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},
"code": "K56.2",
"name": "Заворот кишок",
"icd_name": "Заворот кишок",
"gender": 0,
"age_min": 40,
"age_max": 100,
"cause": [
"0"
],
"periodicity": 1,
"slug": "k56.2_zavorot_kishok",
"lead": "один из видов кишечной непроходимости, при котором происходит закручивание одного из отделов кишечника вокруг своей оси или вокруг оси брыжейки",
"description": "",
"etiology": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Чтобы произошел поворот, должны возникнуть нетипичные, форс-мажорные обстоятельства. Это могут быть внутренние и внешние причины:</span></p>\r\n<ul style=\"margin-top: 0; margin-bottom: 0; padding-inline-start: 48px;\">\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">физические: травма живота, удар об воду, резкий поворот тела, вынужденная непривычная поза, сдавление, сотрясение, подъем тяжестей;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">пищевые:</span><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\"> переедание, особенно после длительного голодания, употребление незнакомой экзотической пищи, большое количество трудноперевариваемой клетчатки в рационе, продолжительный голод;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">органические: спаечные процессы, пупочная грыжа, новообразования, нарушение иннервации кишечника вследствие поражения центральной нервной системы, беременность;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">химические: отравление свинцом или лекарственными препаратами.</span></li>\r\n</ul>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">При анализе данных о пациентах были выявлены факторы, увеличивающие вероятность возникновения перекручивания кишок:</span></p>\r\n<p> </p>\r\n<ul style=\"margin-top: 0; margin-bottom: 0; padding-inline-start: 48px;\">\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">возраст старше 55 лет (заворот сигмовидной кишки часто возникает у пациентов старше 60 лет, ободочной и слепой кишки – после 50 лет);</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">регулярные запоры или поносы;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">малоподвижный образ жизни;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">тяжелый физический труд;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">фанатичная приверженность различным системам голодания для похудения, нерегулярное питание;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">переедание, особенно перед сном.</span></li>\r\n</ul>",
"pathogenesis": "<p> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Перекрут петель кишки вокруг брыжейки вызывает сдавливание сосудов и нервов, из-за чего нарушается питание и иннервация кишечника. Перистальтика сначала усиливается, а затем возникает парез приводящего отдела кишечника - развивается механическая кишечная непроходимость.</span></p>",
"diagnostics": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Подтвердить или опровергнуть кишечную непроходимость по причине заворота кишечника можно при помощи:</span></p>\r\n<ul style=\"margin-top: 0; margin-bottom: 0; padding-inline-start: 48px;\">\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">рентген контрастного исследования с барием;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">ультразвуковой диагностики;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">эндоскопии;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">лапароскопии;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">лабораторной диагностики.</span></li>\r\n</ul>\r\n<p> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Специализированных лабораторных тестов нет, но общий анализ и биохимия крови могут показать глубину интоксикации и воспалительного процесса. </span></p>",
"treatment": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">В начале диагностического поиска пациент может находиться в отделении гастроэнтерологии, однако после постановки диагноза он должен быть переведен в отделение абдоминальной хирургии. Консервативную терапию и подготовку к операции начинают с введения назогастрального зонда для разгрузки кишечника, выведения застойного содержимого и газов. Целью инфузионной терапии является восстановление жидкостного баланса, водно-электролитного равновесия крови, уровня белка. Антибактериальная терапия обязательна, если подозревается перитонит или сепсис – её начинают немедленно после госпитализации больного в стационар, если же данные осложнения исключены – за два часа до операции.