ВОзвращает список болезней. Можно фильтр/искать

search, age, gender, parent, symptoms, sort_name, sort_by, rand, branches
GET /disease/?format=api&ordering=gender&page=50
HTTP 200 OK
Allow: GET, HEAD, OPTIONS
Content-Type: application/json
Vary: Accept

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            },
            "code": "E52",
            "name": "Недостаточность никотиновой кислоты [пеллагра]",
            "icd_name": "Недостаточность никотиновой кислоты [пеллагра]",
            "gender": 0,
            "age_min": 0,
            "age_max": 100,
            "cause": [
                3
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            "periodicity": 1,
            "slug": "e52_nedostatochnost_nikotinovoy_kisloty_pellagra",
            "lead": " Патологическое состояние, обусловленное дефицитом этого витамина в организме. ",
            "description": "При выраженном дефиците развивается пеллагра, в менее тяжелых случаях — гиповитаминоз РР, проявляющийся нарушениями функции нервной системы, кишечника, дистрофическими изменениями кожи.",
            "etiology": "<p>• Первичная — недостаток в поступающей пище, например, при преобладании в рационе кукурузы, поскольку витамин РР, хотя и содержится в ней, находится в связанном состоянии и не ассимилируется вЖКТ. Кроме того, белки кукурузы бедны триптофаном, используемого в организме для синтеза эндогенноговитамина РР<br />• Вторичная — обусловлена нарушением всасывания или усвоения витамина РР, а также увеличениемпотребности в нём <br />• Длительная <a href=\"/symptom/diareya/\" title=\"Перейти на страницу симптома Диарея\">диарея</a> <br />• Заболевания печени (чаще всего к недостаточности приводит цирроз)<br />• Алкоголизм<br />• Парентеральное питание без достаточного возмещения витаминов<br />• Злокачественные карциноидные опухоли (увеличивается потребность в триптофане)<br />• Сахарный диабет <br />• Болезнь Хартнапа</p>",
            "pathogenesis": "",
            "diagnostics": "<p>• Выделение N'-метилникотинамида с мочой менее 4 мг/сут<br />• Снижение содержания никотиновой кислоты<br />• Снижается содержание в крови и моче других витаминов группы В.</p>",
            "treatment": "<p>В среднетяжёлых и тяжёлых случаях необходима госпитализация.<br />• Диета. Включение в рацион продуктов, богатых витамином РР (мясо, яйца) и триптофаном (молоко).<br />• Назначение никотиновой кислоты (или никотинамида) от 300 до 1000 мг/сут внутрь в начале лечения привыраженном снижении содержания витамина РР в плазме крови, затем по 300-500 мг.<br />• При заболеваниях ЖКТ никотиновую кислоту (или никоти-намид) вводят парентерально (50-100 мг в/м 2-5раз/сут или 25-100мг <br />• Одновременно назначают по 5 мг тиамина, рибофлавина и пиридоксина.</p>",
            "prevention": "<p>• Питание с достаточным содержанием в пищевом рационе продуктов, богатых никотиновой кислотой<br />• При эндогенной форме — своевременная диагностика и лечение заболеваний, нарушающих всасывание иусвоение витамина РР, профилактическое введение препаратов никотиновой кислоты; дополнительноевведение витамина РР лицам с увеличенной в нём потребностью.</p>",
            "clinical_picture": "<p>При недостаточности витамина РР возникают нарушения функций кожи, слизистых оболочек, ЖКТ и ЦНС. Характерно наличие трёх Д (дерматит, <a title=\"Перейти на страницу симптома Диарея\" href=\"../../../symptom/diareya/\">диарея</a>, <a title=\"Перейти на страницу симптома Деменция\" href=\"../../../symptom/demenciya/\">деменция</a>) и поражение слизистых оболочек.<br />• Стадия прегиповитаминоза — неспецифические нарушения (<a title=\"Перейти на страницу симптома Слабость\" href=\"../../../symptom/slabost/\">слабость</a>, <a title=\"Перейти на страницу симптома Утомляемость\" href=\"../../../symptom/utomlyaemost/\">утомляемость</a>, <a title=\"Перейти на страницу симптома Снижение аппетита\" href=\"../../../symptom/snizhenie_appetita/\">снижение аппетита</a> ит.д.).