ВОзвращает список болезней. Можно фильтр/искать

search, age, gender, parent, symptoms, sort_name, sort_by, rand, branches
GET /disease/?format=api&ordering=slug&page=40
HTTP 200 OK
Allow: GET, HEAD, OPTIONS
Content-Type: application/json
Vary: Accept

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            },
            "code": "B68",
            "name": "Тениоз",
            "icd_name": "Тениоз",
            "gender": 0,
            "age_min": 0,
            "age_max": 100,
            "cause": [
                2,
                7
            ],
            "periodicity": 1,
            "slug": "b68_tenioz",
            "lead": "Заболевание, вызванное плоским гельминтом, относящимся к тениидозам.",
            "description": "Инфекционное заразное заболевание.",
            "etiology": "<p>Тениоз вызывается цепнем свиным, Taenia solium. Паразит имеет длину 1,5—2 м и число члеников от 800 до 1000. Сколекс около 1—2 мм в диаметре, имеет 4 крестообразно расположенные присоски и хоботок, на котором находится двойная “корона” из 22—32 крючьев. Членики в передней части имеют длину короче ширины, а у зрелых члеников в конце стробилы длина всегда превышает ширину. Яйца практически не отличаются от яиц бычьего цепня. Распространен тениоз повсеместно, особенно в Индии, Северном Китае, в Африке и Южной Америке. Развитие цепня свиного происходит со сменой хозяев. Окончательный хозяин — человек, промежуточный — свинья, которая заражается при поедании члеников или яиц гельминта.</p>\r\n<p>Источником заражения человека тениозом служит недостаточно обеззараженное мясо свиней и реже диких кабанов. Наибольшую опасность представляет свинина, не прошедшая ветеринарный контроль.</p>",
            "pathogenesis": "<p>В желудке свиньи оболочка яйца цепня свиного разрушается, и освободившиеся онкосферы активно проникают в кровеносную систему желудка или кишок и током крови разносятся по телу. Через 24—72 часа онкосферы оседают преимущественно в межмышечной соединительной ткани, где через два месяца превращаются в финну (цистицерк).</p>\r\n<full></full>\r\n<p>Цистицерки превращаются во взрослых паразитов в кишечнике человека, где под действием пищеварительного сока и желчи происходит выворачивание головки, которая с помощью присосок прикрепляется к слизистой оболочке. Паразит начинает расти, формируя членики, и через 2—2,5 месяца достигает зрелости.</p>\r\n<p>В патогенезе тениоза играют значительную роль токсико-аллергические реакции, механическое раздражение слизистой оболочки присосками и крючками, поглощение питательных веществ хозяина.</p>",
            "diagnostics": "<p>Диагностика тениоза основывается на обнаружении в фекалиях больных зрелых члеников свиного цепня, которые отделяются группами по 5—6, реже поодиночке. Активными движениями членики не обладают.</p>",
            "treatment": "<p>Ранее для лечения тениоза применялся фенасал, экстракт мужского папоротника и семена тыквы. <br /><br />Для дегельминтизации тениоза применяется также Вермокс, при применении Вермокса не требуется соблюдение диеты и назначение слабительных средств. <br /><br />Взрослым Вермокс назначают по 200 мг 2 раза в сутки в течение 3 дней. Детям - по 100 мг 2 раза в сутки также в течение 3 дней.</p>",
            "prevention": "<p>Профилактика тениоза диктует необходимость приобретения свинины только в магазинах или на рынках, где осуществляется ветеринарно-санитарная экспертиза мясной продукции. В пищу можно употреблять только хорошо прожаренное (проваренное, запеченное) мясо; дегустация сырого свиного фарша должна быть исключена. По эпидемическим показаниям проводится обследование лиц, занятых уходом за свиньями. Профилактика цистицеркоза требует соблюдения норм личной гигиены (мытье рук после посещения туалета, контакта с землей, перед приемом пищи и т. д.).</p>",
            "clinical_picture": "<p>У больных наблюдаются расстройства функции кишечника, <a href=\"/symptom/slabost/\" title=\"Перейти на страницу симптома Слабость\">слабость</a>, <a href=\"/symptom/golovokruzhenie/\" title=\"Перейти на страницу симптома Головокружение\">головокружение</a>. Отмечается <a href=\"/symptom/toshnota/\" title=\"Перейти на страницу симптома Тошнота\">тошнота</a>, <a href=\"/symptom/rvota/\" title=\"Перейти на страницу симптома Рвота\">рвота</a>, <a href=\"/symptom/bol_v_zhivote/\" title=\"Перейти на страницу симптома Боль в животе\">боли в животе</a>, периодические головные боли, нарушения сна. В некоторых случаях неврологические симптомы связаны с возможным наличием цистицеркоза мозга. Иногда тениоз сопровождается гипохромной анемией. Нередко клинические проявления отсутствуют.</p>",
            "image": null,
            "image_alt": null,
            "standard_type": 3,
            "danger": 16,
            "published": 1,
            "parent": null,
            "block_rubric": 17,
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            },
            "code": "B69",
            "name": "Цистицеркоз",
            "icd_name": "Цистицеркоз",
            "gender": 0,
            "age_min": 0,
            "age_max": 100,
            "cause": [
                7
            ],
            "periodicity": 1,
