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},
"code": "E46",
"name": "Белково-энергетическая недостаточность неуточненная",
"icd_name": "Белково-энергетическая недостаточность неуточненная",
"gender": 0,
"age_min": 0,
"age_max": 100,
"cause": [
3,
0
],
"periodicity": 1,
"slug": "e46_belkovo-energeticheskaya_nedostatochnost_neutochnennaya",
"lead": "Состояние питания, при котором дисбаланс энергии, белков и других питательных веществ ведет к измеримым нежелательным эффектам на ткани, функции и клинические исходы.",
"description": "При недостаточном поступлении белков и энергии уменьшаются безжировая масса тела и количество жировой ткани, причем одно из этих изменений может быть более выраженным.",
"etiology": "<p>Белково-энергетическая недостаточность может быть первичной или вторичной. Первичная белково-энергетическая недостаточность обусловлена неадекватным поступлением питательных веществ, а вторичная белково-энергетическая недостаточность является следствием различных расстройств или приема лекарственных препаратов, которые препятствуют использованию питательных веществ.</p>",
"pathogenesis": "",
"diagnostics": "",
"treatment": "<p>При легкой и умеренной белково-энергетической недостаточности следует устранить возможные причины этого состояния. Суточный приход белков и энергии увеличивают (в соответствии с идеальным весом) для ликвидации их дефицита. Всем больным назначают поливитамины. Проводят также лечение и профилактику дефицита минеральных веществ (в том числе микроэлементов) для предупреждения угрожающих жизни гипокалиемии, гипомагниемии, гипофосфатемии и т. д. Если больной способен принимать пищу и глотать, достаточно самостоятельного питания. При снижении аппетита или в отсутствие зубов дополнительно назначают жидкие питательные смеси для самостоятельного или зондового питания.</p>\r\n<full></full>\r\n<p>При тяжелой белково-энергетической недостаточности требуется более срочное вмешательство. Лечение таких больных затруднено по нескольким причинам:</p>\r\n<ul>\r\n<li>Заболевания, вызвавшие белково-энергетическую недостаточность, протекают тяжело, их труднее лечить. Иногда азотистый баланс можно восстановить только после излечения инфекции и устранения лихорадки.</li>\r\n<li>Белково-энергетическая недостаточность сама по себе может препятствовать излечению вызвавшего ее тяжелого заболевания. В таких случаях необходимо как можно раньше начать зондовое или парентеральное питание.</li>\r\n<li>Поступление пищи через ЖКТ способствует поносу из-за атрофии слизистой и дефицита ферментов кишечника и поджелудочной железы. При этом может быть показано полное парентеральное питание.</li>\r\n<li>Следует устранить сопутствующий дефицит других компонентов пищи (витаминов, незаменимых минеральных веществ, микроэлементов).</li>\r\n</ul>\r\n<p>У взрослых восстановление питательного статуса наступает медленно и не всегда полностью; у детей выздоровление происходит в течение 3-4 мес. Во всех случаях необходимы образовательные и реабилитационные программы, а также меры психологической и социальной поддержки.</p>",
"prevention": "",
"clinical_picture": "<p>Легкая и умеренная белково-энергетическая недостаточность. У детей отсутствует прибавка веса и роста. У взрослых отмечается снижение веса, хотя при отеках или ожирении оно может быть не столь заметным. Уменьшаются толщина кожной складки над трехглавой мышцей плеча и мышечная масса в области плеча.</p>\r\n<p>В отсутствие болезней почек чувствительным показателем белковой недостаточности служит отношение суточной экскреции креатинина к росту (показатель измеряют еженедельно). Снижаются уровни альбумина, трансферрина и транстиретина (преальбумина) в крови. Уровень Т3 снижается, а реверсивного Т3 - повышается. Замедляется метаболизм. Возможны лимфопения и нарушение толерантности к глюкозе . Размеры сердца уменьшены .