<br />• Стадии гипо- и авитаминоза<br />• Дерматит. Поражение кожи может быть четырёх типов. Чаще всего оно симметрично и возникает научастках кожи, подвергающихся солнечному облучению или травматизации (давлению)<br />• Остро возникают кожные проявления в виде пеллагрической эритемы с последующим образованиемпузырьков, пузырей, корочек. Часто присоединяется вторичная инфекция <br />• Поражение кожных складок — <a title=\"Перейти на страницу симптома Покраснение кожи\" href=\"../../../symptom/pokrasnenie_kozhi/\">покраснение</a>, мацерация, эрозирование и присоединение вторичнойинфекции</p>\r\n<full></full>\r\n<p><br />• Хронические гипертрофические изменения -кожа утолщена, теряет эластичность, становится складчатой. Отмечают выраженную пигментацию на открытых участках тела и в местах, подвергающихся давлению. Очаги поражения имеют чёткую границу и окружены бордюром восстанавливающегося эпителия<br />• Хронические атрофические изменения кожи с потерей эластичности и шелушением. Характерны длядлительно текущих форм пеллагры. <br />• Поражение слизистых оболочек первично затрагивает ротовую полость, хотя возможно вовлечениеслизистых оболочек влагалища и уретры<br />• Для острой недостаточности характерны <a title=\"Перейти на страницу симптома Глоссит\" href=\"../../../symptom/glossit/\">глоссит</a> и стоматит, причём язык приобретает ярко-краснуюокраску. Первоначально в процесс вовлекаются кончик и края языка, а также участки слизистой оболочки, окружающие выходное отверстие стенбнова протока<br />• Постепенно вовлекается вся слизистая оболочка, язык становится болезненным и отёчным, возникаетгиперсаливация<br />• На поздних стадиях заболевания возникает характерная картина лакового языка (ярко-красный язык сгладкой поверхностью вследствие атрофии сосочков)<br />• Возможно появление язв на слизистой оболочке дна полости рта, нижней губы, а также напротив моляров. <br />• <a title=\"Перейти на страницу симптома Диарея\" href=\"../../../symptom/diareya/\">Диарея</a> — самый характерный признак нарушения функций пищеварительной системы. Поражение ЖКТвозникает на поздних стадиях заболевания. Характеризуется появлением чувства жжения слизистой оболочкиполости рта, глотки, пищевода; возникает желудочная <a title=\"Перейти на страницу симптома Диспепсия\" href=\"../../../symptom/dispepsiya/\">диспепсия</a> в виде тошноты, реже рвоты, чтообусловлено атрофическими изменениями слизистой оболочки желудка и возникновением ахилии. Возможнадиарея, чередующийся с запорами. О тяжёлом течении свидетельствует <a title=\"Перейти на страницу симптома Диарея\" href=\"../../../symptom/diareya/\">диарея</a> с прожилками крови врезультате образования эрозий и язв слизистой оболочки кишечника.<br />• <a title=\"Перейти на страницу симптома Деменция\" href=\"../../../symptom/demenciya/\">Деменция</a> — самый яркий признак поражения ЦНС; может сопровождаться органическим психозом и/илиэнцефало-патическим синдромом. На ранних стадиях появляется <a title=\"Перейти на страницу симптома Раздражительность и озлобление\" href=\"../../../symptom/razdrazhitelnost_i_ozloblenie/\">раздражительность</a>, возможны признакиполиневрита<br />• Органический <a title=\"Перейти на страницу симптома Психоз\" href=\"../../../symptom/psihoz/\">психоз</a> проявляется галлюцинаторно-параноидной, аффективной симптоматикой, психомоторным возбуждением<br />• Энцефалопатический синдром, характеризующийся затемнением сознания, гипертонусом мышцконечностей, появлением неконтролируемых сосательного и хватательного рефлексов.<br />• На поздних стадиях нарушаются функции эндокринной системы, возникает гипопротеинемия.</p>",
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            },
            "code": "E53",
            "name": "Недостаточность других витаминов группы B",
            "icd_name": "Недостаточность других витаминов группы B",
            "gender": 0,
            "age_min": 0,
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            "cause": [
                3
            ],
            "periodicity": 4,
            "slug": "e53_nedostatochnost_drugih_vitaminov_gruppy_b",
            "lead": " Патологическое состояние, обусловленное дефицитом этого витамина в организме и проявляющееся нарушениями преимущественно со стороны нервной системы, сердца и кишечника.",
            "description": "",
            "etiology": "<p>Гиповитаминоз B1 в развитых странах встречается нередко и наблюдается в основном у больных с хроническими заболеваниями желудочно-кишечного тракта, хроническим алкоголизмом, полиневритами, эндокринными заболеваниями (сахарный диабет, токсический зоб) и др. Недостаточность тиамина часто сочетается с дефицитом других витаминов группы В.</p>",
