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"lead": "сужение отверстия трехстворчатого клапана, препятствующее поступлению крови из правого предсердия в правый желудочек",
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"etiology": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">В этиологии органического трикуспидального стеноза ведущая роль отводится ревматизму и инфекционному эндокардиту. Ревматическое поражение трехстворчатого клапана всегда сочетается с повреждением аортального и митрального клапанов; при этом отмечается склерозирование створок клапанов, фиброзного кольца, сухожильных хорд и паппиллярных мышц, сращение комиссур. Реже трикуспидальный стеноз бывает обусловлен субэндокардиальным фиброэластозом, болезнью Уиппла, болезнью Андерсона-Фабри, карциноидным синдромом, системной красной волчанкой.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Развитие функционального трикуспидального стеноза обычно связано с миокардитами, легочной и сердечной недостаточностью. Причинами механической обструкции правого атриовентрикулярного отверстия могут выступать метастатические опухоли (нефробластома, меланома, семинома, рак печени и рак щитовидной железы), миксома или тромб правого предсердия.</span></p>",
"pathogenesis": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Стеноз правого предсердно-желудочкового отверстия вызывает затруднение физиологического поступления крови из правого предсердия в правый желудочек. При этом в диастолу создается повышенный транстрикуспидальный градиент давления, обусловленный увеличением давления в полости правого предсердия. В течение короткого времени компенсация нарушенной гемодинамики обеспечивается гиперфункцией и гипертрофией правого предсердия. Однако компенсаторные механизмы при трикуспидальном стенозе неустойчивы и ограничены, поскольку мощность миокарда правого предсердия оказывается недостаточной.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Прогрессирующий рост давления в правом предсердии приводит к застою во всей венозной системе. В портальной системе и печени депонируется большое количество крови, поэтому рост портальной гипертензии способствует развитию асцита, фиброза и цирроза печени.</span></p>",
"diagnostics": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Предварительный диагноз стеноза трехстворчатого клапана ставится на основании анамнеза, объективного обследования и результатов обследования. С этой целью врач может назначить следующие виды обследования:</span></p>\r\n<ul style=\"margin-top: 0; margin-bottom: 0; padding-inline-start: 48px;\">\r\n<li><span style=\"font-size: 7pt; color: #000000; background-color: transparent; font-style: normal; font-variant-numeric: normal; font-variant-east-asian: normal; vertical-align: baseline; white-space: pre-wrap;\">эхокардиографию;</span></li>\r\n<li><span style=\"font-size: 7pt; color: #000000; background-color: transparent; font-style: normal; font-variant-numeric: normal; font-variant-east-asian: normal; vertical-align: baseline; white-space: pre-wrap;\">электрокардиографию (ЭКГ);</span></li>\r\n<li><span style=\"font-size: 7pt; color: #000000; background-color: transparent; font-style: normal; font-variant-numeric: normal; font-variant-east-asian: normal; vertical-align: baseline; white-space: pre-wrap;\">рентгенологическое исследование грудной клетки;</span></li>\r\n<li><span style=\"font-size: 7pt; color: #000000; background-color: transparent; font-style: normal; font-variant-numeric: normal; font-variant-east-asian: normal; vertical-align: baseline; white-space: pre-wrap;\">катетеризацию сердца.</span></li>\r\n</ul>",
"treatment": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Для снижения хирургических рисков в предоперационном периоде проводится терапия сердечной недостаточности с назначением бессолевой диеты, диуретиков, венозных вазодилататоров, сердечных гликозидов, блокаторов бета-адренорецепторов, антикоагулянтов, метаболических препаратов.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Выбор операции зависит от структуры и причин порока. Больным с митральным и трикуспидальным стенозом показано проведение двойной митрально-трикуспидальной комиссуротомии. При комбинированном трикуспидальном пороке выполняется открытая комиссуротомия в условиях ИК (при этом спайки между передней и задней створками не рассекают во избежание недостаточности), пластика или протезирование трикуспидального клапана биопротезом. Протезирование проводится в случае грубых изменений створок клапана и подклапанных структур. При изолированном трикуспидальном стенозе может быть предпринята баллонная вальвулопластика.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">При миксомах сердца производится радикальное удаление внутриполостной опухоли, при метастатической обструкции выполняются паллиативные операции. К числу возможных послеоперационных осложнений следует отнести протезный эндокардит, тромбоз и кальциноз протеза, ТЭЛА, паравальвулярные фистулы, АВ-блокаду.</span></p>",
