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"code": "R75",
"name": "Лабораторное обнаружение вируса иммунодефицита человека [ВИЧ]",
"icd_name": "Лабораторное обнаружение вируса иммунодефицита человека [ВИЧ]",
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"slug": "r75_laboratornoe_obnaruzhenie_virusa_immunodeficita_cheloveka_vich",
"lead": "Нарушение здоровья, относящееся к группе отклонения от нормы, выявленные при исследовании крови, при отсутствии установленного диагноза",
"description": "ВИЧ-инфекция представляет собой заболевание, вызываемое вирусом иммунодефицита человека, характеризующееся синдромом приобретенного иммунодефицита, способствующего возникновению вторичных инфекций и злокачественных образований в связи с глубоким угнетением защитных ",
"etiology": "<p>ВИЧ-инфекцию вызывает вирус иммунодефицита человека, относящийся к семейству ретровирусов </p>\r\n<p>ВИЧ – нестойкий вирус. Быстро погибает вне организма носителя, чувствителен к воздействию температуры (снижает инфекционные свойства при температуре 56 °С, погибает через 10 минут при нагревании до 70-80 °С). Хорошо сохраняется в крови и ее препаратах, подготовленных для переливания. Антигенная структура вируса весьма изменчива. </p>\r\n<p>Резервуаром и источником ВИЧ-инфекции является человек: страдающий СПИД и носитель. Вирус содержится в высоких концентрациях в крови, сперме, секрете вагинальных желез и менструальных выделениях. Может выделяться из женского молока, слюны, слезного секрета и ликвора, но эти биологические жидкости представляют меньшую эпидемиологическую опасность. </p>",
"pathogenesis": "<p>ВИЧ поражает клетки крови человека, имеющие на своей поверхности CD4-рецепторы: Т-лимфоциты, макрофаги и дендритные клетки. . Инфицированные вирусом Т-лимфоциты гибнут из-за разрушения вирусом, апоптоза или уничтожения цитотоксическими Т-лимфоцитами. После того, как число CD4+ T-лимфоцитов становится ниже 200 в одном микролитре крови, система клеточного иммунитета перестает защищать организм </p>\r\n<p>Первое время организм компенсирует массовую гибель иммунных клеток, вырабатывая новые, со временем компенсация становится недостаточной, количество лимфоцитов и макрофагов в крови значительно снижается, иммунная система разрушается, организм становится беззащитен как по отношению к экзогенной инфекции, так и к бактериям, населяющим органы и ткани в норме (что ведет к развитию оппортунистических инфекций). Кроме того, нарушается механизм защиты от размножения дефектных бластоцитов - злокачественных клеток. </p>\r\n<p>Заселение вирусом иммунных клеток часто провоцирует различные аутоиммунные состояния, в частности характерны неврологические расстройства в результате аутоиммунного поражения нейроцитов, которые могут развиться даже раньше, чем проявится клиника иммунодефицита. </p>",
"diagnostics": "<p>В настоящее время существуют следующие методы диагностики ВИЧ: косвенные тесты позволяют обнаружить специфические антитела к ВИЧ, которые имеются почти у 100 % ВИЧ-инфицированных; прямые тесты определяют собственно ВИЧ, антигены ВИЧ или нуклеиновые кислоты ВИЧ (вирусная нагрузка). </p>\r\n<p>Вирусную нагрузку(число молекул геномной РНК ВИЧ в мл крови) напрямую связывают со скоростью снижения количества CD4+ лимфоцитов, эта характеристика является важным прогностическим показателем на ранних стадиях заболевания </p>\r\n<p>Для определения антител к ВИЧ, необходимо использовать, по крайней мере, два разных теста: предварительный тест (скрининг-тест) и подтверждающий тест. Большинства современных скрининг-тестов основываются на иммуноферментом анализе (ИФА) или сходных методах; они обладают высокой чувствительностью (до 99 %) и специфичностью (до 99,5 %). </p>\r\n<p>Методы экспресс-анализа основаны на методах реакция агглютинации, ИФА на полимерных мембранах (тест-полоски), иммунологическом фильтрационном анализе и иммунохроматографии. Экспресс-тесты дают результат в течение 15—30 минут. </p>\r\n<p>Подсчёт CD4+ лимфоцитов является критическим параметром в мониторинге ВИЧ-инфекции и позволяет оценивать состояние иммунной системы и склонность к развитию СПИД. </p>",
"treatment": "<p>До настоящего времени не разработано лечения ВИЧ-инфекции, которое могло бы устранить вирус иммунодефицита человека из организма. Современная высокоактивная антиретровирусная терапия замедляет прогрессирование ВИЧ-инфекции и её переход в стадию СПИД, позволяя ВИЧ-инфицированному человеку жить полноценной жизнью. </p>\r\n<p>На сегодняшний день преимущественным этиотропным лечением является назначение препаратов, снижающих репродуктивные способности вируса. Антиретровирусную терапию следует начинать у пациентов с ВИЧ-инфекцией независимо от клинической стадии заболевания, при любом количестве клеток СД4, в том числе всем ВИЧ-инфицированным беременным женщинам. </p>\r\n<p>Лечение ВИЧ-инфицированных включает общеукрепляющие и поддерживающие организм средства (витамины и биологически активные вещества) и методики физиотерапевтической профилактики вторичных заболеваний. </p>",