            "pathogenesis": "<p>Витамин B1 хорошо растворяется в воде; кофермент тиаминдифосфат (кокарбоксилаза) играет существенную роль в углеводном обмене. Наряду с этим тиамин участвует в обмене аминокислот, нуклеотидов, ацетилхолина и холинэстеразы. При дефиците витамина В1 в тканях и крови накапливаются недоокисленные продукты: пировиноградная кислота, молочная кислота и др. При этом нарушается деятельность различных отделов нервной системы (особенно периферической нервной системы), скелетных мышц и миокарда, а также функция желудочно-кишечного тракта.</p>",
            "diagnostics": "<p>Диагноз гиповитаминоза B1 подтверждается уменьшением содержания тиамина в крови (в сыворотке ниже 5 мкг/л, в эритроцитах ниже 30 мкг/л), резким повышением концентрации пировиноградной кислоты в крови (выше 0,01 г/л) и моче (выше 30 мг/сут), повышением содержания в крови молочной кислоты (выше 200 мг/л). В последнее время с этой целью используют также ряд более сложных современных методов.</p>",
            "treatment": "<p>Лечение больных обычно проводится амбулаторно. Рекомендуется диета, богатая тиамином, в сочетании с препаратами тиамина внутрь (50-100 мг/сут) или парентерально (30-60 мг/сут).</p>\r\n<p>Больные с выраженными формами болезни часто нуждаются в стационарном лечении. Парентерально вводят тиамин по 1 мл и кокарбоксилазу. Одновременно назначают другие витамины группы В (рибоксин, пиридоксин, никотиновая кислота) и аскорбиновую кислоту.</p>",
            "prevention": "",
            "clinical_picture": "<p>Характерны ухудшение аппетита, <a href=\"/symptom/toshnota/\" title=\"Перейти на страницу симптома Тошнота\">тошнота</a>, наклонность к запорам, парестезии в ногах, боль в икроножных мышцах при ходьбе, ухудшение сна, <a href=\"/symptom/razdrazhitelnost_i_ozloblenie/\" title=\"Перейти на страницу симптома Раздражительность и озлобление\">раздражительность</a>, плаксивость, снижение физической и психической работоспособности. Иногда умеренное похудание, лабильность пульса на фоне общей психоэмоциональной лабильности. Язык суховат, живот нерезко вздут, умеренно снижена болевая и глубокая чувствительность в области стоп и голеней, нерезко ослаблены сухожильные рефлексы (больше на ногах).</p>",
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            },
            "code": "I00",
            "name": "Ревматическая лихорадка без упоминания о вовлечении сердца",
            "icd_name": "Ревматическая лихорадка без упоминания о вовлечении сердца",
            "gender": 0,
            "age_min": 1,
            "age_max": 94,
            "cause": [
                3,
                2,
                4,
                9
            ],
            "periodicity": 1,
            "slug": "i00_revmaticheskaya_lihoradka_bez_upominaniya_o_vovlechenii_serdca",
            "lead": "Нарушение здоровья, относящееся к группе острая ревматическая лихорадка",
            "description": "Согласно международной классификации болезней, острая ревматическая лихорадка (ОРЛ) — системное заболевание соединительной ткани с преимущественной локализацией процесса в сердечно-сосудистой системе, развивающееся в связи с острой А-стрептококковой инфекцией у предрасположенных к нему лиц, главным образом в возрасте 7–15 лет.",
            "etiology": "<p>Ревматической атаке обычно предшествует стрептококковая инфекция, вызываемая β-гемолитическим стрептококком группы А: скарлатина, тонзиллит, родильная горячка, острый отит, фарингит, рожа. У 97% пациентов, перенесших стрептококковую инфекцию, формируется стойкий иммунный ответ. У остальных лиц стойкого иммунитета не вырабатывается, и при повторном инфицировании β-гемолитическим стрептококком развивается сложная аутоиммунная воспалительная реакция. </p>\r\n<p>Развитию ревматизма способствуют сниженный иммунитет, молодой возраст, большие коллективы (школы, интернаты, общежития), неудовлетворительные социальные условия (питание, жилье), переохлаждение, отягощенный семейный анамнез.  </p>",
            "pathogenesis": "<p>В патогенезе условно выделяются два периода: </p>\r\n<p>1. Не­посредственное повреждающее и токсическое действие на организм, оказываемое персистирующими стрептококками и их антигенами.</p>\r\n<p>2. Развитие иммунного воспаления в соединительной ткани организма с преимущественным вовлечением в процесс сердца и сосудов в результате запуска механизма синтезааутоантител.</p>",