</p>\r\n<p>Тяжелая белково-энергетическая недостаточность. Тяжелая белково- энергетическая недостаточность сопровождается более выраженными изменениями клинических и лабораторных показателей. При физикальном исследовании обнаруживают западение межреберий, атрофию височных мышц и атрофию мышц конечностей. Подкожная клетчатка атрофирована или отсутствует. Характерны <a href=\"/symptom/apatiya/\" title=\"Перейти на страницу симптома Апатия\">апатия</a>, быстрая <a href=\"/symptom/utomlyaemost/\" title=\"Перейти на страницу симптома Утомляемость\">утомляемость</a>, чувство холода, депигментация кожи и депигментация волос, заострившиеся черты лица; кожа сухая, покрыта трещинами. В запущенных случаях образуются пролежни, кожа изъязвляется. АД снижено, также как и температура тела, пульс ослаблен. Нарушаются функции всех органов и систем.</p>",
"image": null,
"image_alt": null,
"standard_type": 1,
"danger": 52,
"published": 1,
"parent": null,
"block_rubric": 49,
"standards": []
},
{
"id": 6251,
"symptoms": [
{
"id": 2779,
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{
"name": "резкое похудание"
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{
"name": "резкая потеря веса"
},
{
"name": "сильное похудание"
},
{
"name": "Снижение массы тела"
},
{
"name": "похудение"
}
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"slug": "anormalnaya_poterya_vesa",
"lead": "Если вес снизился аномально или резко, обратитесь к врачу",
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"danger": false
},
{
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"name": "локальная гипертермия "
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{
"name": "горячая на ощупь"
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{
"name": "отек на коже"
},
{
"name": "припухлость"
},
{
"name": "уплотнение"
},
{
"name": "Локализованный отек"
}
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"treatment": "<p>При легкой и умеренной белково-энергетической недостаточности следует устранить возможные причины этого состояния. Суточный приход белков и энергии увеличивают (в соответствии с идеальным весом) для ликвидации их дефицита. Всем больным назначают поливитамины. Проводят также лечение и профилактику дефицита минеральных веществ (в том числе микроэлементов) для предупреждения угрожающих жизни гипокалиемии, гипомагниемии, гипофосфатемии и т. д. Если больной способен принимать пищу и глотать, достаточно самостоятельного питания. При снижении аппетита или в отсутствие зубов дополнительно назначают жидкие питательные смеси для самостоятельного или зондового питания.</p>\r\n<full></full>\r\n<p>При тяжелой белково-энергетической недостаточности требуется более срочное вмешательство. Лечение таких больных затруднено по нескольким причинам:</p>\r\n<ul>\r\n<li>Заболевания, вызвавшие белково-энергетическую недостаточность, протекают тяжело, их труднее лечить. Иногда азотистый баланс можно восстановить только после излечения инфекции и устранения лихорадки.</li>\r\n<li>Белково-энергетическая недостаточность сама по себе может препятствовать излечению вызвавшего ее тяжелого заболевания. В таких случаях необходимо как можно раньше начать зондовое или парентеральное питание.</li>\r\n<li>Поступление пищи через ЖКТ способствует поносу из-за атрофии слизистой и дефицита ферментов кишечника и поджелудочной железы. При этом может быть показано полное парентеральное питание.</li>\r\n<li>Следует устранить сопутствующий дефицит других компонентов пищи (витаминов, незаменимых минеральных веществ, микроэлементов).</li>\r\n</ul>\r\n<p>У взрослых восстановление питательного статуса наступает медленно и не всегда полностью; у детей выздоровление происходит в течение 3-4 мес. Во всех случаях необходимы образовательные и реабилитационные программы, а также меры психологической и социальной поддержки.</p>",