            "slug": "b69_cisticerkoz",
            "lead": "Цистицеркоз - паразитарное заболевание человека, вызываемое личинками свиного цепня - цистицерками.",
            "description": "Цистицеркоз - паразитарное заболевание человека, вызываемое личинками свиного цепня - цистицерками. В данном случае человек становится промежуточным факультативным хозяином гельминта. ",
            "etiology": "",
            "pathogenesis": "<p>Заражение цистицеркозом происходит при попадании в желудок яиц, онкосфер свиного цепня, развивающихся в организме человека так же, как у основного промежуточного хозяина - свиньи. В желудке под действием пепсина оболочка онкосфер разрушается, освободившиеся шестикрючные зародыши активно вбуравливаются в стенку кишки, попадают в кровеносные суды и гематогенно разносятся по организму, оседая в различных органах, где онкосферы превращаются в цистицерки. Цистиц представляет собой прозрачный пузырек величиной в диаметром от просяного зерна до 1,5 см, наполненный жидкостью, со сколексом внутри. Чаще всего, по мнению многих исследователей цистицеркоз поражает центральную нервную систему (74,5 - 82%), глаза и мышцы. <br /><br />Известны два пути заражения цистицеркозом - экзогенный и эндогенный. При экзогенном заражении онкосферы попадают извне. Так, больные тениозом могут заразиться через собственные грязные руки, лаборанты - в случаях нарушения правил работы с исследуемым материалом, а также любой человек через овощи, ягоды, фрукты, загрязненные онкосферами при удобрении почвы огородов и ягодников необезвреженными нечистотами, инвазированными свиным цепнем. <br /><br />Эндогенным путем заражаются только больные тениозом при антиперистальтике, попадании члеников свиного цепня из кишечника в желудок с последующим их перевариванием и освобождением десятков тысяч яиц. В этих случаях инвазия очень интенсивна. Причины антиперистальтики различны: любое пищевое <a href=\"/symptom/ostraya_intoksikaciya/\" title=\"Перейти на страницу симптома Острая интоксикация\">отравление</a>, алкогольное опьянение, выход из состояния наркоза, а также введение зонда и др. <br /><br />Цистицерки оказывают местное и общее влияние на организм: местное обусловлено механическим их действием и локализацией, а общее связано с поступлением в кровь и ликвор токсических продуктов жизнедеятельности личинок. Наиболее выраженное влияние связано с процессом отмирания цистицерка, усилением рубцово-сморщивающих процессов в его теле и его обызвествлением. В зависимости от локализации различают цистицеркоз мозга, глаза и мышц.</p>",
            "diagnostics": "<p>Для цистицеркоза мозга типично клиническое течение болезни, характеризующееся внезапными обострениями и длительными ремиссиями, полиморфизмом неврологической симптоматики с превалированием симптомов раздражения, лимфоидно-нейтрофильным плеоцитозом ликвора (в 93%), эозинофилией крови (не всегда). При его диагностировании также учитываются рентгенологические данные (краниография, рентгенография, энцефало- и вентрикулография, ангиография), результаты серологического исследования - РСК, ELISA, НРИФ, РНГА. <br /><br />Диагноз цистицеркоза кожи, мышц можно поставить на основании биопсии узелков и рентгенографии обызвествленных цистицерков. <br /><br />Цистицеркоз глаза можно определить в результате офтальмоскопии.</p>",
            "treatment": "<p>При поражении мягких тканей цистицерками, если они не вызывают механического раздражения и исключено поражение ЦНС и глаз, лечение не проводится, больной остается под наблюдением. Это связано с тем, что специфические препараты  вызывают гибель паразитов, продукты распада которых могут давать тяжёлые побочные реакции аллергического характера. <br /><br />Лечение цистицеркоза глаза и единичных цистицерков головного мозга хирургическое. Дополнительно - этиотропная терапия в течение 15 дней перорально на фоне применения дексаметазона по 4-16 мг в сутки. Этиотропная терапия проводится также в неоперабельных случаях цистицеркоза головного мозга. </p>",
            "prevention": "<p>Профилактикой цистицеркозов является успешная борьба с тениозом и соблюдение мер личной профилактики.</p>",
            "clinical_picture": "<p>Цистицеркоз мозга. По особенностям клиники выделяют следующие четыре формы этого заболевания: <br />1. цистицеркоз больших полушарий; <br />2. цистицеркоз желудочковой системы; <br />3. цистицеркоз основания; <br />4. смешанный цистицеркоз. <br /><br />При первой форме инвазия проявляется симптомами повышенного внутричерепного давления вследствие нарушения ликворооттока (больные жалуются на приступообразные головные боли, <a href=\"/symptom/golovokruzhenie/\" title=\"Перейти на страницу симптома Головокружение\">головокружение</a>, рвоту; нередко у них выявляется застойный сосок зрительного нерва). Частым симптомом ее являются эпилептические припадки. <br /><br />Наиболее распространенной формой желудочкового цистицеркоза является цистицеркоз IV желудочка. Ему свойственно внезапное и бурное начало, характеризующееся приступами головных болей с рвотой. Типичны для этой формы заболевания брунсовские приступы, связанные с изменением положения головы. У большинства больных наблюдается