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"lead": "Нарушение здоровья, относящееся к группе общие симптомы и признаки",
"description": " Гипергидроз — обильное потоотделение, происходящее независимо от физических факторов: повышенной двигательной активности, перегревания, высокой температуры окружающей среды или других.",
"etiology": "<p>Различают общий и локализованный гипергидроз: </p>\r\n<ul>\r\n<li>Общий гипергидроз наблюдается при воздействии высокой температуры окружающей среды, физических и эмоциональных напряжениях (физиологический гипергидроз), а также при ряде болезней (туберкулёз, поражения нервной системы, резком падении уровня глюкозы в сыворотке крови). </li>\r\n<li>Наиболее частые формы локализованного гипергидроза — ладонно-подошвенный гипергидроз и гипергидроз крупных складок. Нередко эти формы являются проявлением вегетососудистой дистонии, а также результатом несоблюдения личной гигиены, плоскостопия, использования тесной, резиновой обуви, одежды из синтетических тканей и т.д.</li>\r\n</ul>",
"pathogenesis": "<p>Терморегуляционныйгипергидроз. Потоотделение – один из важнейших путей терморегуляции наряду c излучением тепла, теплопроводностью и конвекцией. Утрата тепла путём испарения находится под контролем главного механизма регуляции потоотделения в организме – гипоталамуса, и осуществляется в автономном режиме. Данный процесс является основным способом охлаждения при повышении температуры тела человека. На интенсивность терморегуляционного потоотделения влияют многочисленные факторы – температура окружающего воздуха и тела, влажность и интенсивность движения воздуха, физическое напряжение, теплоизоляционные свойства одежды, площадь поверхности тела, количество функционирующих потовых желез, прямое и отражённое излучение и т.д. </p>\r\n<p><br />Психогенный гипергидроз.Психогенный (эмоциональный) гипергидроз возникает в результате эмоционального и психического напряжения (тревога, гнев, страх, боль). Повышение потоотделения может иметь при этом различную интенсивность, усиливаться как на ограниченных участках (преимущественно на ладонях и подошвах), так и на всей поверхности тела.</p>\r\n<p><br />Пищевой гипергидроз.Пищевой гипергидроз может наблюдаться после еды, богатой экстрактивными веществами (острых блюд, алкоголя), и связан с наличием нейронных связей в стволе мозга между слюноотделительными ядрами и симпатическими путями. Бывает локализованным (преимущественно в области лица) и генерализованным (по всему телу).</p>\r\n<p><br />Спонтанный гипергидроз.Спонтанный гипергидроз возникает внезапно и обусловлен психогенными факторами.<br />Фармакологический гипергидроз.Вызывается приёмом лекарственных препаратов, воздействующих преимущественно на парасимпатический отдел вегетативной нервной системы.</p>",
"diagnostics": "<p>Диагноз устанавливается на основании жалоб больного и показании йодно-крахмальная проба Минора. Суть ее состоит в следующем: на обследуемые участки тела пациента наносят раствор йода, после высыхания которого притрушивают пятно крахмалом и следят за реакцией. При выделении пота йод растворяется и вступает в реакцию с крахмалом, от чего места, на которых выделился пот, чернеют. По размерам почерневшего пятна врачи определяют зону повышенного потоотделения и соответственно объемы предстоящего лечения. </p>",
"treatment": "<p>Современная медицина располагает различными методами лечения гипергидроза: </p>\r\n<ul>\r\n<li>безоперационными – применение медицинских антиперспирантов, ионофорез; </li>\r\n<li>малоинвазивными – инъекции ботулотоксина; </li>\r\n<li>инвазивными – симпатэктомия, кюретаж, лечение лазером. </li>\r\n</ul>\r\n<p>Начинают лечение обычно с наименее инвазивных способов – антиперспирантов и ионофореза. И только если они не дают желаемого результата, проводят инъекции ботоксом или оперативное вмешательство. </p>\r\n<p>МЕДИЦИНСКИЕ АНТИПЕРСПИРАНТЫ </p>\r\n<p>Эти средства могут использоваться для области подмышек, рук, стоп и даже лица. По своему действию они похожи на косметические антиперспиранты, однако концентрация активных веществ (обычно солей алюминия) в них намного выше. </p>\r\n<p>ИОНОФОРЕЗ </p>\r\n<p>Ионофорез – это достаточно эффективный и безопасный метод лечения гипергидроза. Суть данного метода заключается в том, что токи и ионизирующие вещества, проходя через кожу, «выключают» потовые