"prevention": "<p> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Для предупреждения трикуспидального стеноза наиболее эффективным является целенаправленная профилактика ревматизма и инфекционного эндокардита, которая должна осуществляться под руководством ревматолога и кардиолога. Важное значение придается лечению очагов хронической инфекции (хронического тонзиллита, кариеса зубов), закаливанию.</span></p>",
"clinical_picture": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Признаками трикуспидальной недостаточности являются общая слабость и быстрая утомляемость, тахикардия, тяжесть или боли в эпигастрии и правом подреберье. Беспокоят диспеп­сические расстройства: тошнота, отрыжка, метеоризм. При осмотре больных с трикуспидальным стенозом обнаруживается цианоз в сочетании с желтушностью кожных покровов, отеки нижних конечностей, асцит, застойное набухание и пульсация шейных вен и печени. На фоне повышенного венозного давления отмечается артериальная гипотония. При тяжелом трикуспидальном стенозе развивается цирроз печени, истощение, анасарка, спленомегалия. В стадии декомпенсации трикуспидального стеноза нередко возникают флеботромбозы и тромбоэмболия легочных артерий.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Как и в случае трикуспидальной недостаточности, беременность у женщины с трикуспидальным стенозом может осложниться развитием гестоза, фетоплацентарной недостаточности и преждевременными родами.</span></p>",
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Миксоматозная дегенерация (увеличение толщины и снижение плотности створок клапана).</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Приобретенная органическая (связанная с изменениями створок клапана) недостаточность трикуспидального клапана может возникнуть по следующим причинам:</span></p>\r\n<ul>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Закрытая травма сердца с разрывом створок трехстворчатого клапана;</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Карциноидная болезнь или карциноидный синдром (поражение различных органов в результате наличия в организме карциноида). Карциноид – это маленькая опухоль, чаще всего расположенная в тонком или толстом кишечнике. Опухоль производит активные вещества, которые током крови приносятся в правую половину сердца, повреждают эндокард (внутреннюю оболочку сердца). Выходя из правого желудочка с током крови, эти вещества попадают в сосуды легких, где разрушаются и не доходят до левых отделов сердца. При карциноидной болезни трехстворчатый клапан может быть постоянно закреплен в полуоткрытой позиции.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Ревматизм (системное (то есть с поражением различных органов и систем организма) воспалительное заболевание с преимущественным поражением сердца) – самая частая причина недостаточности трикуспидального клапана. Трикуспидальная недостаточность при ревматизме всегда сочетается с поражением других клапанов.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Инфекционный эндокардит (воспалительное заболевание внутренней оболочки сердца).</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Оперативное лечение митрального стеноза: при митральной комиссуротомии (хирургическом разделении сросшихся створок митрального клапана) возможно появление недостаточности трикуспидального клапана, так увеличение тока крови делает явной скрытую до этого трикуспидальную недостаточность.