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"code": "B19",
"name": "Вирусный гепатит неуточненный",
"icd_name": "Вирусный гепатит неуточненный",
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"cause": [
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"slug": "b19_virusnyy_gepatit_neutochnennyy",
"lead": "Нарушение здоровья, относящееся к группе вирусный гепатит",
"description": "Вирусные гепатиты – группа инфекционных заболеваний, вызываемых первично-гепатотропными вирусами, характеризующихся преимущественным поражением печени.",
"etiology": "<p>Гепатит может быть вызван многими причинами, но наиболее часто гепатит вызывается вирусами. На данный момент известно большое количество вирусов, способных вызвать вирусный гепатит: вирус гепатита A, вирус гепатита B, вирус гепатита C, вирус гепатита D, вирус гепатита E, вирус гепатита F и вирус гепатита G, вирусы гепатитов TTV и SEN, цитомегаловирус, вирус Эпштейна — Барр, ВИЧ и другие. Некоторые вирусы, вызывающие вирусный гепатит, недостаточно изучены.</p>\r\n<p>Известны также вирусы гепатитов животных:</p>\r\n<ul>\r\n<li>собак (аденовирус),</li>\r\n<li>мышей (коронавирус),</li>\r\n<li>уток (энтеровирус) и, вероятно,</li>\r\n<li>обезьян (пегивирус).</li>\r\n</ul>\r\n<p>Кроме того, гепатит может вызываться такими вирусами, как вирус желтой лихорадки, герпесвирусы, вирус краснухи, вирусы Коксаки, вирус лихорадки Ласса, вирусы лихорадок Марбург-Эбола и другими.</p>",
"pathogenesis": "<p>Вирусы гепатита попадают в организм разными путями: гепатит А и Е — через загрязненные пищевые продукты и воду; гепатит В — через небезопасную кровь и другие жидкости организма; гепатит С — в основном, через зараженную кровь и гепатит D — в качестве дополнительной инфекции при наличии гепатита В.</p>\r\n<p>Вирусы обладают тропностью к ткани печени, при этом они вызывают острое ее воспаление, повреждение и разрушение печеночных клеток (гепатоцитов), воспалительный отек и снижение функциональной деятельности органа. Недостаточность желчеобразующая функции печени является первопричиной повышенного накопления билирубина в крови (билирубинемии) и, как следствие, желтухи. Поскольку в тканях печени нет болевых рецепторных зон, болевой синдром редко выражен и связан с увеличением печени, растяжении ее хорошо иннервированной капсулы и воспалительными процессами в желчном пузыре.</p>\r\n<p>Хроническое воспаление, как правило, развивается вследствие нелеченного или недостаточно леченного острого гепатита. Нередко хронический гепатит постепенно переходит в цирроз печени.</p>",
"diagnostics": "<p><strong></strong></p>\r\n<p>Диагностика гепатитов осуществляется на основании анамнеза, клинических данных, данных физикального осмотра, функциональных и лабораторных исследований.</p>\r\n<p> </p>\r\n<p>Лабораторные исследования включают: </p>\r\n<ul>\r\n<li>Биохимический анализ крови (повышение уровня печеночно-клеточных ферментов – АЛТ, АСТ; повышение общего билирубина, изменение соотношения прямой и непрямой фракций билирубина);</li>\r\n<li>Общий анализ мочи – появление в моче билирубина, накопление уробилина;</li>\r\n<li>Коагулограмма – оценка свертывающей системы крови;</li>\r\n<li>Тимоловая и сулемовая пробы;</li>\r\n<li>ПЦР-диагностика крови – выявление генома (РНК или ДНК) вируса;</li>\r\n<li>ИФА – выявление в сыворотке крови специфических антител (иммуноглобулинов класса М)</li>\r\n</ul>\r\n<p> </p>\r\n<p>Инструментальные методы диагностики включают:</p>\r\n<ul>\r\n<li>УЗИ органов брюшной полости – отмечают увеличение печени и изменение ее звуковой проницаемости и, кроме этого, отмечают увеличение селезенки и, возможно, расширение полой вены;</li>\r\n<li>Реогепатография(исследование печеночного кровотока), </li>\r\n<li>Гепатохолецистосцинтиография(радиоизотопное исследование желчевыводящих путей), </li>\r\n<li>Пункционная биопсия печени.</li>\r\n</ul>\r\n<p> </p>",