вынужденное положение тела, так как перемена позы вызывает резкое обострение головной боли и рвоту. <br /><br />Клиника цистицеркоза основания мозга также вариабельна проявляется нарушением ликвороциркуляции. Довольно часто отмечаются вынужденное положение головы, боли в затылочно-шейной области, снижение вкусовой чувствительности, слуха и вестибулярной возбудимости. Могут возникать чувствительные и двигательные расстройства, вплоть до парезов. В 1/5 части случаев заболевания наблюдаются различные психические нарушения. <br /><br />Смешанная форма цистицеркоза мозга протекает тяжело прогноз ее неблагоприятный. Наиболее типичны для нее эпилептический симптомокомплекс, грубые психические нарушения, галлюцинации и др.; спинной мозг поражается редко. <br /><br />Цистицеркоз глаза. Цистицерки могут локализоваться во всех оболочках глаза, что клинически проявляется различным по тяжести и характеру нарушением зрения, до полной его потери, чувством давления и боли. <br /><br />Наиболее прогностически и клинически благоприятной формой является цистицеркоз кожи, подкожной клетчатки, мышц. <br /><br />Цистицеркоз кожи. <br />При расположении личинок в подкожном жировом слое возникают опухолевидные образования, возвышающиеся над поверхностью кожи, при их пальпации можно определить полостный характер опухолей. Наиболее частая локализация: внутренние поверхности плеч, верхняя половина грудной клетки, ладони. <br /><br />Болезнь сопровождается выраженной эозинофилией в крови (до 40%), крапивницей. <br /><br />Цистицеркоз кожи, мышц протекает бессимптомно. <br /><br />Цистицеркоз сердца. <br />При локализации паразитов в сердце, в области предсердно-желудочного пучка, нарушается сердечный ритм. <br /><br />Цистицеркоз лёгких. <br />Цистицеркоз легких в большинстве случаев, протекает без выраженных клинических симптомов и обнаруживается случайно при рентгенологическом исследовании. На рентгенограмме видны круглые тени, в основном интенсивные с четкими границами величиной от зерна перца до маленькой вишни. Эти тени, как правило, разбросаны в обоих легочных полях, число их может быть различным от нескольких единиц до нескольких десятков. Очаги паразитов могут быть частично или полностью обызвествлены.</p>",
            "image": null,
            "image_alt": null,
            "standard_type": 1,
            "danger": 26,
            "published": 1,
            "parent": null,
            "block_rubric": 17,
            "standards": []
        },
        {
            "id": 8686,
            "symptoms": [
                {
                    "id": 3105,
                    "synonyms": [
                        {
                            "name": "язык болит"
                        },
                        {
                            "name": "боль в ротовой полости"
                        },
                        {
                            "name": "жжет язык"
                        },
                        {
                            "name": "рот болит"
                        }
                    ],
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                            "y": 1333,
                            "r": 300
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                    "id": 2953,
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                            "name": "Живот болит"
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                        {
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                        {
                            "name": "Боль выше пупка"
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                        {
                            "name": "Боль ниже пупка"
                        },
                        {
                            "name": "Острые боли в животе"
                        },
                        {
                            "name": "Боль в области живота "
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                        {
                            "name": "Боль в области пупка"
                        },
                        {
                            "name": "Боль в правой жасти живота"
                        },
                        {
                            "name": "Пупок тянет"
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                        {
                            "name": "Болит пупок"
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                        {
                            "name": "Болит живот снизу"
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                        {
                            "name": "Болит живот сверху"
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                        {
                            "name": "Боль в правой части живота"
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                        {
                            "name": "Боль в животе у ребенка"
                        },
                        {
                            "name": "У ребенка болит живот"
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                        {