</span></li>\r\n</ul>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\"> </span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">При расширении фиброзного кольца (плотного кольца внутри стенок сердца, к которому крепятся створки клапанов) по следующим причинам:</span></p>\r\n<ul>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">миокардит (воспаление мышцы сердца);</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">дилатационная кардиомиопатия (заболевание сердца, при котором происходит увеличение его полостей и уменьшение толщины мышцы сердца);</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">увеличение правого желудочка при некоторых врожденных пороках сердца, например, при дефекте межжелудочковой перегородки (наличии отверстия в перегородке между левым и правым желудочками сердца со сбросом крови слева направо);</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">разрывы хорд (сухожильных нитей, соединяющие мышцу сердца с папиллярными мышцами – внутренними мышцами сердца, обеспечивающими движение клапанов);</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">тяжелая легочная гипертензия (значительное повышение давления крови в артериях легких);</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">препятствие току крови из правого желудочка (например, опухоль). </span></li>\r\n</ul>\r\n<p> </p>",
"pathogenesis": "",
"diagnostics": "<p> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Инструментальная диагностика при трикуспидальной недостаточности включает полипозиционную рентгенографию грудной клетки, рентгенографию сердца с контрастированием пищевода, фонокардиографию, ЭКГ, ЭхоКГ с допплеровским исследованием, зондирование правых отделов сердца, вентрикулографию, яремную флебографию и др.</span></p>",
"treatment": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Консервативная тактика при трикуспидальной недостаточности носит, главным образом, паллиативный характер и направлена на борьбу с сердечной недостаточностью и легочной гипертензией. Больным назначаются диуретики, венозные вазодилататоры, ингибиторы АПФ, b-адреноблокаторы, сердечные гликозиды, метаболические препараты, антикоагулянты. При наличии гидроторакса прибегают к проведению плевральных пункций или дренирования; в случае скопления выпота в брюшной полости – к лапароцентезу.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">При трикуспидальной недостаточности I степени хирургическое лечение не показано. Основаниями для кардиохирургического вмешательства служат гемодинамически значимые нарушения при трикуспидальной недостаточности II-IV степень. Основными видами операций служат пластика трикуспидального клапана и протезирование трикуспидального клапана биологическим или искусственным протезом. Протезированию отдается предпочтение при инфекционном эндокардите, грубых структурных изменениях клапана, неэффективности аннулопластики.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Оперативное лечение трикуспидальной недостаточности сопряжено с риском развития протезного эндокардита, дегенерации биологического протеза, тромбоза или кальциноза искусственного протеза, АВ-блокады.</span></p>",
"prevention": "<p> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Основу профилактики трикуспидальной недостаточности служит предупреждение рецидивов ревматизма, лечение основного заболевания для предупреждения развития относительной недостаточности клапана, систематическое наблюдение кардиолога и кардиохирурга.</span></p>",
"clinical_picture": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Симптомы трикуспидальной недостаточности:</span></p>\r\n<ul>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Одышка — возникает в результате недостаточного поступления крови в сосуды легких.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Ощущение неритмичного сердцебиения, замирания сердца, переворотов в левой половине грудной клетки возникает при развитий аритмий (нарушений ритма сердца) вследствие повреждение мышцы сердца тем же процессом, который вызвал трикуспидальную недостаточность (например, травма сердца или миокардит – воспаление мышцы сердца) и вследствие изменения структуры предсердия. </span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Боль и тяжесть в правом подреберье связаны с застоем жидкости в печени.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Чувство переполнения живота, тяжесть в его верхних отделах, отрыжка — возникают при переполнении кровью сосудов брюшной полости.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Общая слабость и снижение работоспособности — связаны с нарушением распределения крови в организме. </span></li>\r\n</ul>\r\n<p> </p>",