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"clinical_picture": "<p>Легкая и умеренная белково-энергетическая недостаточность. У детей отсутствует прибавка веса и роста. У взрослых отмечается снижение веса, хотя при отеках или ожирении оно может быть не столь заметным. Уменьшаются толщина кожной складки над трехглавой мышцей плеча и мышечная масса в области плеча.</p>\r\n<p>В отсутствие болезней почек чувствительным показателем белковой недостаточности служит отношение суточной экскреции креатинина к росту (показатель измеряют еженедельно). Снижаются уровни альбумина, трансферрина и транстиретина (преальбумина) в крови. Уровень Т3 снижается, а реверсивного Т3 - повышается. Замедляется метаболизм. Возможны лимфопения и нарушение толерантности к глюкозе . Размеры сердца уменьшены .</p>\r\n<p>Тяжелая белково-энергетическая недостаточность. Тяжелая белково- энергетическая недостаточность сопровождается более выраженными изменениями клинических и лабораторных показателей. При физикальном исследовании обнаруживают западение межреберий, атрофию височных мышц и атрофию мышц конечностей. Подкожная клетчатка атрофирована или отсутствует. Характерны <a href=\"/symptom/apatiya/\" title=\"Перейти на страницу симптома Апатия\">апатия</a>, быстрая <a href=\"/symptom/utomlyaemost/\" title=\"Перейти на страницу симптома Утомляемость\">утомляемость</a>, чувство холода, депигментация кожи и депигментация волос, заострившиеся черты лица; кожа сухая, покрыта трещинами. В запущенных случаях образуются пролежни, кожа изъязвляется. АД снижено, также как и температура тела, пульс ослаблен. Нарушаются функции всех органов и систем.</p>",
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"description": "У каждого человека в норме уровень содержания глюкозы в крови должен быть от 3,3 до 5,5 ммоль/л. Конечно, глюкоза - очень важная постоянная гомеостаза. При расстройствах углеводного обмена (гипогликемия) в организме происходят необратимые изменения, которые связаны с деятельностью центральной нервной системы.",
"etiology": "<p>Основными причинами нарушений процессов, связанных с углеводным обменом, могут стать:</p>\r\n<ul>\r\n<li>Нарушение фосфорилирования глюкозы в стенке кишечника (обычно это происходит в том случае, если воспаляется кишечная стенка, при отравлениях флоридзином и монойодацетагом).</li>\r\n<li>Нарушение нервно-гормональной регуляции.</li>\r\n<li>Нарушение углеводной функции печени.</li>\r\n<li>Нарушение нейрогормональной регуляции углеводного обмена.</li>\r\n</ul>",
"pathogenesis": "<p>При нарушении разных видов углеводного обмена наблюдается снижение глюкокиназы. Возрастает активность глюко-фосфатазы, следовательно, глюкофосфат поступает в кровь в виде глюкозы. Через мембраны клеток глюкоза практически не проходит, вследствие чего она не способна усваиваться тканями. Ускоряется глюконеогенез (образование глюкозы из пирувата, лактата, амиокислот жирных веществ и др.).</p>",
"diagnostics": "<p>Чтобы правильно диагностировать заболевание, вам необходимо обратиться к врачу, который предложит вам пройти следующие анализы и обследования:</p>\r\n<ul>\r\n<li>В первую очередь нужно сдать анализ крови.</li>\r\n<li>Затем биохимический анализ крови.</li>\r\n<li>Сделать глюкозотолерантный тест.</li>\r\n<li>Липидный профиль.</li>\r\n<li>Гликированный гемоглобин.</li>\r\n<li>Определить уровень секреции инсулина, а также С-пептида.</li>\r\n<li>Провести иммунологические анализы.</li>\r\n<li>Проверить, расположены ли вы к возникновению развития нарушений углеродного обмена вследствие наследственности.</li>\r\n<li>Диагностика нефропатии.</li>\r\n<li>ЭКГ и УЗИ.</li>\r\n<li>Сканирование ультразвуком сосудов.</li>\r\n</ul>\r\n<p>Врач, который специализируется на нарушениях углеводного обмена, – эндокринолог.</p>",
"treatment": "<p>Для восстановления углеводного обмена можно воспользоваться медикаментозным лечением – препаратами, которые снижают сахар в крови. Это бигуаниды, ингибиторы альфа-глюкозидаза. Однако препараты не принесут пользы, если вы не будете соблюдать правильный режим жизни. Сюда входят: правильное питание, физические нагрузки, сахароснижающая терапия.</p>",