                            "name": "Острая боль в животе"
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                        {
                            "name": "Резь в животе"
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                        {
                            "name": "Боль под ложечкой"
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                        {
                            "name": "Боль после еды"
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                        {
                            "name": "Боль при аппендеците"
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                    "lead": "Боль в животе может быть признаком многих  заболеваний,  если боль появилась внезапно или продолжительная, обратитесь к врачу",
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                        {
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                        {
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                    "age_min": 0,
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                    "slug": "snizhenie_appetita",
                    "lead": "Частичный или полный отказ от приема пищи. Проявляется снижением общего аппетита (не хочется ничего), изменением аппетита (нарушаются вкусовые потребности в отношении какой либо группы продуктов),  полным отсутствием аппетита (анорексия).",
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                {
                    "id": 2572,
                    "synonyms": [
                        {
                            "name": "Гипотония"
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                        {
                            "name": "Артериальная гипотензия"
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                        {
                            "name": "Давление упало"
                        },
                        {
                            "name": "Давление падает"
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            "etiology": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Возбудителями дифиллоботриоза человека могут выступать 12 видов паразитических червей&ndash;лентецов, наиболее частым представителем которых является Diphyllobothrium latum (лентец широкий). D. Latum &ndash; крупный паразитический червь, длина которого может достигать 7-10 м. Стробила (тело) широкого лентеца состоит из 3-4 тысяч члеников &ndash; проглоттид; сколекс (головка) снабжена двумя щелевидными присосками, посредством которых он крепится к стенке тонкой кишки.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Жизненный цикл развития широкого лентеца предполагает смену трех хозяев; при этом окончательным хозяином выступает человек или животные (кошки, собаки, лисы, медведи), промежуточным - пресноводные рачки, дополнительным - пресноводные рыбы (окунь, щука, ерш, налим, лососевые). Из кишечника окончательного хозяина яйца лентеца с фекалиями попадают в окружающую среду.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Их дальнейшее развитие происходит в воде, где через несколько недель из яиц выходят личинки &ndash; корацидии. В воде они заглатываются промежуточными хозяевами - веслоногими рачками, в организме которых переходят в следующую личиночную стадию развития - процеркоиды. Инвазированные рачки служат пищей для пресноводных рыб, в организме которых процеркоиды превращаются в инвазионных для человека личинок &ndash; плероцеркоид. В мышцах, икре, печени рыб личинок лентеца можно рассмотреть невооруженным глазом.</span></p>\r\n<p>&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Заражение человека дифиллоботриозом происходит при употреблении в пищу сырой, плохо термически обработанной, малосольной рыбы или икры, содержащей плероцеркоиды. Попадая в тонкую кишку окончательного хозяина, личинки прикрепляются к ее слизистой оболочке и превращаются в половозрелых гельминтов, способных выделять яйца. В организме человека широкий лентец способен паразитировать 20-25 лет. Человек, больной дифиллоботриозом, для окружающих не заразен.</span></p>",
            "pathogenesis": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">После употребления заражённой рыбы в результате переваривания освобождается личинка лентеца &mdash; плероцеркоид, которая в тонком кишечнике человека прикрепляется к слизистой оболочке посредством присосок &mdash; ботрий. Там происходит её рост и развитие: личинка всей поверхностью своего тела активно поглощает находящиеся в просвете кишечника питательные вещества, витамины и микроэлементы.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Примерно через месяц от момента заражения червь достигает половой зрелости и начинает отделять яйца, периодически отбрасывая часть зрелых, наполненных яйцами члеников.