            "diagnostics": "<p>Клинико-диагностические критерии ОРЛ Киселя-Джонса:  </p>\r\n<p>Большие:  </p>\r\n<p>ревмокардит (преимущественно поражения миокарда в виде миокардита и эндокардит с пороками сердца и без);  </p>\r\n<p>полиартрит (преимущественно крупных суставов, мигрирующий характер, без остаточных деформаций, рентгенологически негативный);  </p>\r\n<p>малая хорея;  </p>\r\n<p>ревматические узелки (подкожные узловатые уплотнения);  </p>\r\n<p>анулярная эритема (кольцевидные покраснения кожи туловища и проксимальных отделов конечностей).  </p>\r\n<p>Малые:  </p>\r\n<p>клинические: лихорадка, боли в суставах;  </p>\r\n<p>лабораторно-инструментальные:   </p>\r\n<p>а) лабораторные (С-реактивный белок, антистрептолизин-О,  ревматоидный фактор);   </p>\r\n<p>б) инструментальные (ЭКГ, электромиография, УЗИ сердца).  </p>",
            "treatment": "<p>Активная фаза ревматизма требует госпитализации пациента и соблюдения постельного режима. Применяются гипосенсибилизирующие и противовоспалительные препараты, кортикостероидные гормоны, нестероидные противовоспалительные препараты, иммунодепрессанты. <br class=\"SCXW246008330\" /> </p>",
            "prevention": "<p>Первичная: своевременная санация стрептококковых инфекций (ангины, стрептодермии, лечение болезней полости рта, болезней ЛОР-органов, правильная гигиена и т. д.), закаливание и т. д.  </p>\r\n<p>Вторичная (бициллинопрофилактика): экстенциллин 2,4 млн единиц раз в 3 недели:  </p>\r\n<ul>\r\n<li>стандартно — в течение 5 лет;  </li>\r\n<li>при манифестации ОРЛ в препубертате/пубертате без порока сердца — до 18 лет;  </li>\r\n<li>при манифестации ОРЛ в препубертате/пубертате с пороком сердца — до 25 лет.  </li>\r\n</ul>\r\n<p>Текущая: обязательное назначение при интеркуррентных заболеваниях пенициллинов и НПВС.  </p>\r\n<p> </p>",
            "clinical_picture": "<p>Симптомы ревматизма крайне полиморфны и зависят от степени остроты и активности процесса, а также вовлеченности в процесс различных органов. Типичная клиника ревматизма имеет прямую связь с перенесенной стрептококковой инфекцией (тонзиллитом, скарлатиной, фарингитом) и развивается спустя 1-2 недели после нее. Заболевание начинается остро с субфебрильной температуры (38—39 °С), слабости, утомляемости, головных болей, потливости. Одним из ранних проявлений ревматизма служат артралгии - боли в средних или крупных суставах (голеностопных, коленных, локтевых, плечевых, лучезапястных). </p>\r\n<p>При ревматизме артралгии носят множественный, симметричный и летучий (боли исчезают в одних и появляются в других суставах) характер. Отмечается припухлость, отечность, локальное покраснение и повышение температуры, резкое ограничение движений пораженных суставов. Течение ревматического полиартрита обычно доброкачественно: через несколько дней острота явлений стихает, суставы не деформируются, хотя умеренная болезненность может сохраняться в течение длительного времени. </p>\r\n<p>При ревматизме может поражаться центральная нервная система, специфическим признаком при этом служит, так называемая, ревматическая или малая хорея: появляются гиперкинезы – непроизвольные подергивания групп мышц, эмоциональная и мышечная слабость.</p>\r\n<p>Реже встречаются кожные проявления ревматизма: кольцевидная эритема (у 7–10% пациентов) и ревматические узелки. Кольцевидная эритема(аннулярная сыпь) представляет собой кольцевидные, бледно-розовые высыпания на туловище и голенях; ревматические подкожные узелки - плотные, округлые, безболезненные, малоподвижные, единичные или множественные узелки с локализацией в области средних и крупных суставов. </p>\r\n<p> </p>",
            "image": null,
            "image_alt": null,
            "standard_type": 3,
            "danger": 82,
            "published": 2,
            "parent": null,
            "block_rubric": 90,
            "standards": []
        },
        {
            "id": 7001,
            "symptoms": [],
            "alternative_names": [],
            "complications": [],
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                    "id": 1,
                    "disease_count": null,
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                    "slug": "akusherstvo_i_ginekologiya",