"prevention": "<p>В настоящее время Всемирная организация здравоохранения проводит общие профилактические мероприятия по снижению заболеваемости ВИЧ-инфекцией по четырем основным направлениям: </p>\r\n<p>просвещение в вопросах безопасности половых отношений, распространение презервативов, лечение заболеваний, передающихся половым путем, пропагандирование культуры половых взаимоотношений; </p>\r\n<p>контроль над изготовлением препаратов из донорской крови; </p>\r\n<p>ведение беременности ВИЧ-инфицированных женщин, обеспечение их медицинской помощи и предоставление им средств химиопрофилактики (в последнем триместре беременности и в родах женщины получают антиретровирусные препараты, которые также на первые три месяца жизни назначаются новорожденным детям); </p>\r\n<p>организация психологической и социальной помощи и поддержки ВИЧ-инфицированных граждан, консультирование. </p>\r\n<p>В настоящее время в мировой практике особое внимание уделяют таким эпидемиологически важным в отношении заболеваемости ВИЧ-инфекцией факторам, как наркомания, беспорядочная половая жизнь. В качестве профилактической меры во многих странах производится бесплатная раздача одноразовых шприцов, метадоновая заместительная терапия. В качестве меры, способствующей снижению половой неграмотности, в учебные программы вводятся обучающие половой гигиене курсы. </p>\r\n<p> </p>\r\n<p> </p>",
"clinical_picture": "<p>Инкубационный период, время от момента заражения ВИЧ до появления симптоматики, называют серологическим окном. Сывороточные реакции на вирус иммунодефицита отрицательны: специфические антитела ещё не определяются. Средняя продолжительность инкубации составляет 12 недель; сроки могут сокращаться до 14 дней при сопутствующих ЗППП, туберкулёзе, общей астении, либо увеличиваться до 10-20 лет. В течение всего периода пациент опасен как источник ВИЧ-инфекции. </p>\r\n<p>Стадия первичных проявлений характеризуется проявлением реакции организма на активную репликацию вируса в виде клиники острой инфекции и иммунной реакции (выработка специфических антител). Вторая стадия может протекать бессимптомно, единственным признаком развивающейся ВИЧ-инфекции будет положительная серологическая диагностика на антитела к вирусу. </p>\r\n<p>Клинические проявления второй стадии протекают по типу острой ВИЧ-инфекции. Начало острое, отмечается у 50-90% пациентов спустя три месяца после момента заражения, зачастую предшествуя формированию ВИЧ-антител. Острая инфекция без вторичных патологий имеет довольно разнообразное течение: могут отмечаться лихорадка, разнообразные полиморфные высыпания на кожных покровах и видимых слизистых оболочках, увеличение лимфатических узлов, боль в горле, увеличение селезенки, диарея. </p>\r\n<p>Острая ВИЧ-инфекция обычно длится от нескольких дней до нескольких месяцев, в среднем 2-3 недели, после чего в подавляющем большинстве случаев переходит в латентную стадию. </p>\r\n<p>Латентная стадия характеризуется постепенным нарастанием иммунодефицита. Гибель иммунных клеток на этой стадии компенсируется их повышенным производством. В это время диагностировать ВИЧ можно с помощью серологических реакций (в крови присутствуют антитела к ВИЧ). Клиническим признаком может быть увеличение нескольких лимфатических узлов из разных, не связанных между собой групп, исключая паховые лимфоузлы. При этом других патологических изменений со стороны увеличенных лимфоузлов (болезненность, изменения окружающих тканей) не отмечается. Латентная стадия может продолжаться от 2-3 лет, до 20 и более. В среднем она длиться 6-7 лет.</p>",
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"code": "P23",
"name": "Врожденная пневмония",
"icd_name": "Врожденная пневмония",
"gender": 0,
"age_min": 0,
"age_max": 1,
"cause": [
4,
5,
9,
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],
"periodicity": 2,
"slug": "p23_vrozhdennaya_pnevmoniya",
"lead": "Нарушение здоровья, относящееся к группе дыхательные и сердечно-сосудистые нарушения, характерные для перинатального периода",
"description": " \r\nВрожденная пневмония — это остро протекающее инфекционно-воспалительное заболевание легких, которое проявляется в первые 72 часа жизни. ",