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"etiology": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Миокардиты включают большую группу заболеваний сердечной мышцы воспалительного генеза, проявляющихся поражением и нарушением функции миокарда. Частой причиной миокардита являются различные инфекционные заболевания:</span></p>\r\n<ul>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">вирусные (вирусы Коксаки, гриппа, аденовирусы, герпеса, гепатита В и С);</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">бактериальные (коринеобактерии дифтерии, стафилококки, стрептококки, сальмонеллы, хламидии, риккетсии);</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">грибковые (аспергиллы, кандиды),;</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">паразитарные (трихинеллы, эхинококки) и др.</span></li>\r\n</ul>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Тяжелая форма миокардита может возникать при дифтерии, скарлатине, сепсисе. Высокой кардиотропностью обладают вирусы, вызывающие миокардит в 50% случаев. Иногда миокардит развивается при системных заболеваниях соединительной ткани: системной красной волчанке, ревматизме, васкулитах, ревматоидном артрите, при аллергических заболеваниях. Также причиной миокардита может служить токсическое воздействие некоторых лекарственных препаратов, алкоголя, ионизирующее излучение. Тяжелое прогрессирующее течение отличает идиопатический миокардит невыясненной этиологии.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Провоцирующими моментами возникновения миокардита являются острые инфекции (чаще вирусные), очаги хронической инфекции; аллергозы, нарушенные иммунологические реакции; токсическое воздействие на организм (лекарств, алкоголя, наркотиков, ионизирующего излучения, при тиреотоксикозе, уремии и др.).</span></p>\r\n<p> </p>",
"pathogenesis": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Наиболее важную патогенетическую роль играет М-протеин, который обладает сильной иммуногенностью, т. е. способностью вызывать иммунный ответ. Этот белок входит в состав мембраны стрептококка и имеет ту же молекулярную структуру, что и мембраны кардиомиоцитов (феномен молекулярной мимикрии). Благодаря такому феномену возникает перекрестная аутоиммунная реакция, при которой Т-лимфоциты наряду с бактериями атакуют собственные клетки сердца. В патогенезе также принимают участие экзоферменты стрептококка, которые обладают прямым повреждающим действием на клетки соединительной ткани (стрептолизин О, стрептолизин S, гиалуронидаза, стрептокиназа).</span></p>\r\n<p> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Патоморфологически ревмокардит протекает в 4 стадии, каждая из которых продолжается 1-2 месяца: мукоидное набухание, фибриноидное набухание, образование ревматической гранулемы, склероз. В процессе этих стадий происходит разрушение основного вещества соединительной ткани, образуются очаги некроза и ревматические гранулемы. Исходом является склероз, т. е. рубцевание. Наиболее пагубное влияние эти процессы оказывают на сердечные клапаны, которые деформируются, что приводит к формированию сердечных пороков. Именно пороки сердца в исходе ревматического воспаления и являются чрезвычайно актуальной проблемой.</span></p>",
"diagnostics": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Ревмокардит является основным проявлением ОРЛ, для диагностирования которой используются специально разработанные клинические и лабораторные критерии. Они включают кардит, артрит, малую хорею, кольцевидную эритему, подкожные узелки, лихорадку и данные, свидетельствующие о наличии стрептококковой инфекции в организме. Диагностика непосредственно ревмокардита проводится следующими методами:</span></p>\r\n<ul>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Аускультативными. При ревмокардите аускультативная картина разнообразна. У пациентов выслушиваются шумы в проекции митрального и аортального клапанов (наиболее специфичны протодиастолический шум и мезосистолический шум Кера-Кумбса), ослабление и глухость тонов сердца, дополнительные тоны, трение перикарда, изменение частоты сердечных сокращений, неритмичное сердцебиение, ритм галопа.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Лабораторными. В общем и биохимическом анализе крови выявляется наличие воспалительных маркеров (нейтрофильный лейкоцитоз, сдвиг лейкоцитарной формулы влево, увеличение скорости оседания эритроцитов, С-реактивного белка). Также обнаруживаются высокие титры антител к стрептококку - антистретолизин-О (АСЛО), антистрептогиалуронидаза (АСГ), антистрептокиназа (АСК), антидезоксирибонуклеаза-В (АДНК-В).</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Инструментальными. На ЭКГ обнаруживаются нарушения ритма и проводимости сердца – тахи- или брадикардия, удлинение интервала PQ, экстрасистолия, предсердно-желудочковая блокада I или II степени, признаки перегрузки левых отделов сердца. Эхо-КГ – наиболее точный визуализирующий метод. Выявляет утолщение или деформацию створок клапанов, признаки регургитации (обратного тока крови), снижение сократительной функции сердца (фракции выброса), расширение полостей сердца, утолщение листков перикарда, выпот в перикардиальную полость.</span></li>\r\n</ul>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>",