"treatment": "<p>Лечение острого гепатита обязательно проводится в стационаре.</p>\r\n<p>Назначается:</p>\r\n<p>прописывается диета №5, полупостельный режим (при тяжелом течении – постельный);</p>\r\n<p> </p>\r\n<p>Этиотропная терапия</p>\r\n<ul>\r\n<li>Интерферон-а</li>\r\n<li>Индукторы интерферона</li>\r\n<li>Ламивудин</li>\r\n</ul>\r\n<p>Патогенетическая и посиндромная терапия:</p>\r\n<ul>\r\n<li>Интенсивная дезинтоксикационная инфузионная терапия, при необходимости экстракорпоральная детоксикация;</li>\r\n<li>Желчегонные препараты или тюбажи по Демьянову;</li>\r\n<li>Ферментные препараты;</li>\r\n<li>Гепатопротективные препараты;</li>\r\n<li>Глюкокортикоиды;</li>\r\n<li>Ингибиторы протеолиза;</li>\r\n<li>Поливитамины, витамино-минеральные комплексы, фитотерапия.</li>\r\n</ul>\r\n<p>Хорошие результаты по улучшению общего состояния при выраженной гипоксии дает кислородотерапия, оксигенобаротерапия. Если присутствуют признаки геморрагического диатеза, назначают витамин К внутривенно.</p>",
"prevention": "<p>Первичная профилактика вирусных гепатитов – соблюдение гигиенических предписаний, осуществление санитарно-эпидемических мер, санитарный надзор над предприятиями, могущими стать очагом распространения инфекции, вакцинация. Профилактикой других форм гепатитов является избегание действия гепатотравмирующих факторов – алкоголя, лекарственных средств, токсических веществ.</p>\r\n<p>Вторичная профилактика хронического гепатита заключается в соблюдении диеты, режима, врачебных рекомендаций, регулярном прохождении обследования, контроле клинических показателей крови. Больным рекомендовано регулярное санаторно-курортное лечение, водолечение.</p>\r\n<p> </p>\r\n<p> </p>",
"clinical_picture": "<p>Выделяют две основные формы клинического течения гепатитов: острую и хроническую.</p>\r\n<p>Острая форма течения наиболее характерна для гепатитов вирусной природы. Иногда начало гепатита напоминает грипп: с повышением температуры тела, головной болью, общим недомоганием, ломотой в теле. Как правило, это — так называемая «маска» начинающегося вирусного гепатита, характеризующаяся слабостью.</p>\r\n<p>При острой форме развития гепатита наблюдается заметное ухудшение общего состояния больного, развития признаков общей интоксикации организма и нарушения функции печени (повышение температуры тела, в ряде случаев развитие желтухи и др.), а также повышение уровня печеночных ферментов (трансаминаз) и общего билирубина крови. Моча темнеет, при выраженных нарушениях синтеза желчных кислот кал теряет цвет, становится беловато-глинистым.</p>\r\n<p>Больные могут отмечать такие симптомы, как кожный зуд, возникновение на коже красных точек – петехий, брадикардию, невротические симптомы.</p>\r\n<p>Острый гепатит, как правило, заканчивается полным выздоровлением больного, однако в некоторых случаях наблюдается переход острого течения болезни в хроническое.