                    "lead": "Область медицины, включающая в себя лечение 739 заболеваний. В 1042 клиниках России оказывается помощь по этому направлению медицины.",
                    "image": null
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                {
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            "code": "I01",
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            "lead": "Нарушение здоровья, относящееся к группе острая ревматическая лихорадка",
            "description": "Ревмокардит — это основное клиническое проявление ревматизма, относящегося к системным заболеваниям соединительной ткани, при котором патологический процесс локализуется преимущественно в сердце. Ревматизм поражает чаще детей в возрасте 7—15 лет, реже встречается в дошкольном возрасте. ",
            "etiology": "<p>Ревматической атаке обычно предшествует стрептококковая инфекция, вызываемая β-гемолитическим стрептококком группы А: скарлатина, тонзиллит, родильная горячка, острый отит, фарингит, рожа. У 97% пациентов, перенесших стрептококковую инфекцию, формируется стойкий иммунный ответ. У остальных лиц стойкого иммунитета не вырабатывается, и при повторном инфицировании β-гемолитическим стрептококком развивается сложная аутоиммунная воспалительная реакция. </p>\r\n<p>Развитию ревматизма способствуют сниженный иммунитет, молодой возраст, большие коллективы (школы, интернаты, общежития), неудовлетворительные социальные условия (питание, жилье), переохлаждение, отягощенный семейный анамнез.  </p>",
            "pathogenesis": "<p>В патогенезе ОРЛ также можно говорить о ревматической патогенетической триаде: </p>\r\n<ol>\r\n<li>Прямое повреждение миокарда факторами патогенности стрептококка </li>\r\n<li>Аутоиммунный механизм - повреждение миокарда циркулирующими иммунными комплексами (антиген-антитело). </li>\r\n<li>Сосудистый механизм (поражение сосудов миокарда по вышеописанным механизмам ведет к нарушению циркуляции в миокарде — ишемии, ацидозу, — способствуя развитию воспаления, с одной стороны, и реактивному фиброзу, с другой). </li>\r\n</ol>",
            "diagnostics": "<p>Клинико-диагностические критерии ОРЛ Киселя-Джонса:  </p>\r\n<p>Большие:  </p>\r\n<ul>\r\n<li>ревмокардит (преимущественно поражения миокарда в виде миокардита и эндокардит с пороками сердца и без);  </li>\r\n<li>полиартрит (преимущественно крупных суставов, мигрирующий характер, без остаточных деформаций, рентгенологически негативный);  </li>\r\n<li>малая хорея;  </li>\r\n<li>ревматические узелки (подкожные узловатые уплотнения);  </li>\r\n<li>анулярная эритема (кольцевидные покраснения кожи туловища и проксимальных отделов конечностей).  </li>\r\n</ul>\r\n<p>Малые:  </p>\r\n<ul>\r\n<li>клинические: лихорадка, боли в суставах;  </li>\r\n<li>лабораторно-инструментальные:   </li>\r\n</ul>\r\n<p>а) лабораторные (С-реактивный белок, антистрептолизин-О,  ревматоидный фактор);   </p>\r\n<p>б) инструментальные (ЭКГ, электромиография, УЗИ сердца)  </p>",
            "treatment": "<p>Активная фаза ревматизма требует госпитализации пациента и соблюдения постельного режима. Лечение проводится ревматологом и кардиологом. Применяются гипосенсибилизирующие и противовоспалительные препараты, кортикостероидные гормоны, нестероидные противовоспалительные препараты, иммунодепрессанты. <br class=\"SCXW182360504\" /> </p>",
            "prevention": "<p>Первичная: своевременная санация стрептококковых инфекций (ангины, стрептодермии, лечение болезней полости рта, болезней ЛОР-органов, правильная гигиена и т. д.), закаливание и т. д. </p>\r\n<p>Вторичная (бициллинопрофилактика): экстенциллин 2,4 млн единиц раз в 3 недели: </p>\r\n<ul>\r\n<li>стандартно — в течение 5 лет; </li>\r\n<li>при манифестации ОРЛ в препубертате/пубертате без порока сердца — до 18 лет; </li>\r\n<li>при манифестации ОРЛ в препубертате/пубертате с пороком сердца — до 25 лет. </li>\r\n</ul>\r\n<p>Текущая: обязательное назначение при интеркуррентных заболеваниях пенициллинов и НПВС. </p>",