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Заворот тонкого кишечника лечится исключительно оперативным путем. Во время операции хирург должен попытаться расправить перекрут петель, при наличии некроза кишок производится сегментарная резекция тонкой кишки, наложение анастомоза тонкой кишки в тонкую или толстую. Если же во время операции выявляется перитонит – накладывается илеостома, через три месяца производится реконструктивная операция. Операция при завороте слепой кишки преследует своей целью проведение гемиколэктомии с наложением межкишечного анастомоза либо илеостомы (при наличии воспалительного экссудата в брюшной полости). У ослабленных больных может быть проведена чрескожная тифлостомия – она позволит улучшить состояние кишечника, стабилизировать пациента.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Терапию заворота сигмовидной кишки предпочтительно начинать с ректороманоскопии или колоноскопии, во время которых в сигмовидную кишку вводят гибкую трубку для разгрузки кишечника. При необходимости после декомпрессии производится радикальная операция, показаниями к которой являются острый заворот сигмовидной кишки, отсутствие эффекта от консервативной терапии. После операции необходимо продолжить массивную антибактериальную и инфузионную терапию, осуществлять наблюдение за пациентом для раннего выявления осложнений: присоединение инфекции, несостоятельность швов на кишке, образование межкишечных абсцессов и свищей, развитие генерализованной инфекции.</span></p>",
"prevention": "<p><span id=\"docs-internal-guid-5394b655-7fff-aecf-39ae-5fc830ccf9b1\"><span style=\"font-size: 7pt; font-family: Verdana; background-color: transparent; font-variant-numeric: normal; font-variant-east-asian: normal; vertical-align: baseline; white-space: pre-wrap;\">Для профилактики требуется контроль назначения и приема психотропных и слабительных средств, препаратов, усиливающих или замедляющих перистальтику кишечника. Количество грубых пищевых волокон в рационе должно быть достаточным, но не избыточным. После оперативных вмешательств в обязательном порядке должна проводиться профилактика образования спаек.</span></span></p>",
"clinical_picture": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Симптомы по степени их появления и нарастания:</span></p>\r\n<ul style=\"margin-top: 0; margin-bottom: 0; padding-inline-start: 48px;\">\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">острая, схваткообразная боль в животе;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">в течение суток боль становится постоянной и труднопереносимой, может дойти до состояния шока;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">асимметрия контуров живота, вздутие;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">тошнота, многократная рвота (может быть с желчью, а в тяжелых случаях с запахом или примесью кала);</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">слабость, бледность, низкое артериальное давление, учащенный пульс;</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">головная боль, головокружение, возможно спутанное сознание (и другие признаки интоксикации);</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">обезвоживание (сухие и потрескавшиеся губы, вязкая слюна, тусклая кожа);</span></li>\r\n<li><span style=\"background-color: transparent; font-family: Verdana; font-size: 7pt; white-space: pre-wrap;\">отсутствие дефекации.</span></li>\r\n</ul>\r\n<p> </p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Патология развивается быстро и состояние больного стремительно ухудшается. Летальный исход возможен в течение 2–3 суток, если не будет оказана экстренная медицинская помощь.</span></p>",
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"etiology": "<p>Основным патогенетическим фактором является повышенная чувствительность кожи к раздражителям, по-видимому, за счет разрастания нервных окончаний, и предрасположенность к гиперплазии эпидермиса в ответ на механическую травму. В возникновении заболевания важную роль играют функциональные нарушения нервной и эндокринной систем, аллергическое состояние организма, заболевания желудочно-кишечного тракта. Указывают также на наследственную предрасположенность.</p>",
"pathogenesis": "<p>Патофизиологические процессы, лежащие в основе расстройства, неизвестны, однако могут включать нарушение восприятия и обработки нервной системой ощущения зуда. Кожа, склонная к развитию экзематозных заболеваний (например, атопического дерматита), более склонна к лихенификации.</p>",
"diagnostics": "<p>Диагноз устанавливается при осмотре. Полностью сформировавшаяся бляшка имеет внешнюю зону, состоящую из дискретных коричневатых папул, и центральной зоны, представленной сливающимися папулами, покрытыми чешуйками. К клинически похожим состояниям относятся микоз гладкой кожи, красный плоский лишай и псориаз; простой хронический лишай можно отличить от этих состояний при помощи микроскопического исследования соскоба во влажном препарате с гидроксидом калия и биопсии.</p>",