</p>\r\n<p>Клиническая картина при хроническом гепатите бедная, заболевание долгое время протекает бессимптомно. Хронический гепатит характеризуется постепенным развитием следующих клинических синдромов:</p>\r\n<ul>\r\n<li>астеновегетативный (слабость, повышенная утомляемость, расстройства сна, психическая лабильность, головные боли – обусловлен интоксикацией организма в связи с нарастающей печеночной недостаточностью;</li>\r\n<li>диспепсический (тошнота, иногда - рвота, понижение аппетита, метеоризм, поносы, чередующиеся с запорами, отрыжка горечью, неприятный привкус во рту) связан с нарушениями пищеварения в связи с недостаточностью выработки печенью необходимых для пищеварения ферментов и желчных кислот);</li>\r\n<li>болевой синдром (боль постоянного, ноющего характера локализуется в правом подреберье, усиливается при физических нагрузках и после резких нарушений диеты) – может отсутствовать или выражаться в умеренном ощущении тяжести в эпигастрии;</li>\r\n<li>субфебрилитет (умеренное повышение температуры до 37,3 – 37,5 градусов может держаться по нескольку недель);</li>\r\n<li>телеангиэктазии (сосудистые звездочки на коже) на шее, лице, плечах;</li>\r\n<li>геморрагический (петехии, склонность к образованию синяков и кровоподтеков, носовые, геморроидальные, маточные кровотечения) связан с понижением свертываемости крови вследствие недостаточного синтеза факторов свертываемости в клетках печени;</li>\r\n<li>желтуха – наиболее известный симптом, возникает, когда билирубин, не переработанный в печени, попадает в кровь и придаёт коже характерный желтоватый оттенок. Однако часто бывают и безжелтушные формы гепатита;</li>\r\n<li>увеличение печени, может сочетаться с увеличением селезенки.</li>\r\n</ul>\r\n<p> </p>",
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"code": "B21",
"name": "Болезнь, вызванная вирусом иммунодефицита человека [ВИЧ], проявляющаяся в виде злокачественных новообразований",
"icd_name": "Болезнь, вызванная вирусом иммунодефицита человека [ВИЧ], проявляющаяся в виде злокачественных новообразований",
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2,
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"lead": "Нарушение здоровья, относящееся к группе болезнь, вызванная вирусом иммунодефицита человека [вич]",
"description": "ВИЧ-инфекция представляет собой заболевание, вызываемое вирусом иммунодефицита человека, характеризующееся синдромом приобретенного иммунодефицита, способствующего возникновению вторичных инфекций и злокачественных образований в связи с глубоким угнетением защитных ",
"etiology": "<p>ВИЧ-инфекцию вызывает вирус иммунодефицита человека, относящийся к семейству ретровирусов. </p>\r\n<p>ВИЧ – нестойкий вирус. Быстро погибает вне организма носителя, чувствителен к воздействию температуры (снижает инфекционные свойства при температуре 56 °С, погибает через 10 минут при нагревании до 70-80 °С). Хорошо сохраняется в крови и ее препаратах, подготовленных для переливания. Антигенная структура вируса весьма изменчива. </p>\r\n<p>Резервуаром и источником ВИЧ-инфекции является человек: страдающий СПИД и носитель. Вирус содержится в высоких концентрациях в крови, сперме, секрете вагинальных желез и менструальных выделениях. Может выделяться из женского молока, слюны, слезного секрета и ликвора, но эти биологические жидкости представляют меньшую эпидемиологическую опасность. </p>",
"pathogenesis": "<p>ВИЧ поражает клетки крови человека, имеющие на своей поверхности CD4-рецепторы: Т-лимфоциты, макрофаги и дендритные клетки. Инфицированные вирусом Т-лимфоциты гибнут из-за разрушения вирусом, апоптоза или уничтожения цитотоксическими Т-лимфоцитами. После того, как число CD4+ T-лимфоцитов становится ниже 200 в одном микролитре крови, система клеточного иммунитета перестает защищать организм </p>\r\n<p>Первое время организм компенсирует массовую гибель иммунных клеток, вырабатывая новые, со временем компенсация становится недостаточной, количество лимфоцитов и макрофагов в крови значительно снижается, иммунная система разрушается, организм