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">В процессе роста и развития паразита на организм человека оказывается ряд патологических влияний:</span></p>\r\n<ul style=\"margin-top: 0; margin-bottom: 0; padding-inline-start: 48px;\">\r\n<li><span style=\"font-size: 7pt; color: #000000; background-color: transparent; font-style: normal; font-variant-numeric: normal; font-variant-east-asian: normal; vertical-align: baseline; white-space: pre-wrap;\">в результате присасывания к слизистой оболочке происходит местное нарушение её целостности, строения и функционирования &mdash; нарушается кровообращение, питание тканей, изменение нервной проводимости и нарушение пищеварения;</span></li>\r\n<li><span style=\"font-size: 7pt; color: #000000; background-color: transparent; font-style: normal; font-variant-numeric: normal; font-variant-east-asian: normal; vertical-align: baseline; white-space: pre-wrap;\">поглощение лентецом большого количества питательных веществ, витаминов и железа вызывает алиментарное истощение различной степени выраженности, развитие специфической В12-дефицитной (иногда фолиевой) анемии, другие гиповитаминозы, обострение и длительное течение имеющихся хронических заболеваний;</span></li>\r\n<li><span style=\"font-size: 7pt; color: #000000; background-color: transparent; font-style: normal; font-variant-numeric: normal; font-variant-east-asian: normal; vertical-align: baseline; white-space: pre-wrap;\">жизнедеятельность крупного червя в кишечнике человека неизбежно сопровождается выделением значительного количества продуктов переработки, которые, всасываясь из кишечника, попадают в кровь и разносятся по всему организму, вызывая токсико-аллергическую перестройку организма и угнетение иммунного ответа.</span></li>\r\n</ul>",
            "diagnostics": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Диагностика дифиллоботриоза включает в себя:</span></p>\r\n<ul>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">клинический анализ крови &mdash; на начальном этапе может отмечаться умеренно выраженная эозинофилия и относительный лимфоцитоз, общие лейкоциты обычно не изменены; при запущенных и тяжёлых случаях развивается анемия &mdash; снижение уровня гемоглобина и эритроцитов при сохранении высокого цветового показателя; в небольшом количестве обнаруживаются мегалобластные клетки, в эритроцитах &mdash; тельца Жоли (обломки ядер нормобластов), умеренная тромбоцитопения и повышение СОЭ;</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">биохимический анализ крови &mdash; при тяжёлых формах происходит снижение уровня фолиевой кислоты и витамина В12, повышение уровня общего билирубина за счёт непрямой фракции и гипопротеинемия (крайне низкий уровень белка в крови);</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">копроовоскопия &mdash; выявляются многочисленные яйца широкого лентеца в нативном мазке кала;</span></li>\r\n<li><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">прямая визуализация тела червя при отрыве его части и выходе из прямой кишки при дефекации</span></li>\r\n</ul>",
            "treatment": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">К лечению дифиллоботриоза приступают незамедлительно после подтверждения диагноза. Больным назначается полужидкая легкоусвояемая диета, прием препаратов железа, фолиевой кислоты, инъекции цианокобаламина. Специфическая терапия дифиллоботриоза проводится антигельминтными препаратами празиквантел или никлозамид. Возможно использование фитопрепаратов - мужского папоротника или отвара семян тыквы. После проведения дегельминтизации двукратно выполняются контрольные исследования кала; при обнаружении яиц гельминта назначается повторное лечение.</span></p>\r\n<p><span id=\"docs-internal-guid-62b1e904-7fff-ff05-faf2-806b90b8f8c0\">&nbsp;</span></p>",
            "prevention": "<p>&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Профилактика дифиллоботриоза предполагает охрану водоемов от фекальных загрязнений, соблюдение технологии разделки и приготовления рыбы, просвещение населения в очагах распространения гельминтной инвазии. Обязательно проведение паразитологического обследования работников рыбной промышленности и промысловых рыб, поступающих в торговую сеть.</span></p>",
            "clinical_picture": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Течение дифиллоботриоза может быть латентным или клинически выраженным (манифестным). Однако и в том и в другом случае специфическим признаком служит отхождение с каловыми массами крупных фрагментов стробилы гельминта в виде белесых лент. От момента заражения до развития клинических симптомов проходит около 1,5 месяцев. Проявления дифиллоботриоза развиваются постепенно и вначале неспецифичны: снижение аппетита, тошнота, неустойчивый стул, субфебрилитет, утомляемость, похудание. Характерны схваткообразные боли в животе, гиперсаливация.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\">&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Развитие анемии сопровождается головокружением, слабостью, тахикардией, артериальной гипотонией. Патогномоничным признаком дифиллоботриозной В12-дефицитной анемии служит глоссит Хантера, сопровождающийся появлением на языке ярко-красных пятен и болезненных трещин, позднее - блестящего (&laquo;лакированного&raquo;) языка. Воспалительно-дистрофические изменения могут распространяться на десны, слизистую оболочку нёба, щек, глотки, пищевода, вызывая боль и жжение при приеме пищи.</span></p>",