железы. </p>\r\n<p>ЛЕЧЕНИЕ БОТУЛОТОКСИНОМ </p>\r\n<p>Инъекции ботокса, широко используемые для омоложения, применяются и для лечения гипергидроза. При внутрикожном введении ботулотоксин блокирует передачу нервных импульсов от нервов к потовым железам и тем самым уменьшает выделение пота. Особенно эффективен этот метод при аксиллярном (подмышечном) гипергидрозе. Эффект от применения данного метода лечения сохраняется 6-8 месяцев. </p>\r\n<p>ИНВАЗИВНЫЕ МЕТОДЫ ЛЕЧЕНИЯ ГИПЕРГИДРОЗА </p>\r\n<p>Кюретаж – хирургический метод лечения аксиллярного гипергидроза. Суть заключается в механическом разрушении потовых желез с помощью специального приспособления – кюретки. Проводится данная процедура под местной анестезией, для введения кюретки на коже делается небольшой разрез. Эффект после кюретажа сохраняется 4-6 месяцев, потом появляются новые потовые железы, однако такой повышенной потливости, как до операции, как правило уже не наблюдается. Побочным действием применения кюретажа является временное нарушение чувствительности под мышками из-за повреждения нервных волокон. </p>\r\n<p>Самый современный метод лечения гипергидроза – это применение лазера. Во время операции под кожу аксиллярной области через небольшой разрез вводится световод, после чего лазерный луч разрушает потовые железы. Восстановление после такой операции проходит намного быстрее, чем после обычного кюретажа. </p>\r\n<p>Симпатэктомия – еще один метод лечения гипергидроза, заключающийся в пересечении или клипировании симпатических нервных волокон. При повышенной потливости ладоней проводится эндоскопическая торакальная симпатэктомия, а при гипергидрозе стоп – поясничная симпатэктомия. Осуществляются эти операции под общим наркозом. </p>",
"prevention": "<p>Главными методами профилактики в борьбе с гипергидрозом должно стать тщательное соблюдение правил личной гигиены: следует ежедневно принимать душ, делать теплые ванночки для ладоней и стоп (лучше с настоем ромашки или дубовой коры), делать обтирания. Для уничтожения запаха пота рекомендуется пользоваться дезодорантами. Не рекомендуется носить одежду, особенно нижнее белье и носки, из синтетических тканей, поскольку они воздухонепроницаемы. Менять белье, носки, колготки следует как можно чаще. Обувь должна быть удобной, легкой со специальными стельками-вкладышами, а летом – открытой. Необходимо ограничить, а по возможности и исключить из рациона совсем, слишком горячую, острую пищу, алкоголь, никотин, кофе. Усиливают неприятный запах пота такие продукты, как лук и чеснок, специи.</p>",
"clinical_picture": "<p>Гипергидроз подмышек </p>\r\n<p>Повышенная потливость подмышек — наиболее распространенная форма гипергидроза. У человека заметны: </p>\r\n<ul>\r\n<li>Огромные мокрые следы в подмышечных впадинах; </li>\r\n<li>При высыхании пот оставляет следы на одежде желтоватого, красного и фиолетового цвета; </li>\r\n<li>Резкий запах, заметный для окружающих даже при использовании дезодоранта. </li>\r\n</ul>\r\n<p>Гипергидроз стопы </p>\r\n<p>Неприятный запах от ног, быстро возникающий после мытья, обусловлен изменением кислотности кожи. При этом на «вечно мокрых от пота» ногах быстро развивается грибок, что еще более усугубляет внешние проявления гипергидроза. </p>\r\n<p>Гипергидроз ладоней </p>\r\n<p>Мокрые, всегда холодные ладони — в большинстве случаев признак неустойчивой нервной системы и высокой эмоциональности. Даже минимальное переживание может спровоцировать моментальное потение ладоней. Человек стесняется при рукопожатии и общении с другими людьми. </p>\r\n<p>Гипергидроз ног </p>\r\n<p>В этом случае потеют не только стопы, мокрой испариной покрывается вся кожа ног. Капли пота стекают от паховой области, вызывая потертости на внутренней поверхности бедер и раздражая наиболее нежную кожу подколенных ямок. </p>\r\n<p>Другие признаки гипергидроза </p>\r\n<p>Гипергидроз может локализоваться и на других участках кожи. Несколько реже встречается общий гипергидроз тела. При этом появляется: </p>\r\n<ul>\r\n<li>Покраснение и блеск лица, капли пота на лбу (человек постоянно вытирает лицо салфеткой); </li>\r\n<li>Потертости под грудью у женщин; </li>\r\n<li>Мацерация кожи в паху; </li>\r\n<li>Стекающие ручейки пота по позвоночнику; </li>\r\n<li>Мокрое нижнее белье — в таких случаях человек говорит: «мокрый, хоть выжимай». </li>\r\n</ul>",
