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"diagnostics": "<p>По характеру регулирующих механизмов выделяют обычно расстройства нервной, эндокринной (гормональной), гормоноидной и метаболической регуляции. В соответствии с регулируемыми сферами жизнедеятельности можно выделить нарушения регуляции поведения, сенсорных, локомоторных, вегетативных и трофических функций. В изменениях регуляции поведения выделяют обычно регуляторные механизмы нарушения мотивации (например, при токсикоманиях, булимии, парарексии и др.), эмоций (аффективные состояния), памяти (в частности, при амнезии). </p>\r\n<p>Регуляторные механизмы расстройств чувствительности в свою очередь обычно разделяют по уровню поражения сенсорных систем (рецепторный, проводниковый, центральный — спинальный, стволовой, таламический, корковый) и их модальности (зрительная, слуховая, обонятельная и т.д.). Нарушения регуляции локомоторных функций включают нарушения регуляции произвольных (парезы и параличи) и непроизвольных (например, судорожные состояния) движений. Изменения регуляции вегетативных функций раскрывают расстройства симпатической и парасимпатической регуляции и также разделяются в соответствии с нарушениями работы анатомо-физиологических систем: патология регуляции кровообращения, дыхания, пищеварения и тд. Патология регуляции трофических функций касается изменения нейроплазматического тока, изменения синтеза в нейронах трофогенов и появления патотрофогенов, возникновения нейрогенных атрофии и дистрофии.</p>\r\n<p>Нарушения регуляции можно дифференцировать по ведущим механизмам, лежащим в основе ее формирования. В связи с этим следует выделять расстройства регуляции, возникающие вследствие изменения прямых и обратных регуляторных связей различного характера (положительных и отрицательных). Чрезмерное усиление прямых положительных (стимулирующих) влияний имеет место при возрастании выработки либеринов гипоталамуса или тропных гормонов гипофиза при гормонпродуцирующих опухолях; обратное явление вплоть до прекращения образования этих соединений имеет место при белковом голодании, повторяющихся микротравмах мозга (у боксеров), расстройствах кровообращения в мозге и в гипофизе и т.д.</p>",
"treatment": "<p>Связь местного и общего в патологии осуществляется не с помощью каких-либо особых механизмов; это взаимодействие обусловлено включением обычных интегративных связей, к которым следует отнести рефлекторные, эндокринные, гормоноидные и метаболические акты. Они существуют и в норме (медиаторы, гормоны, тканевые гормоны, продукты обмена) и проявляют себя при патологии. Интегративные связи, определяющие целостность организма, являются ведущим механизмом формирования в патологии приспособительных процессов — от поведенческих высшей степени сложности до молекулярных. В большинстве случаев взаимоотношения части (местного) и целого (общего) при различных патологических процессах отражаются в виде общеизвестных формул: \"нет ничего местного, все есть общее с местным проявлением\" и соответственно \"всякая болезнь есть страдание всего организма\". Эти формулировки стали незыблемыми, обязательным постулатом, вошедшим во все энциклопедии, руководства и учебники. Ими подчеркивается единство организма как целостной системы и неразрывная связь местного и общего. Однако сегодня на эту связь мы смотрим не только более широко, но по-иному; в указанных выше постулатах не просматриваются диалектические отношения, которые складываются между частью и целым, но отчетливо виден примат последнего, т.е. общего. Объективно существуют и местные (часть), и общие (целое) изменения; утверждения о том, что тот или иной процесс в норме или при патологии есть либо местное, либо общее явление, следует считать односторонним. Любое целое состоит из его частей; целое не представляет собой арифметическую сумму частей, а в соответствии с системным принципом приобретает новое качество, отсутствующее у его частей. Существует организменный уровень состояния системы, например здоровье или болезнь; вместе с тем части целого отражают его свойства и сами вносят вклад в их формирование. Проблема части (местного) и целого (общего) в условиях патологии состоит не столько в их утверждении, сколько в раскрытии конкретных механизмов взаимодействия и роли этих механизмов в развитии и исходе заболеваний.</p>",
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]
}