"etiology": "<p>Воспаление легких у новорожденных вызывают различного рода болезнетворные микроорганизмы, грибы, вирусы, простейшие. К ним относятся возбудители: стафилококк, стрептококк, пневмококк, вирус простого герпеса, цитомегаловирус, микоплазмы, хламидии, грибы рода Кандида и множество других. Пути попадания инфекции различны. </p>\r\n<p>Классифицируют бронхогенный (заражение происходит через легкие во время прохождения по родовым путям) и гематогенный (инфицирование ребенка матерью через кровь во время вынашивания плода) способ ее проникновения в детский организм. </p>\r\n<p>К группе риска относятся младенцы, период дородового развития которых происходил с такими осложнениями, как: </p>\r\n<ul>\r\n<li>внутриутробная гипоксия (недостаток получения кислорода плодом); </li>\r\n<li>застойные явления в легких при гипотрофии, то есть недоразвитие дыхательных путей; </li>\r\n<li>пороки сердца, легких; </li>\r\n<li>нарушение иммунитета, передающееся по наследству; </li>\r\n<li>недостаток или избыток витаминов в организме плода; </li>\r\n<li>хронические болезни матери и перенесенные инфекции в период созревания ребенка; </li>\r\n<li>патологические кровотечения во время беременности; </li>\r\n<li>слишком раннее отхождение околоплодных вод; </li>\r\n<li>повреждения головного мозга и верхних дыхательных путей при родах. </li>\r\n</ul>",
"pathogenesis": "<p>Возбудитель, проникнув в легкие через кровь или дыхательные пути, вызывает здесь воспалительный процесс (как правило, двусторонний). Это сопровождается отеком тканей, патологической продукцией секрета, очаговым спаданием альвеол. В результате — легкие не в состоянии качественно исполнять свою функцию, а организм новорожденных страдает от недостаточности кислорода, избытка углекислого газа, нарушения обменных процессов. При отсутствии адекватного лечения возможны опасные последствия: тяжелые дыхательные расстройства, отек легких, нарушение сердечной деятельности, полиорганная недостаточность. </p>",
"diagnostics": "<p>В общем анализе крови выявляют повышение (или снижение) числа лейкоцитов, сдвиг формулы влево, появление незрелых форм. </p>\r\n<p>Исследование мочи поможет определить кетоацидоз, поражение почек. </p>\r\n<p>На рентгенографии грудной клетки выявляют характерные воспалительные инфильтраты, усиление легочного рисунка, расширение прикорневой зоны. </p>\r\n<p>Бактериологические и серологические исследования крови (ликвора) дают возможность определить возбудителя и его чувствительность к лекарственным средствам. </p>",
"treatment": "<p>Уход. Необходимо поддерживать оптимальную температуру окружающей среды, недопустимо переохлаждение и перегревание ребенка. Правильный уход предусматривает свободное пеленание, частые перемены положения ребенка, регулярную обработку кожи и слизистых оболочек. </p>\r\n<p>Оксигенотерапия применяется для борьбы с дыхательной недостаточностью и в зависимости от ее степени тяжести осуществляется путем ингаляций, помещения ребенка в кислородную палатку, </p>\r\n<p>Вскармливание. Способ кормления (грудь, соска, зонд) и количество пищи определяется возрастом ребенка и его состоянием. </p>\r\n<p>В качестве этиотропной (направленной на возбудителя) терапии назначаются антибактериальные препараты с учетом чувствительности к ним возбудителя. </p>\r\n<p>Инфузионная терапия с целью детоксикации. </p>\r\n<p>Кислородотерапия. </p>\r\n<p>Пассивная иммунотерапия. При тяжелом течении пневмонии показаны специфические иммуноглобулины. </p>\r\n<p>Местная терапия: - СВЧ на грудную клетку 5-7 сеансов, далее электрофорез с эуфиллином, новокаином, препаратами кальция 7-10 сеансов; - отвлекающая терапия - горчичное обертывание грудной клетки; - регулярные санации трахеобронхиального дерева или бронхоскопии с промыванием бронхов антисептическими растворами. </p>",
"prevention": "<p>Профилактика врожденной пневмонии заключается в устранении предрасполагающих факторов к развитию пневмонии, соблюдении санэпидрежима в родильном доме, в отделениях больниц для новорожденных. </p>",
"clinical_picture": "<p>Клиническая картина заболевания выглядит следующим образом: </p>\r\n<ul>\r\n<li>гипотония (понижение кровяного давления); </li>\r\n<li>асфиксия (затрудненное дыхание, удушье); </li>\r\n<li>бледный с серым оттенком цвет кожи; </li>\r\n<li>рвота, частое срыгивание; </li>\r\n<li>отечность конечностей; </li>\r\n<li>наличие хрипов при прослушивании легких; </li>\r\n<li>вялость, слабость, сонливость, отказ от кормления, заторможенность; </li>\r\n<li>появление кашля и одышки; </li>\r\n<li>нередко сердечно-сосудистая недостаточность, вздутие живота; </li>\r\n<li>повышение у доношенных детей температуры тела до 40°С; </li>\r\n<li>понижение у недоношенных детей температуры тела до 35°С; </li>\r\n<li>при взятии анализа крови определяется повышенный лейкоцитоз; </li>\r\n<li>резкое снижение веса в первые дни жизни.</li>\r\n</ul>",
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