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},
"code": "R64",
"name": "Кахексия",
"icd_name": "Кахексия",
"gender": 0,
"age_min": 0,
"age_max": 100,
"cause": [
3
],
"periodicity": 1,
"slug": "r64_kaheksiya",
"lead": "Крайняя степень истощения организма, для которой характерно резкое исхудание, общая слабость, снижение активности физиологических процессов, расстройства психики",
"description": "Выделяют 2 формы кахексии:\r\nПервичную (экзогенная) — при низком поступлении пищи в организм или в том случае, если ее энергетическая и питательная ценность низки;\r\nВторичную (эндогенная) — является осложнением различных заболеваний.\r\n",
"etiology": "<p><a href=\"/symptom/otkaz_ot_pischi/\" title=\"Перейти на страницу симптома Отказ от пищи\">Голодание</a> или длительное <a href=\"/symptom/snizhenie_appetita/\" title=\"Перейти на страницу симптома Снижение аппетита\">недоедание</a>, поражения пищевода, болезни органов желудочно-кишечного тракта, психогенная <a href=\"/symptom/anoreksiya/\" title=\"Перейти на страницу симптома Анорексия\">анорексия</a>, длительная интоксикация при хронических инфекционных болезнях, амилоидоз, истощающие диффузные заболевания соединительной ткани, тяжелая сердечная недостаточность, злокачественные опухоли, эндокринные заболевания с нарушением обмена энергии, болезни щитовидной железы.</p>",
"pathogenesis": "<p><a href=\"/symptom/kaheksiya/\" title=\"Перейти на страницу симптома Кахексия\">Кахексия</a> приводит не только к упадку сил, она меняет реакцию на лечение и ухудшает переносимость, что фатально сказывается на продолжительности жизни. При потере за полгода не более 5% массы тела, сопровождающейся патологическим отвращением к пище и признаками воспаления, предполагается прекахексия. О системной воспалительной реакции можно говорить при температуре выше 38 °C или ниже 36 °C, тахикардии от 90 ударов, учащённом дыхании, лейкоцитах выше 12 тысяч или ниже 4 тысяч. Только на стадии прекахексии лечением можно остановить потерю мышечной массы, что изменит ближайшую судьбу больного. При потере более 5% массы со всеми указанными признаками говорят о собственно кахексии. Это же состояние возникает у больного с изначально низким весом при утрате только 2% массы тела. Дальнейшее не просто снижение, а форменное <a href=\"/symptom/kaheksiya/\" title=\"Перейти на страницу симптома Кахексия\">истощение</a> с невозможностью встать с постели или из-за слабости проводить в ней более половины дня, безответное применение противоопухолевой терапии — уже рефрактерная <a href=\"/symptom/kaheksiya/\" title=\"Перейти на страницу симптома Кахексия\">кахексия</a>.</p>",
"diagnostics": "<p>Чаще всего диагноз «<a href=\"/symptom/kaheksiya/\" title=\"Перейти на страницу симптома Кахексия\">кахексия</a>» можно установить только по внешнему виду пациента, настолько очевидны проявления кахексии при простом визуальном осмотре. Сбор анамнеза помогает уточнить сроки и стадию заболевания, тяжесть поражения организма. Для более точной оценки степени изменений производят стандартные лабораторные обследования: общий и биохимический анализы крови, общий анализ мочи (при необходимости – бакпосев мочи для исключения сепсиса), лабораторные и инструментальные методы обследования для выявления основного заболевания и его тяжести (рентген, УЗИ, МРТ, КТ, гастроскопия). При необходимости к обследованию могут привлечены и другие специалисты – гастроэнтеролог, кардиолог, гинеколог, невропатолог, психиатр, эндокринолог, онколог.</p>",
"treatment": "<p>Направлено на основное заболевание и должно включать мероприятия по восстановлению питания больного и обеспечению тщательного ухода.</p>\r\n<p> </p>\r\n<p>В рацион вводится пища, богатая белками, жирами, витаминами, предпочтительно употреблять легкоусваиваемые продукты. При нарушениях переваривания и всасывания пищи назначают полиферментные препараты. В тяжелых случаях парентерально вводят растворы глюкозы, электролитов, витамины, белковые гидролизаты, аминокислотные смеси. По показаниям применяют анаболические гормоны.</p>",
"prevention": "<p>Профилактика кахексии сводится к своевременному и адекватному лечению заболеваний, на фоне которых может развиться <a href=\"/symptom/kaheksiya/\" title=\"Перейти на страницу симптома Кахексия\">кахексия</a>. При чрезмерном употреблении алкоголя и приеме наркотиков необходимо лечение от этих зависимостей. </p>",