становится беззащитен как по отношению к экзогенной инфекции, так и к бактериям, населяющим органы и ткани в норме (что ведет к развитию оппортунистических инфекций). Кроме того, нарушается механизм защиты от размножения дефектных бластоцитов - злокачественных клеток. </p>\r\n<p>Заселение вирусом иммунных клеток часто провоцирует различные аутоиммунные состояния, в частности характерны неврологические расстройства в результате аутоиммунного поражения нейроцитов, которые могут развиться даже раньше, чем проявится клиника иммунодефицита. </p>\r\n<p> </p>",
"diagnostics": "<p>В настоящее время существуют следующие методы диагностики ВИЧ: косвенные тесты позволяют обнаружить специфические антитела к ВИЧ, которые имеются почти у 100 % ВИЧ-инфицированных; прямые тесты определяют собственно ВИЧ, антигены ВИЧ или нуклеиновые кислоты ВИЧ (вирусная нагрузка). </p>\r\n<p>Вирусную нагрузку(число молекул геномной РНК ВИЧ в мл крови) напрямую связывают со скоростью снижения количества CD4+ лимфоцитов, эта характеристика является важным прогностическим показателем на ранних стадиях заболевания </p>\r\n<p>Для определения антител к ВИЧ, необходимо использовать, по крайней мере, два разных теста: предварительный тест (скрининг-тест) и подтверждающий тест. Большинства современных скрининг-тестов основываются на иммуноферментом анализе (ИФА) или сходных методах; они обладают высокой чувствительностью (до 99 %) и специфичностью (до 99,5 %). </p>\r\n<p>Методы экспресс-анализа основаны на методах реакция агглютинации, ИФА на полимерных мембранах (тест-полоски), иммунологическом фильтрационном анализе и иммунохроматографии. Экспресс-тесты дают результат в течение 15—30 минут. </p>\r\n<p>Подсчёт CD4+ лимфоцитов является критическим параметром в мониторинге ВИЧ-инфекции и позволяет оценивать состояние иммунной системы и склонность к развитию СПИД. </p>\r\n<p>Для подтверждения предполагаемого диагноза необходимо исследование клеточного субстрата опухоли. С этой целью выполняются диагностические операции: пункционная или эксцизионная биопсия, лапароскопия, торакоскопия, пункция костного мозга с последующими иммуногистохимическими, цитологическими, цитогенетическими и другими исследованиями диагностического материала. </p>\r\n<p>Увеличение медиастинальных и внутрибрюшных лимфоузлов обнаруживается с помощью УЗИ средостения, рентгенографии и КТ грудной клетки, брюшной полости. В алгоритм обследования по показаниям входят УЗИ лимфатических узлов, печени, селезенки, молочных желез, щитовидной железы, органов мошонки и др.; лимфосцинтиграфия, сцинтиграфия костей и внутренних органов, МРТ. </p>",
"treatment": "<p>Варианты лечения онкологических заболеваний при ВИЧ-инфекции включают оперативный метод, лучевую терапию и химиотерапию; их выбор определяется морфологическим типом, распространенностью, локализацией опухоли, сохранностью и возрастом больного. </p>\r\n<p>Лечение онкологических заболеваний включает лечение основного заболевания. До настоящего времени не разработано лечения ВИЧ-инфекции, которое могло бы устранить вирус иммунодефицита человека из организма. Современная высокоактивная антиретровирусная терапия замедляет прогрессирование ВИЧ-инфекции и её переход в стадию СПИД, позволяя ВИЧ-инфицированному человеку жить полноценной жизнью. </p>\r\n<p>На сегодняшний день преимущественным этиотропным лечением является назначение препаратов, снижающих репродуктивные способности вируса. Антиретровирусную терапию следует начинать у пациентов с ВИЧ-инфекцией независимо от клинической стадии заболевания, при любом количестве клеток СД4, в том числе всем ВИЧ-инфицированным беременным женщинам. </p>\r\n<p>Лечение ВИЧ-инфицированных включает общеукрепляющие и поддерживающие организм средства (витамины и биологически активные вещества) и методики физиотерапевтической профилактики вторичных заболеваний. </p>",