            "clinical_picture": "<p>Поражение сердца при ревматизме отмечается у 70-85% пациентов. При ревмокардите воспаляются все или отдельные оболочки сердца. Чаще происходит одновременное поражение эндокарда и миокарда (эндомиокардит), иногда с вовлеченностью перикарда (панкардит), возможно развитие изолированного поражения миокарда (миокардит). Во всех случаях при ревматизме в патологический процесс вовлекается миокард. </p>\r\n<p>При диффузном миокардите появляются одышка, сердцебиения, перебои и боли в сердце, кашель при физической нагрузке, в тяжелых случаях – недостаточность кровообращения, сердечная астма или отек легких. Пульс малый, тахиаритмичный. Благоприятным исходом диффузного миокардита считается миокардитический кардиосклероз. </p>\r\n<p>При эндокардите и эндомиокардите в ревматический процесс чаще вовлекается митральный (левый предсердно-желудочковый) клапан, реже аортальный и трикуспидальный (правый предсердно-желудочковый) клапаны. Клиника ревматического перикардита аналогична перикардитам иной этиологии.</p>",
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            "code": "A67",
            "name": "Пинта [карате]",
            "icd_name": "Пинта [карате]",
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            "lead": "Нарушение здоровья, относящееся к группе другие болезни, вызываемые спирохетами",
            "description": "Пинта (карате) является редким кожным инфекционным заболеванием и поражает кожу и слизистые оболочки без вовлечения в процесс внутренних органов, суставов и костей. ",
            "etiology": "<p>Возбудитель заболевания Treponema Carateum Herrejin. По морфологическим и антигенным свойствам она сходна с бледной трепонемой. Возбудитель пинты обитает в почве, на растениях, фруктах. Инфицирование происходит при прямом и непрямом контактах с больным человеком. Возбудитель проникает через поврежденную кожу или слизистые оболочки. Доказана передача возбудителя насекомыми (комары, клопы, мошки). Заболеванию особенно подвержены дети. Среди эндогенных факторов следует выделить низкую иммунную реактивность, повышенную потливость кожи и щелочную реакцию ее водно-липидной мантии. Среди экзогенных факторов особенно выделяются скученность населения, микротравмы и мацерация при высокой температуре и влажности тропического климата.  </p>",
            "pathogenesis": "<p>Проникают трепанемы через поврежденные участки кожи и слизистые оболочки. Затем они локализуются преимущественно в роговом слое эпидермиса, где развивается гиперпигментация, утолщение кожи, межклеточный отек. Также происходит нарушение пигментной функции, так как наблюдается гибель меланоцитов. Поражаются регионарные лимфатические узлы. В поздней стадии развивается очаговая атрофия эпидермиса. </p>",
            "diagnostics": "<p>Диагноз пинты основываются на появлении характерных пятен на коже и стадийности их изменений без нарушения общего состояния, за исключением кожного зуда. Диагноз подтверждается обнаружением в соскобах кожи трепонем; в поздних стадиях болезни становится положительной реакция Вассермана. </p>\r\n<p>Применяются серологические реакции, реакции иммунофлюоресценции - обнаруживаются специфические антитела к трепонеме. </p>",
            "treatment": "<p>Лечение проводят дюрантным и препаратами пенициллина, типа бициллина, или новыми препаратами – экстенциллнн, ретарпен и др. Детям и взрослым, контактировавшим с больными, проводят профилактическое лечение бициллином.</p>",
            "prevention": "<p> </p>\r\n<p>Профилактические мероприятия сводятся к выявлению и лечению больных и лиц, находящихся с ними в тесном контакте. Специфической профилактики не разработано. </p>",
            "clinical_picture": "<p>Инкубационный период 7-20 дней.  </p>\r\n<p>На месте внедрения трепонемы образуется эритематозная зудящая папула (узелок), размеры которой постепенно увеличиваются до нескольких сантиметров (примерно на 1 см в течение месяца). Вокруг папулы через 6-12 мес появляются мелкие пинтиды, зудящие, многочисленные, различного цвета (голубого, серого, бурого, желтого, фиолетового, красного и др.) разной величины и формы: круглые, овальные, неправильные; они шелушатся и напоминают чешуйчатый лишай или становятся лихеноидными (утолщенным).  </p>\r\n<p>В поздней третьей стадии болезни - на коже обилие ахроматичных и пигментированных пятен; появляется утолщение подошв и ладоней, болезненные трещины, пигментные нарушения ногтей. </p>\r\n<p>Физическое состояние больных не нарушается, сохраняется трудоспособность, и заболевание продолжается неопределенно длительное время. </p>",