"clinical_picture": "<p>Резко выраженная <a href=\"/symptom/slabost/\" title=\"Перейти на страницу симптома Слабость\">слабость</a>, утрата трудоспособности, <a href=\"/symptom/anormalnaya_poterya_vesa/\" title=\"Перейти на страницу симптома Анормальная потеря веса\">резкое похудание</a>, признаки обезвоживания, могут наблюдаться безбелковые отеки подкожной клетчатки.</p>\r\n<p> </p>\r\n<p>В тяжелых случаях потеря массы достигает 50 % и более. Кожа бледная или землисто-серая, дряблая, морщинистая. Отмечаются трофические изменения волос и ногтей. Развивается стоматит, нередко выпадают зубы, отмечаются упорные запоры. Понижается половая функция, у женщин наступает аменорея. Уменьшается объем циркулирующей крови, АД обычно снижено.</p>",
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"etiology": "<p>ВИЧ-инфекцию вызывает вирус иммунодефицита человека, относящийся к семейству ретровирусов </p>\r\n<p>ВИЧ – нестойкий вирус. Быстро погибает вне организма носителя, чувствителен к воздействию температуры (снижает инфекционные свойства при температуре 56 °С, погибает через 10 минут при нагревании до 70-80 °С). Хорошо сохраняется в крови и ее препаратах, подготовленных для переливания. Антигенная структура вируса весьма изменчива. </p>\r\n<p>Резервуаром и источником ВИЧ-инфекции является человек: страдающий СПИД и носитель. Вирус содержится в высоких концентрациях в крови, сперме, секрете вагинальных желез и менструальных выделениях. Может выделяться из женского молока, слюны, слезного секрета и ликвора, но эти биологические жидкости представляют меньшую эпидемиологическую опасность. </p>",
"pathogenesis": "<p>ВИЧ поражает клетки крови человека, имеющие на своей поверхности CD4-рецепторы: Т-лимфоциты, макрофаги и дендритные клетки. . Инфицированные вирусом Т-лимфоциты гибнут из-за разрушения вирусом, апоптоза или уничтожения цитотоксическими Т-лимфоцитами. После того, как число CD4+ T-лимфоцитов становится ниже 200 в одном микролитре крови, система клеточного иммунитета перестает защищать организм </p>\r\n<p>Первое время организм компенсирует массовую гибель иммунных клеток, вырабатывая новые, со временем компенсация становится недостаточной, количество лимфоцитов и макрофагов в крови значительно снижается, иммунная система разрушается, организм становится беззащитен как по отношению к экзогенной инфекции, так и к бактериям, населяющим органы и ткани в норме (что ведет к развитию оппортунистических инфекций). Кроме того, нарушается механизм защиты от размножения дефектных бластоцитов - злокачественных клеток. </p>\r\n<p>Заселение вирусом иммунных клеток часто провоцирует различные аутоиммунные состояния, в частности характерны неврологические расстройства в результате аутоиммунного поражения нейроцитов, которые могут развиться даже раньше, чем проявится клиника иммунодефицита. </p>",
"diagnostics": "<p>В настоящее время существуют следующие методы диагностики ВИЧ: косвенные тесты позволяют обнаружить специфические антитела к ВИЧ, которые имеются почти у 100 % ВИЧ-инфицированных; прямые тесты определяют собственно ВИЧ, антигены ВИЧ или нуклеиновые кислоты ВИЧ (вирусная нагрузка). </p>\r\n<p>Вирусную нагрузку(число молекул геномной РНК ВИЧ в мл крови) напрямую связывают со скоростью снижения количества CD4+ лимфоцитов, эта характеристика является важным прогностическим показателем на ранних стадиях заболевания </p>\r\n<p>Для определения антител к ВИЧ, необходимо использовать, по крайней мере, два разных теста: предварительный тест (скрининг-тест) и подтверждающий тест. Большинства современных скрининг-тестов основываются на иммуноферментом анализе (ИФА) или сходных методах; они обладают высокой чувствительностью (до 99 %) и специфичностью (до 99,5 %). </p>\r\n<p>Методы экспресс-анализа основаны на методах реакция агглютинации, ИФА на полимерных мембранах (тест-полоски), иммунологическом фильтрационном анализе и иммунохроматографии. Экспресс-тесты дают результат в течение 15—30 минут. </p>\r\n<p>Подсчёт CD4+ лимфоцитов является критическим параметром в мониторинге ВИЧ-инфекции и позволяет оценивать состояние иммунной системы и склонность к развитию СПИД. </p>",