"prevention": "<p>В настоящее время Всемирная организация здравоохранения проводит общие профилактические мероприятия по снижению заболеваемости ВИЧ-инфекцией по четырем основным направлениям: </p>\r\n<ul>\r\n<li>просвещение в вопросах безопасности половых отношений, распространение презервативов, лечение заболеваний, передающихся половым путем, пропагандирование культуры половых взаимоотношений; </li>\r\n<li>контроль над изготовлением препаратов из донорской крови; </li>\r\n<li>ведение беременности ВИЧ-инфицированных женщин, обеспечение их медицинской помощи и предоставление им средств химиопрофилактики (в последнем триместре беременности и в родах женщины получают антиретровирусные препараты, которые также на первые три месяца жизни назначаются новорожденным детям); </li>\r\n<li>организация психологической и социальной помощи и поддержки ВИЧ-инфицированных граждан, консультирование. </li>\r\n</ul>\r\n<p>В настоящее время в мировой практике особое внимание уделяют таким эпидемиологически важным в отношении заболеваемости ВИЧ-инфекцией факторам, как наркомания, беспорядочная половая жизнь. В качестве профилактической меры во многих странах производится бесплатная раздача одноразовых шприцов, метадоновая заместительная терапия. В качестве меры, способствующей снижению половой неграмотности, в учебные программы вводятся обучающие половой гигиене курсы. </p>\r\n<p> </p>",
"clinical_picture": "<p>К онкологическим заболеваниям, возникающим при СПИДе, относятся: саркома Капоши, инвазивный рак шейки матки, неходжкинская лимфома и лимфома центральной нервной системы. При синдроме приобретенного иммунодефиците высока вероятность развития рака прямой кишки, болезни Ходжкина, рака ротовой полости, опухолевых заболеваний кожи, рака легких и яичек. </p>\r\n<p>Саркома Капоши характеризуется образованием темно-фиолетовых и коричневых пятен (лезии), возникающих на разных участках тела, преимущественно на стопе, боковых поверхностях голени и поверхностях кистей. В редких случаях злокачественные новообразования возникают на слизистых оболочках и веках. </p>\r\n<p>Примерно у 5-10% больных СПИДом встречаются лимфомы. Неходжкинская лимфома – заболевание лимфоидной ткани, которое при прогрессировании может распространиться и на другие органы. Все проявления лимфосарком укладываются в три синдрома: увеличение лимфоузлов, лихорадки и интоксикации. </p>\r\n<p>У ВИЧ-инфицированных также встречаются и первичные лимфомы центральной нервной системы. Частыми симптомами первичной лимфомы ЦНС являются судороги, потеря памяти, паралич лицевого нерва, сонливость. </p>\r\n<p>У ВИЧ-позитивных женщин высока вероятность развития предракового состояния в шейке матки, которое со временем может перейти в инвазивный рак шейки матки, когда раковые клетки поражают глубокие слои шейки матки. Наиболее характерным проявлением рака шейки матки служат кровянистые выделения и кровотечения: межменструальные, постменопаузальные, контактные (после полового акта, осмотра гинекологом, спринцевания и т. д.), меноррагии. Больные отмечают появление белей - жидких, водянистых, желтоватого или прозрачного цвета влагалищных выделений, обусловленных лимфореей. При распаде раковой опухоли выделения принимают гноевидных характер, иногда имеют цвет «мясных помоев» и зловонный запах. </p>",
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