты; </li>\r\n<li>гипертоническую болезнь и резкие колебания артериального давления; </li>\r\n<li>сифилис; </li>\r\n<li>очаговые некрозы среднего слоя сосуда бактериально-эмболического происхождения при острых инфекционных заболеваниях; </li>\r\n<li>врожденные дефекты развития аорты (коарктация, двойная дуга, гипоплазия, незаращение боталлова протока, синдром Марфана); </li>\r\n<li>физическое и нервно-эмоциональное перенапряжение; </li>\r\n<li>повреждение аорты смежным процессом (спондилит, рак пищевода, пенетрация пептической язвы пищевода, двенадцатиперстной кишки) или инородным телом; </li>\r\n<li>закрытую травму грудной или брюшной полости. </li>\r\n</ul>",
"pathogenesis": "<p>Аорта имеет три наиболее уязвимых сегмента - корень, перешеек и диафрагму. Гемодинамическое напряжение указанных зон и их хроническая травматизация приводят к дегенеративным изменениям средней оболочки сосуда. Длительное повышение артериального давления всегда ведет к той или иной степени дилатации аорты. Известно также, что внутренняя оболочка по своей структуре менее растяжима, чем средняя и наружная оболочки, обладающие большей эластичностью. Именно поэтому при одном из очередных «критических» повышений артериального давления происходит разрыв внутренней оболочки. Однако гидравлический удар о стенку аорты и разрыв внутренней оболочки сами по себе могут привести лишь к развитию внутристеночной гематомы. Дальнейшее расслоение происходит лишь при наличии патологического процесса в средней оболочке аорты. Если расслоение прорывает стенку аорты полностью (все три слоя), то происходит быстрая массивная кровопотеря и гибель больного. </p>",
"diagnostics": "<p>В случае подозрения на появление расслаивающей аневризмы аорты требуется срочная диагностика, основными методами которой являются: </p>\r\n<ul>\r\n<li>рентгенография грудной клетки; </li>\r\n<li>УЗДГ; </li>\r\n<li>ЭхоКГ; </li>\r\n<li>компьютерная томография; </li>\r\n<li>магнитно-резонансная томография; </li>\r\n<li>аортография. </li>\r\n</ul>\r\n<p>Рентгенография грудной клетки позволяет определить признаки расслаивающей аневризмы: расширение аорты, плевральный выпот, отсутствие пульсации, деформацию контуров аорты. </p>\r\n<p>ЭхоКГ (УЗИ сердца) – более информативный и доступный метод, позволяющий обнаружить отслоившийся лоскут, определить истинный и ложный канал, оценить атеросклеротическое поражение, состояние аортального клапана, состояние грудной аорты. </p>\r\n<p>Аортография дает возможность определить место начального разрыва, протяженность и местонахождения расслоения, состояние аортального клапана, ветвей аорты, коронарных артерий. </p>\r\n<p>МРТ дает возможность точно определить локализацию разрыва и направление расслоения, оценить состояние ветвей аорты и клапана. </p>",
"treatment": "<p>Расслоение аорты требует неотложной медицинской помощи, немедленного лечения. Терапия может заключаться в проведении операции, употреблении лекарственных средств, а также в комбинированном лечении — схема зависит от области аорты, которая подверглась расслоению. </p>\r\n<p>Хирургическое вмешательство - хирурги удаляют как можно большую площадь, подвергшуюся расслоению, а затем реконструируют аорту при помощи синтетической трубки-трансплантата. Если в результате расслоения повреждаются аортальные клапаны, они так же могут быть заменены; </p>\r\n<p>Интенсивная терапия расслаивающейся аневризмы аорты направлена на купирование болевого синдрома (путем введения ненаркотических и наркотических анальгетиков), выведение из шокового состояния, снижение артериального давления. Лекарственные препараты, например, бета-блокаторы и нитропруссид, уменьшают частоту сердечных сокращений и нормализуют низкое кровяное давление, что предотвращает усугубление расслоения. </p>",
"prevention": "<p>Профилактика формирования расслаивающейся аневризмы аорты заключается в контроле за течением сердечно-сосудистых заболеваний. Предупреждение расслоения аорты включает наблюдение кардиолога, мониторинг артериального давления и уровня холестерина крови, периодическое ультразвуковая диагностика сердца и аорты. <br class=\"SCXW45342897\" /> <br class=\"SCXW45342897\" /> </p>",
"clinical_picture": "<p>Для расслаивающейся аневризмы аорты характерно внезапное начало с имитацией симптомов различных сердечно-сосудистых, неврологических, урологических заболеваний. Расслоение аорты проявляется резким нарастанием раздирающей, нестерпимой боли с широкой областью иррадиации (за грудиной, между лопатками и по ходу позвоночника, в эпигастральной области, пояснице), мигрирующей по ходу расслоения. </p>\r\n<p>Отмечается повышение артериального давления с последующим спадом, асимметрия пульса на верхних и нижних конечностях, обильное потоотделение, слабость, синюшность, двигательное беспокойство. Большая часть больных с расслаивающейся аневризмой аорты умирает от развития осложнений. </p>\r\n<p>Неврологическими проявлениями расслаивающейся аневризмы аорты могут служить ишемическое поражение головного или спинного мозга парезы, параличи), периферическая нейропатия, нарушения сознания(обморок, кома). </p>\r\n<p>Расслаивающаяся аневризма восходящей аорты может сопровождаться ишемией миокарда, сдавлением органов средостения (появлением охриплости, дисфагии, одышки, синдрома Горнера, синдрома верхней полой вены), гемоперикарда, тампонады сердца (скопление жидкости между листками околосердечной сумки). </p>\r\n<p>Расслаивающаяся аневризма нисходящей грудной и брюшной части аорты выражается развитием выраженного повышения давления в почечных сосудах и острой почечной недостаточности, острой ишемии органов пищеварения, ишемии кишечника, острой ишемии нижних конечностей. </p>",
"image": null,
"image_alt": null,
"standard_type": 3,
"danger": 1,
"published": 2,
"parent": null,
"block_rubric": 97,
"standards": [
5167
]
}
]
}