"treatment": "<p>До настоящего времени не разработано лечения ВИЧ-инфекции, которое могло бы устранить вирус иммунодефицита человека из организма. Современная высокоактивная антиретровирусная терапия замедляет прогрессирование ВИЧ-инфекции и её переход в стадию СПИД, позволяя ВИЧ-инфицированному человеку жить полноценной жизнью. </p>\r\n<p>На сегодняшний день преимущественным этиотропным лечением является назначение препаратов, снижающих репродуктивные способности вируса. Антиретровирусную терапию следует начинать у пациентов с ВИЧ-инфекцией независимо от клинической стадии заболевания, при любом количестве клеток СД4, в том числе всем ВИЧ-инфицированным беременным женщинам. </p>\r\n<p>Лечение ВИЧ-инфицированных включает общеукрепляющие и поддерживающие организм средства (витамины и биологически активные вещества) и методики физиотерапевтической профилактики вторичных заболеваний. </p>",
"prevention": "<p>В настоящее время Всемирная организация здравоохранения проводит общие профилактические мероприятия по снижению заболеваемости ВИЧ-инфекцией по четырем основным направлениям: </p>\r\n<p>просвещение в вопросах безопасности половых отношений, распространение презервативов, лечение заболеваний, передающихся половым путем, пропагандирование культуры половых взаимоотношений; </p>\r\n<p>контроль над изготовлением препаратов из донорской крови; </p>\r\n<p>ведение беременности ВИЧ-инфицированных женщин, обеспечение их медицинской помощи и предоставление им средств химиопрофилактики (в последнем триместре беременности и в родах женщины получают антиретровирусные препараты, которые также на первые три месяца жизни назначаются новорожденным детям); </p>\r\n<p>организация психологической и социальной помощи и поддержки ВИЧ-инфицированных граждан, консультирование. </p>\r\n<p>В настоящее время в мировой практике особое внимание уделяют таким эпидемиологически важным в отношении заболеваемости ВИЧ-инфекцией факторам, как наркомания, беспорядочная половая жизнь. В качестве профилактической меры во многих странах производится бесплатная раздача одноразовых шприцов, метадоновая заместительная терапия. В качестве меры, способствующей снижению половой неграмотности, в учебные программы вводятся обучающие половой гигиене курсы. </p>\r\n<p> </p>\r\n<p> </p>",
"clinical_picture": "<p>Инкубационный период, время от момента заражения ВИЧ до появления симптоматики, называют серологическим окном. Сывороточные реакции на вирус иммунодефицита отрицательны: специфические антитела ещё не определяются. Средняя продолжительность инкубации составляет 12 недель; сроки могут сокращаться до 14 дней при сопутствующих ЗППП, туберкулёзе, общей астении, либо увеличиваться до 10-20 лет. В течение всего периода пациент опасен как источник ВИЧ-инфекции. </p>\r\n<p>Стадия первичных проявлений характеризуется проявлением реакции организма на активную репликацию вируса в виде клиники острой инфекции и иммунной реакции (выработка специфических антител). Вторая стадия может протекать бессимптомно, единственным признаком развивающейся ВИЧ-инфекции будет положительная серологическая диагностика на антитела к вирусу. </p>\r\n<p>Клинические проявления второй стадии протекают по типу острой ВИЧ-инфекции. Начало острое, отмечается у 50-90% пациентов спустя три месяца после момента заражения, зачастую предшествуя формированию ВИЧ-антител. Острая инфекция без вторичных патологий имеет довольно разнообразное течение: могут отмечаться лихорадка, разнообразные полиморфные высыпания на кожных покровах и видимых слизистых оболочках, увеличение лимфатических узлов, боль в горле, увеличение селезенки, диарея. </p>\r\n<p>Острая ВИЧ-инфекция обычно длится от нескольких дней до нескольких месяцев, в среднем 2-3 недели, после чего в подавляющем большинстве случаев переходит в латентную стадию. </p>\r\n<p>Латентная стадия характеризуется постепенным нарастанием иммунодефицита. Гибель иммунных клеток на этой стадии компенсируется их повышенным производством. В это время диагностировать ВИЧ можно с помощью серологических реакций (в крови присутствуют антитела к ВИЧ). Клиническим признаком может быть увеличение нескольких лимфатических узлов из разных, не связанных между собой групп, исключая паховые лимфоузлы. При этом других патологических изменений со стороны увеличенных лимфоузлов (болезненность, изменения окружающих тканей) не отмечается. Латентная стадия может продолжаться от 2-3 лет, до 20 и более. В среднем она длиться 6-7 лет.</p>",
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