"prevention": "<p>В связи с нарушением обмена веществ могут развиться довольно сложные и серьезные заболевания, поэтому необходимо предпринять все меры для того, чтобы нарушения не начали развиваться. В первую очередь необходимо сбалансированно и правильно питаться. Это основной залог здоровья. Также существует множество витаминных препаратов, биологических пищевых добавок. Если вы – сладкоежка, от продуктов, содержащих много глюкозы, придется отказаться. Оптимальное употребление воды – 2 л в день.</p>",
"clinical_picture": "<p>Человек при пониженном уровне глюкозы в крови чувствует общую слабость, быстро утомляется, ему тяжело выполнять обычную ежедневную работу. При сильных отклонениях от нормы начинаются судороги, помутнения сознания. Самый страшный симптом этого заболевания – гликемическая кома.</p>",
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"description": "У каждого человека в норме уровень содержания глюкозы в крови должен быть от 3,3 до 5,5 ммоль/л. Конечно, глюкоза - очень важная постоянная гомеостаза. При расстройствах углеводного обмена (гипогликемия) в организме происходят необратимые изменения, которые связаны с деятельностью центральной нервной системы.",
"etiology": "<p>Основными причинами нарушений процессов, связанных с углеводным обменом, могут стать:</p>\r\n<ul>\r\n<li>Нарушение фосфорилирования глюкозы в стенке кишечника (обычно это происходит в том случае, если воспаляется кишечная стенка, при отравлениях флоридзином и монойодацетагом).</li>\r\n<li>Нарушение нервно-гормональной регуляции.</li>\r\n<li>Нарушение углеводной функции печени.</li>\r\n<li>Нарушение нейрогормональной регуляции углеводного обмена.</li>\r\n</ul>",
"pathogenesis": "<p>При нарушении разных видов углеводного обмена наблюдается снижение глюкокиназы. Возрастает активность глюко-фосфатазы, следовательно, глюкофосфат поступает в кровь в виде глюкозы. Через мембраны клеток глюкоза практически не проходит, вследствие чего она не способна усваиваться тканями. Ускоряется глюконеогенез (образование глюкозы из пирувата, лактата, амиокислот жирных веществ и др.).</p>",
"diagnostics": "<p>Чтобы правильно диагностировать заболевание, вам необходимо обратиться к врачу, который предложит вам пройти следующие анализы и обследования:</p>\r\n<ul>\r\n<li>В первую очередь нужно сдать анализ крови.</li>\r\n<li>Затем биохимический анализ крови.</li>\r\n<li>Сделать глюкозотолерантный тест.</li>\r\n<li>Липидный профиль.</li>\r\n<li>Гликированный гемоглобин.</li>\r\n<li>Определить уровень секреции инсулина, а также С-пептида.</li>\r\n<li>Провести иммунологические анализы.</li>\r\n<li>Проверить, расположены ли вы к возникновению развития нарушений углеродного обмена вследствие наследственности.</li>\r\n<li>Диагностика нефропатии.</li>\r\n<li>ЭКГ и УЗИ.</li>\r\n<li>Сканирование ультразвуком сосудов.</li>\r\n</ul>\r\n<p>Врач, который специализируется на нарушениях углеводного обмена, – эндокринолог.</p>",
"treatment": "<p>Для восстановления углеводного обмена можно воспользоваться медикаментозным лечением – препаратами, которые снижают сахар в крови. Это бигуаниды, ингибиторы альфа-глюкозидаза. Однако препараты не принесут пользы, если вы не будете соблюдать правильный режим жизни. Сюда входят: правильное питание, физические нагрузки, сахароснижающая терапия.</p>",
"prevention": "<p>В связи с нарушением обмена веществ могут развиться довольно сложные и серьезные заболевания, поэтому необходимо предпринять все меры для того, чтобы нарушения не начали развиваться. В первую очередь необходимо сбалансированно и правильно питаться. Это основной залог здоровья. Также существует множество витаминных препаратов, биологических пищевых добавок. Если вы – сладкоежка, от продуктов, содержащих много глюкозы, придется отказаться. Оптимальное употребление воды – 2 л в день.</p>",
"clinical_picture": "<p>Человек при пониженном уровне глюкозы в крови чувствует общую слабость, быстро утомляется, ему тяжело выполнять обычную ежедневную работу. При сильных отклонениях от нормы начинаются судороги, помутнения сознания. Самый страшный симптом этого заболевания – гликемическая кома.</p>",
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}
]
}