"treatment": "<p style=\"display: inline !important;\"> </p>\r\n<p>Первичная терапия пациента заключается в его обучении и разъяснении эффектов расчесывания и трения. Вторичное лечение состоит в применении глюкокортикостероидных препаратов (например, триамцинолона ацетонида, флуоцинонида); хирургической пленки, пропитанной флурандренолидом (накладывается утром и меняется на новую пленку вечером, может оказаться более предпочтительной, поскольку окклюзия предотвращает расчесывание). Маленькие зоны поражения можно обколоть (внутриочаговые инъекции) длительно действующим глюкокортикостероидным препаратом, например триамценолона ацетонидом 2,5 мг/мл (разведенным в физиологическом растворе) в дозе 0,3 мл/см2 площади очага поражения; лечение можно повторять каждую 3–4-ю неделю. Пероральный прием антигистаминных препаратов, блокирующих H1-рецепторы, может оказать положительный эффект. Мази также могут быть действенными. Крем с капсаицином местного применения может быть также полезным, но жжение в начале может сделать эту терапию неприемлемой для пациентов.<br /> <br /> Ключевые моменты</p>\r\n<ul>\r\n<li>Хроническое шелушение приводит к дальнейшему зуду, создавая порочный круг.</li>\r\n<li>Зуд, сухость, шелушение, гиперпигментированность, лихениеновые бляшки возникают неправильной овальной или угловой формы на ногах, руках, шее и верхней части туловища, а иногда в аногенитальной области.</li>\r\n<li>Диагноз устанавливается на основе результатов клинических исследований, но исследование влажного препарата с гидроксидом калия и биопсия могут помочь в дифференциальной диагностике.</li>\r\n<li>Пациенты должны быть осведомлены о замкнутом круге, который начинается с царапин и продолжается зудом; кортикостероиды местного применения и антигистаминные препараты помогают контролировать зуд.</li>\r\n</ul>",
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"clinical_picture": "<p align=\"justify\">Заболевание начинается с зуда кожи. Симптомы простого хронического лишая локализуются преимущественно на задней и боковой поверхностях шеи, в подколенных и локтевых сгибах, аногенитальной области, на внутренней поверхности бедер, в межъягодичных складках. Но очаги могут появляться и на других участках кожного покрова, в том числе на волосистой части головы. В начале кожа в местах зуда внешне не изменена. Со временем под влиянием расчесов появляются полигональные папулы плотной консистенции, местами покрытые муковидными чешуйками. Папулы сливаются и образуют бляшки овальной или округлой формы, которые имеют цвет от розового до буровато-красного. Кожа утолщается, становится грубой, выражен кожный рисунок (лихенификация). На высоте развития заболевания в очаге различают три зоны. Периферическая, или наружная, зона пигментации окружает очаг поражения в виде пояса и обычно пи наружная, ни внутренняя границы неясны. Средняя, папулезная, зона состоит из узелковых высыпаний бледно-розового, сероватого или желтоватого цвета, величиной от булавочной головки до небольшой чечевицы. Папулы неправильной формы и нерезко ограничены, почти не возвышаются над окружающей кожей. Их поверхность утолщена, гладкая и в результате расчесов часто покрыта кровянистой корочкой. Внутренняя зона характеризуется выраженной инфильтрацией кожи. Нередко эта зона является единственным проявлением в клинической картине болезни.</p>\r\n<p align=\"justify\">В практике дерматовенеролога часто встречаются следующие атипичные и редкие разновидности ограниченного нейродермита:</p>\r\n<p align=\"justify\">Депигментированный нейродермит. При длительном течении ограниченного нейродермита возникают вторичные гипопигментации (витилигоподобные изменения). Считают, что они появляются в результате расчесов. При этом часто создается впечатление, что имеется сочетание двух процессов - нейродермита и витилиго.</p>\r\n<p>Гипертрофический (бородавчатый) нейродермит. При данной форме на фоне типичной клинической картины ограниченного нейродермита имеются отдельные узелковые и даже узловатые высыпания, весьма сходные с таковыми при узловатой почесухе. Такие очаги возникают преимущественно на внутренних поверхностях бедер, по могут локализоваться и па любых других участках.</p>\r\n<p align=\"justify\">В результате сильного зуда кожи головы выпадают волосы, кожа истончается, блестит, но не атрофична, процесс не связан с фолликулярным аппаратом. Такая форма заболевания получила название декальвирующий нейродермит.</p>",
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"etiology": "<p>Основным патогенетическим фактором является повышенная чувствительность кожи к раздражителям, по-видимому, за счет разрастания нервных окончаний, и предрасположенность к гиперплазии эпидермиса в ответ на механическую травму. В возникновении заболевания важную роль играют функциональные нарушения нервной и эндокринной систем, аллергическое состояние организма, заболевания желудочно-кишечного тракта. Указывают также на наследственную предрасположенность.</p>",