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            "code": "I02",
            "name": "Ревматическая хорея",
            "icd_name": "Ревматическая хорея",
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            "lead": "Нарушение здоровья, относящееся к группе острая ревматическая лихорадка",
            "description": "Ревматическая хорея встречается преимущественно в детском возрасте и проявляется хореическим гиперкинезом, т.е. неритмичными, непроизвольными, хаотическими мышечными сокращениями, прекращающимися во время сна.",
            "etiology": "<p>Причиной малой хореи является перенесенная инфекция β-гемолитического стрептококка группы А. </p>\r\n<p>Этот вид стрептококка может поражать верхние дыхательные пути с развитием ангины и тонзиллита; кожные покровы с развитием рожи, стрептодермии, скарлатины. </p>",
            "pathogenesis": "<p>Организм борется с возбудителем, вырабатывая против него антитела, которые уничтожают стрептококк. У ряда людей одновременно антитела вырабатываются и к базальным ганглиям головного мозга. Это называется перекрестным аутоиммунным ответом. Антитела атакуют нервные клетки базальных ганглиев. Таким образом, возникает воспалительная реакция в подкорковых образованиях головного мозга, которая проявляет себя специфическими симптомами (гиперкинезами). </p>",
            "diagnostics": "<p>Клинико-диагностические критерии ОРЛ Киселя-Джонса: </p>\r\n<p>Большие: </p>\r\n<ul>\r\n<li>ревмокардит (преимущественно поражения миокарда в виде миокардита и эндокардит с пороками сердца и без); </li>\r\n<li>полиартрит (преимущественно крупных суставов, мигрирующий характер, без остаточных деформаций, рентгенологически негативный); </li>\r\n<li>малая хорея; </li>\r\n<li>ревматические узелки (подкожные узловатые уплотнения); </li>\r\n<li>анулярная эритема (кольцевидные покраснения кожи туловища и проксимальных отделов конечностей). </li>\r\n</ul>\r\n<p>Малые: </p>\r\n<ul>\r\n<li>клинические: лихорадка, боли в суставах; </li>\r\n<li>лабораторно-инструментальные:  </li>\r\n</ul>\r\n<p>а) лабораторные (С-реактивный белок, антистрептолизин-О,  ревматоидный фактор);  </p>\r\n<p>б) инструментальные (ЭКГ, электромиография) </p>\r\n<p> </p>",
            "treatment": "<p>Лечение малой хореи комплексное и направлено, в первую очередь, на ликвидацию ревматического процесса в организме, то есть на прекращение выработки антител против клеток собственного организма и борьбу со стрептококком. Немаловажную роль играет ликвидация гиперкинезов. </p>\r\n<p>В остром периоде рекомендован постельный режим, необходимо создать условия с минимальным воздействие раздражителей (света, звуков). Питание должно быть максимально витаминизированным, с небольшим ограничением углеводов. </p>\r\n<p>Назначается антибактериальная терапия - бициллин-1 или бициллин-5. В случае непереносимости пенициллина назначают эритромицин.  </p>\r\n<p>Если малая хорея сопровождается выраженными изменениями в крови (повышенное СОЭ, высокие титры антистрептолизина-О, повышение С-реактивного белка и так далее) и поражением других органов и систем, то таким больным показана терапия противоревматическими препаратами. Это могут быть нестероидные противовоспалительные средства и глюкокортикостероиды. </p>",
            "prevention": "<p>Первичная: своевременная санация стрептококковых инфекций (ангины, стрептодермии, лечение болезней полости рта, болезней ЛОР-органов, правильная гигиена и т. д.), закаливание и т. д. </p>\r\n<p>Вторичная (бициллинопрофилактика): экстенциллин 2,4 млн единиц раз в 3 недели: </p>\r\n<ul>\r\n<li>стандартно — в течение 5 лет; </li>\r\n<li>при манифестации ОРЛ в препубертате/пубертате без порока сердца — до 18 лет; </li>\r\n<li>при манифестации ОРЛ в препубертате/пубертате с пороком сердца — до 25 лет. </li>\r\n</ul>\r\n<p>Текущая: обязательное назначение при интеркуррентных заболеваниях пенициллинов и НПВС. </p>",
            "clinical_picture": "<p>Заболевание может возникнуть остро, но чаще отмечается постепенное развитие патологического процесса. Дети становятся несобранными, раздражительными, изменяется их поведение, появляются непроизвольные движения (гиперкинезы) разной степени выраженности. Гиперкинетический синдром часто больше выражен с одной стороны, нередко только в одной правой или левой половине тела (гемихорея). Одновременно у большинства больных появляется гипотония мышц. У 20-30% больных хореей имеется поражение сердца. </p>",