"treatment": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Первым условием успешного лечения является госпитализация в кардиологический стационар или отделение ревматологии. Необходимо соблюдать постельный режим минимум 4 недели. При тяжелом ревмокардите обязателен строгий постельный режим до исчезновения симптомов недостаточности кровообращения. Рекомендуется питание с повышенным содержанием белка и витаминов. Лечение ревмокардита следующее:</span></p>\r\n<ul>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Этиотропная терапия. Так как основной причиной ревмокардита является стрептококковая инфекция, этиотропная терапия направлена на ее эрадикацию. Для этого применяются антибиотики пенициллинового ряда – бензилпенициллин, феноксиметилпенициллин. При развитии аллергических реакций на пенициллины их заменяют другими группами антибиотиков – макролидами (азитромицин), цефалоспоринами (цефуроксим), линкозамидами (линкомицин).</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Патогенетическая терапия. Подразумевает лечение непосредственного ревматического воспаления. Обычно применяются нестероидные противовоспалительные средства (в основном, это диклофенак). Однако при тяжелом течении ревмокардита необходимы препараты, обладающие мощным противовоспалительным действием. В таких случаях используют глюкокортикостероиды (преднизолон).</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Симптоматическая терапия. Заключается в коррекции сердечной недостаточности. Обязательно нужно ограничить употребление поваренной соли (менее 3 грамм в сутки). Лекарственные средства применяют у пациентов с уже имеющимся пороком сердца и тяжелой недостаточностью кровообращения. Это ингибиторы АПФ, диуретики, бета-блокаторы, сердечные гликозиды, антагонисты альдостерона.</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Хирургическое лечение. Показано при сформировавшихся тяжелых пороках сердца, вызывающих грубые гемодинамические нарушения и резистентных к лекарственной терапии. В зависимости от вида порока (стеноз или недостаточность), а также от пораженного клапана применяются коммисуротомия, вальвулопластика или протезирование клапана искусственными или биологическими протезами.</span></li>\r\n</ul>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">При выписке из стационара необходимо постоянное диспансерное наблюдение у кардиолога или ревматолога, периодическое проведение эхокардиографии для оценки состояния клапанов сердца. Обязательно посещение оториноларинголога, который проводит промывание лакун миндалин. При неэффективности промывания прибегают к радикальному методу ‒ тонзиллэктомии. Ее следует выполнять при стихании активного ревматического процесса и стабильном состоянии пациента.</span></p>",
"prevention": "<p> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Первичная профилактика подразумевает правильное и своевременное лечение тонзиллита. С этой целью применяются те же антибиотики, что и для лечения ревмокардита. Для предупреждения рецидива ревмокардита существует так называемая вторичная профилактика. Она заключается в ежемесячном внутримышечном введении антибиотиков пенициллинового ряда пролонгированного действия (бензатина бензилпенициллин). Продолжительность такой профилактики зависит от того, сформировался после ревмокардита порок сердца или нет.</span></p>",
"clinical_picture": "<p> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Клинические проявления начинают развиваться через 2-3 недели после стрептококковой инфекции верхних дыхательных путей. В начале заболевания симптоматика неспецифична – утомляемость, повышение температуры тела до субфебрильных или фебрильных цифр, потливость. Примерно через неделю присоединяются симптомы со стороны сердца - затруднение дыхания, ощущение сильного сердцебиения и перебоев в работе сердца, боли в области сердца неопределенного характера. В случае тяжелого течения ревмокардита наряду с вышеперечисленными симптомами возникают выраженная одышка, кашель, отеки ног, тяжесть в правом подреберье из-за увеличенной печени. Период атаки ревмокардита продолжается от 2-х до 6-ти месяцев, после чего либо наступает выздоровление, либо формируется порок сердца.</span></p>",
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}
]
}