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"diagnostics": "<p>Диагноз устанавливается при осмотре. Полностью сформировавшаяся бляшка имеет внешнюю зону, состоящую из дискретных коричневатых папул, и центральной зоны, представленной сливающимися папулами, покрытыми чешуйками. К клинически похожим состояниям относятся микоз гладкой кожи, красный плоский лишай и псориаз; простой хронический лишай можно отличить от этих состояний при помощи микроскопического исследования соскоба во влажном препарате с гидроксидом калия и биопсии.</p>",
"treatment": "<p style=\"display: inline !important;\"> </p>\r\n<p>Первичная терапия пациента заключается в его обучении и разъяснении эффектов расчесывания и трения. Вторичное лечение состоит в применении глюкокортикостероидных препаратов (например, триамцинолона ацетонида, флуоцинонида); хирургической пленки, пропитанной флурандренолидом (накладывается утром и меняется на новую пленку вечером, может оказаться более предпочтительной, поскольку окклюзия предотвращает расчесывание). Маленькие зоны поражения можно обколоть (внутриочаговые инъекции) длительно действующим глюкокортикостероидным препаратом, например триамценолона ацетонидом 2,5 мг/мл (разведенным в физиологическом растворе) в дозе 0,3 мл/см2 площади очага поражения; лечение можно повторять каждую 3–4-ю неделю. Пероральный прием антигистаминных препаратов, блокирующих H1-рецепторы, может оказать положительный эффект. Мази также могут быть действенными. Крем с капсаицином местного применения может быть также полезным, но жжение в начале может сделать эту терапию неприемлемой для пациентов.<br /> <br /> Ключевые моменты</p>\r\n<ul>\r\n<li>Хроническое шелушение приводит к дальнейшему зуду, создавая порочный круг.</li>\r\n<li>Зуд, сухость, шелушение, гиперпигментированность, лихениеновые бляшки возникают неправильной овальной или угловой формы на ногах, руках, шее и верхней части туловища, а иногда в аногенитальной области.</li>\r\n<li>Диагноз устанавливается на основе результатов клинических исследований, но исследование влажного препарата с гидроксидом калия и биопсия могут помочь в дифференциальной диагностике.</li>\r\n<li>Пациенты должны быть осведомлены о замкнутом круге, который начинается с царапин и продолжается зудом; кортикостероиды местного применения и антигистаминные препараты помогают контролировать зуд.</li>\r\n</ul>",
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"clinical_picture": "<p align=\"justify\">Заболевание начинается с зуда кожи. Симптомы простого хронического лишая локализуются преимущественно на задней и боковой поверхностях шеи, в подколенных и локтевых сгибах, аногенитальной области, на внутренней поверхности бедер, в межъягодичных складках. Но очаги могут появляться и на других участках кожного покрова, в том числе на волосистой части головы. В начале кожа в местах зуда внешне не изменена. Со временем под влиянием расчесов появляются полигональные папулы плотной консистенции, местами покрытые муковидными чешуйками. Папулы сливаются и образуют бляшки овальной или округлой формы, которые имеют цвет от розового до буровато-красного. Кожа утолщается, становится грубой, выражен кожный рисунок (лихенификация). На высоте развития заболевания в очаге различают три зоны. Периферическая, или наружная, зона пигментации окружает очаг поражения в виде пояса и обычно пи наружная, ни внутренняя границы неясны. Средняя, папулезная, зона состоит из узелковых высыпаний бледно-розового, сероватого или желтоватого цвета, величиной от булавочной головки до небольшой чечевицы. Папулы неправильной формы и нерезко ограничены, почти не возвышаются над окружающей кожей. Их поверхность утолщена, гладкая и в результате расчесов часто покрыта кровянистой корочкой. Внутренняя зона характеризуется выраженной инфильтрацией кожи. Нередко эта зона является единственным проявлением в клинической картине болезни.</p>\r\n<p align=\"justify\">В практике дерматовенеролога часто встречаются следующие атипичные и редкие разновидности ограниченного нейродермита:</p>\r\n<p align=\"justify\">Депигментированный нейродермит. При длительном течении ограниченного нейродермита возникают вторичные гипопигментации (витилигоподобные изменения). Считают, что они появляются в результате расчесов. При этом часто создается впечатление, что имеется сочетание двух процессов - нейродермита и витилиго.</p>\r\n<p>Гипертрофический (бородавчатый) нейродермит. При данной форме на фоне типичной клинической картины ограниченного нейродермита имеются отдельные узелковые и даже узловатые высыпания, весьма сходные с таковыми при узловатой почесухе. Такие очаги возникают преимущественно на внутренних поверхностях бедер, по могут локализоваться и па любых других участках.</p>\r\n<p align=\"justify\">В результате сильного зуда кожи головы выпадают волосы, кожа истончается, блестит, но не атрофична, процесс не связан с фолликулярным аппаратом. Такая форма заболевания получила название декальвирующий нейродермит.</p>",
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