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                    "lead": "Обычно возникает в результате истощающих заболеваний внутренних органов, инфекций, интоксикаций, эмоциональных, умственных и физических перенапряжений",
                    "image": null,
                    "published": 1,
                    "danger": false
                },
                {
                    "id": 2602,
                    "synonyms": [
                        {
                            "name": "Живот болит"
                        },
                        {
                            "name": "Боли в животе"
                        },
                        {
                            "name": "Боль в левой части живота"
                        },
                        {
                            "name": "Боль выше пупка"
                        },
                        {
                            "name": "Боль ниже пупка"
                        },
                        {
                            "name": "Острые боли в животе"
                        },
                        {
                            "name": "Боль в области живота "
                        },
                        {
                            "name": "Боль в области пупка"
                        },
                        {
                            "name": "Боль в правой жасти живота"
                        },
                        {
                            "name": "Пупок тянет"
                        },
                        {
                            "name": "Болит пупок"
                        },
                        {
                            "name": "Болит живот снизу"
                        },
                        {
                            "name": "Болит живот сверху"
                        },
                        {
                            "name": "Боль в правой части живота"
                        },
                        {
                            "name": "Боль в животе у ребенка"
                        },
                        {
                            "name": "У ребенка болит живот"
                        },
                        {
                            "name": "Острая боль в животе"
                        },
                        {
                            "name": "Резь в животе"
                        },
                        {
                            "name": "Боль под ложечкой"
                        },
                        {
                            "name": "Боль после еды"
                        },
                        {
                            "name": "Боль при аппендеците"
                        }
                    ],
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                    ],
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                }
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                "svg_circle": "<svg baseProfile=\"full\" height=\"9525\" version=\"1.1\" width=\"9525\" xmlns=\"http://www.w3.org/2000/svg\" xmlns:ev=\"http://www.w3.org/2001/xml-events\" xmlns:xlink=\"http://www.w3.org/1999/xlink\"><defs /><circle cx=\"4762.5\" cy=\"4762.5\" fill=\"#ffffff\" id=\"svg_1\" r=\"4730\" stroke=\"#2196f3\" stroke-width=\"52.92\" /><path d=\"m5046.45996,686.46c-71,-94 -175,-151 -300,-164c-57,-6 -153,10 -212,20c1,0 0,2 0,2c-6,11 35,-22 -8,11c-19,14 1,0 -26,15c-42,23 -20,7 -52,34c-148,125 -143,175 -116,367c18,134 -37,163 20,311c26,66 29,62 46,142c32,152 107,-49 40,428c-15,103 -79,139 -160,183c-200,109 -373,109 -486,191c-129,94 -173,309 -147,529c44,378 74,157 59,623c-14,458 -37,305 -77,605c-27,207 1,432 13,640c4,61 9,151 0,236c-3,28 -17,61 -19,87c-4,38 -17,64 -23,98c-14,83 -17,167 -14,190c25,48 64,115 78,156c25,8 111,60 133,87c57,31 30,17 112,46l4,-41l-20,-32c-26,3 -81,-52 -105,-108c-7,-16 -5,-36 -5,-64c0,-36 2,-70 8,-87c15,-46 34,-7 46,15c8,15 12,34 11,56c-1,19 -11,41 -12,60c-1,16 8,32 13,43c6,14 33,2 34,0c6,-57 41,-142 40,-237c0,-33 -27,-50 -38,-93c-63,-245 -64,-246 -2,-512c36,-156 91,-326 91,-494c0,-115 -11,-174 16,-286c30,-120 27,-519 3,-775c-59,-639 30,441 80,592c14,65 -1,701 -5,737c-27,234 -69,454 -86,693c-38,552 43,892 123,1408c55,353 -37,516 -55,714c-29,313 191,954 279,1282c29,108 27,84 5,184c-15,66 18,75 -35,137c-69,80 -165,143 -194,180c49,77 400,110 495,42c13,-57 -7,-413 -15,-495c-62,-631 71,-1000 -7,-1420c-38,-206 8,-264 25,-448c8,-86 -3,-166 6,-249c8,-76 21,-146 30,-222c19,-159 37,-298 48,-462c6,-95 -1,-486 29,-540c19,32 76,990 106,1218c11,86 -1,160 6,246c17,202 62,227 28,459c-74,493 19,572 17,951c-2,271 -67,757 -48,955c76,52 134,43 250,37c80,-4 191,-9 250,-59c-11,-45 -24,-43 -57,-68c-141,-106 -111,-124 -170,-167c-6,-63 7,-46 -2,-101c-18,-110 -27,-49 17,-229c80,-322 276,-867 263,-1176c-14,-327 -132,-293 -14,-980c84,-486 119,-830 70,-1338c-21,-218 -69,-416 -81,-638c-12,-228 2,-498 5,-730c66,-183 96,-696 80,-696c0,0 -7,256 -25,464c-12,136 -38,254 -36,260c6,13 7,18 11,34c128,437 2,377 158,976c66,251 35,289 -18,513c-38,158 43,384 43,318c0,-6 39,-55 22,-96c-27,-65 32,-133 34,-114c0,4 18,56 17,110c0,30 -13,59 -21,84c-20,63 -77,93 -79,98c-27,39 -10,16 -19,79c136,-60 149,-60 216,-103c27,-17 31,-42 42,-59c31,-50 49,-85 58,-120c14,-54 -9,-129 -7,-230c-10,-61 -43,-115 -45,-168c-7,-194 12,-391 20,-593c12,-300 -70,-451 -86,-779c-7,-140 -27,-297 7,-429c29,-114 41,-241 53,-289c1,-3 3,-7 3,-9c6,-25 11,-11 -5,-21c8,-369 -39,-473 -360,-572c-426,-132 -429,-195 -454,-489c-17,-201 -10,-48 70,-291c26,-77 83,-137 54,-283c-14,-68 -1,-147 -5,-220c-7,-104 19,-95 -40,-172l4,2z \" fill=\"#ffffff\" fill-opacity=\"0.3\" id=\"svg_2\" stroke=\"#2196f3\" stroke-linecap=\"round\" stroke-linejoin=\"round\" stroke-width=\"52.92\" /><circle cx=\"4714.5\" cy=\"4191\" fill=\"#ff0000\" fill-opacity=\"0.7\" r=\"300\" /><circle cx=\"4726.5\" cy=\"4439\" fill=\"#ff0000\" fill-opacity=\"0.7\" r=\"547\" /><circle cx=\"4726.5\" cy=\"904\" fill=\"#ff0000\" fill-opacity=\"0.7\" r=\"300\" /><circle cx=\"4702.5\" cy=\"3905\" fill=\"#ff0000\" fill-opacity=\"0.7\" r=\"721\" /></svg>"
            },
            "code": "B70",
            "name": "Дифиллоботриоз и спарганоз",
            "icd_name": "Дифиллоботриоз и спарганоз",
            "gender": 0,
            "age_min": 0,
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            "cause": [
                2,
                7
            ],
            "periodicity": 1,
            "slug": "b70_difillobotrioz_i_sparganoz",
            "lead": "гельминтозы из группы цестодозов, возбудителями которых являются ленточные черви",
            "description": "",
            "etiology": "<p>Наиболее распространен широкий лентец, тело которого достигает нескольких метров. Он состоит из множества члеников, каждый из которых имеет самостоятельные половые системы – мужскую и женскую. В стадии половой зрелости гельминты паразитируют в тонкой кишке человека, кошки, собаки, некоторых диких животных. После оплодотворения паразиты начинают продуцировать яйца, которые выделяются с фекалиями. Яйца развиваются в пресноводных водоемах. Личинка, плавая в воде, заглатывается пресноводными рачками (циклопами). В их теле она развивается в личинку 2-й категории (процеркоид). Дальше его развитие происходит в рыбах, которые проглатывают личинку вместе с рачком. Хищные рыбы могут тоже заражаться, поедая более мелких рыб. Личинки локализуются в мышцах и на поверхности внутренних органов. Человек заражается при питании рыбой, содержащей плероцеркоидов. Через 15–20 дней в организме развиваются взрослые особи личинок.</p>\r\n<p> </p>\r\n<p>Широкий лентец оказывает сенсибилизирующее и механическое воздействие, развивается анемия пернициозного типа.</p>",
            "pathogenesis": "",
            "diagnostics": "<p>Диагноз ставят на основании нахождения яиц, а иногда и частей широкого лентеца в фекалиях.</p>",
            "treatment": "",
            "prevention": "<p>Проводится дегельминтизация. При анемии назначаются витамин В12, фолиевая кислота и др.</p>",
            "clinical_picture": "<p>Основными жалобами у больных являются <a href=\"/symptom/slabost/\" title=\"Перейти на страницу симптома Слабость\">слабость</a>, головокружения, <a href=\"/symptom/bol_v_zhivote/\" title=\"Перейти на страницу симптома Боль в животе\">боли в животе</a>, чередование поноса и запора. Периодически с фекалиями отходят части тела лентеца. Постепенно развиваются ахилия желудка, анемия.</p>",
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            "parent": null,
            "block_rubric": 17,
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        },
        {
            "id": 8688,
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                {
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            "etiology": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Гименолепидоз включает в себя гельминтозы, вызываемые ленточными гельминтами - карликовым цепнем, реже - крысиным цепнем, поражающими человека и некоторые виды грызунов (мышей, крыс, хомяков). Карликовый цепень представляет собой мелкую цестоду длиной 1-5 см, шириной 0,5-0,7 мм, имеющую шарообразную головку, шейку и лентовидное тело. На головке гельминта имеются 4 присоски и хоботок с венчиком из 25-30 хитиновых крючков. Тело возбудителя гименолепидоза состоит из нескольких сотен члеников, часть которых, заполненная яйцами, отделяется от гельминта.</span></p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Жизненный цикл карликового цепня (личиночная и взрослая стадии) проходит в организме человека, который служит промежуточным и конечным хозяином паразита. Механизм передачи гименолепидоза - фекально-оральный; заражение человека происходит пероральным путем при заглатывании инвазионных яиц с водой, пищевыми продуктами, немытыми овощами и фруктами, а также через загрязненные руки и предметы обихода.&nbsp;</span></p>\r\n<p><span id=\"docs-internal-guid-b866083c-7fff-7275-2388-6a250a4c653c\">&nbsp;</span></p>",