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                    "id": 2848,
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                        {
                            "name": "мышечная боль"
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                        {
                            "name": "боль в мышцах"
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                            "name": "мышцы болят"
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                            "name": "Миалгия"
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                            "name": "ломота"
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                        {
                            "name": "боль в мышцах ног"
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                        {
                            "name": "боль в мышцах после тренировки"
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                        {
                            "name": "боль в мышцах спины"
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            },
            "code": "E55",
            "name": "Недостаточность витамина D",
            "icd_name": "Недостаточность витамина D",
            "gender": 0,
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                "0"
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            "lead": "Представляет собой серьезное состояние организма, которое характеризуется нарушением всасывания в организме фосфора и кальция из пищи.",
            "description": "",
            "etiology": "<p>Люди в возрасте испытывают авитаминоз витамина Д вследствие нарушения работы почек, которые утрачивают способность перерабатывать данный элемент. Существуют также болезни кишечника, которые нарушают всасывание витамина D: Болезнь Крона, муковисцидоз и целиакия. У взрослых людей, страдающих ожирением, также наблюдается нехватка витамина Д.</p>\r\n<p>Нехватка витамина D провоцируются следующими факторами:</p>\r\n<ul>\r\n<li>Пятидесятилетний возраст;</li>\r\n<li>Беременность и период грудного вскармливания;</li>\r\n<li>Смуглая кожа;</li>\r\n<li>Болезни почек и печени;</li>\r\n<li>Применением антацидов;</li>\r\n<li>Веганская и вегетарианская диета.</li>\r\n</ul>",
            "pathogenesis": "",
            "diagnostics": "<p>Для диагноза у взрослых основное значение имеют данные биохимических лабораторных исследований (снижение содержания неорганического фосфора в крови ниже 30 мг/л; повышение активности щелочной фосфатазы).</p>",
            "treatment": "<p>Лечебным препаратом является эргокальциферол, выпускаемый в виде драже по 500 МЕ и масляный раствор по 500 и 1000 МЕ в капсулах (для профилактических целей). С лечебной целью назначают внутрь по 500 - 1000 МЕ в день, курсом, согласно инструкции по лечению рахита. Однако применение витаминов группы D (в силу возможности возникновения токсических гипервитаминозов) требует известной осторожности и должно проводиться под наблюдением врача.</p>",
            "prevention": "<p>Если говорить о профилактических мерах характерного недуга, то стоит обязательно отметить, что начинаться они должны с изменения своего привычного рациона. Врач назначает ребенку специальную лечебную диету, которая восполнит нехватку витамина Д. Питаться таким образом необходимо продолжительный период времени, помня о слабых местах организма пациента.Если говорить о лекарственных средствах, способствующих профилактике гиповитаминоза витамина Д, то в данном случае врач назначает поливитаминные комплексы, БАДы и витаминно-минеральные комплексы, также укрепляющие весь организм.</p>\r\n<p>&nbsp;</p>\r\n<p>&nbsp;</p>",
            "clinical_picture": "<p>Симптомы у детей &ndash; рахит, у взрослых &ndash; немотивированная <a title=\"Перейти на страницу симптома Слабость\" href=\"/symptom/slabost/\">слабость</a>, <a title=\"Перейти на страницу симптома Утомляемость\" href=\"/symptom/utomlyaemost/\">утомляемость</a>, деминерализация зубов с быстрым прогрессированием кариеса (особенно у беременных женщин), остеопороз, боль в области костей, в мышцах, парестезии.</p>",
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