            "pathogenesis": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">В тонком кишечнике личинка выходит из яйца и внедряется в ворсинки или лимфоидные фолликулы, где протекает тканевая фаза инвазии. Через 6-8 суток шестикрючная личинка (онкосфера) превращается в цистицеркоид, который еще через несколько дней попадает в просвет тонкой кишки, где начинается кишечная фаза развития гельминта. С помощью крючьев и присосок цистицеркоид прикрепляется к слизистой оболочке тонкой кишки и через 2-2,5 недели превращается в половозрелую особь. В некоторых случаях (у ослабленных пациентов с гименолепидозом, детей, лиц с интеркуррентными заболеваниями) может иметь место внутрикишечная аутоинвазия, когда яйца карликового цепня не выделяются во внешнюю среду, а достигают зрелости в кишечнике.</span></p>\r\n<p>&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Возбудители гименолепидоза вызывают механическое повреждение кишечной стенки, приводя к воспалению, развитию патогенной микробной флоры, нарушению ферментативных процессов в кишечнике. Также в патогенезе гименолепидоза играет роль токсическое воздействие на организм продуктов жизнедеятельности гельминтов, иммуносупрессивное влияние, сенсибилизация антигенами карликового цепня, раздражение нервных рецепторов слизистой оболочки тонкого кишечника и другие факторы, вызывающие характерные клинические проявления.</span></p>",
            "diagnostics": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">При объективном обследовании пациентов с гименолепидозом обращает внимание бледность кожных покровов, снижении массы тела, артериальная гипотония. В периферической крови выявляется гипохромная анемия, ускорение СОЭ, умеренная эозинофилия и лейкопения.</span></p>\r\n<p>&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Диагноз гименолепидоза подтверждается путем обнаружения яиц гельминтов или особей карликового или крысиного цепня на разных стадиях развития при исследовании кала. Поскольку выделение яиц происходит циклично, целесообразно проведение анализа трехкратно с интервалами 5 дней, а также сочетание различных методов исследования. Чтобы повысить эффективность паразитологического обследования накануне пациенту назначается прием противогельминтных препаратов, разрушающих членики гельминта и способствующих выходу в просвет кишечника большего количества яиц. </span></p>",
            "treatment": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Особенности развития гельминта и возможность аутоинвазии требуют проведения дегельминтизации, симптоматической терапии и активных профилактических мероприятий. Для специфической терапии гименолепидоза применяются противогельминтные препараты празиквантел (однократно) или никлозамид (в виде 3-х семидневных, 4-х пятидневных или 6-7 двухдневных циклов либо по другой схеме), экстракт мужского папоротника. В перерывах между циклами дегельминтизации проводится общеукрепляющая терапия (поливитамины, препараты кальция). Контрольные анализы кала выполняются через 15 дней и затем ежемесячно в течение полугода после окончания основного курса лечения гименолепидоза. При обнаружении яиц гельминтов проводится повторный курс терапии.</span></p>\r\n<p>&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Во время лечения гименолепидоза важно придерживаться полноценного питания, соблюдать санитарно-гигиенический режим (дезинфицировать предметы ухода, своевременно менять нательное и постельное белье, регулярно проводить личную гигиену). Излеченными считаются пациенты с отрицательными результатами контрольных исследований кала в течение 6 месяцев после окончания курса терапии гименолепидоза.</span></p>",
            "prevention": "<p><span id=\"docs-internal-guid-42df817c-7fff-1cca-a40c-1efb444cea98\"><span style=\"font-size: 7pt; font-family: Verdana; background-color: transparent; font-variant-numeric: normal; font-variant-east-asian: normal; vertical-align: baseline; white-space: pre-wrap;\">Профилактика гименолепидоза требует привития детям гигиенических навыков; уничтожения грызунов, мух, блох, тараканов, вредителей муки. Важно проведение регулярного копрологического обследования детей, работников ДОУ, общепита, пациентов детских, инфекционных, гастроэнтерологических стационаров и других групп населения, снижение заболеваемости энтеробиозом у детей.</span></span></p>",
            "clinical_picture": "<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">Примерно в 30% имеет место бессимптомное, субклиническое течение гименолепидоза; в остальных случаях развиваются манифестные формы гельминтоза с болевым, диспепсическим, астеноневротическим и аллергическим синдромами. Травматизация слизистой стенок кишечника вызывает тупые или резкие боли в животе, которые повторяются в виде приступов ежедневно или через несколько дней. Диспепсические проявления гименолепидоза включают потерю аппетита, изжогу, тошноту, диарею с примесью крови, снижение массы тела. Астеноневротический синдром протекает с головокружениями, общей астенией, раздражительностью, головной болью. Хронический аллергоз при гименолепидозе характеризуется уртикарной сыпью и кожным зудом, вазомоторным ринитом, астматическим бронхитом, отеком Квинке.</span></p>\r\n<p>&nbsp;</p>\r\n<p style=\"line-height: 1.38; margin-top: 0pt; margin-bottom: 0pt;\" dir=\"ltr\"><span style=\"font-size: 6.999999999999999pt; font-family: Verdana; color: #000000; background-color: transparent; font-weight: 400; font-style: normal; font-variant: normal; text-decoration: none; vertical-align: baseline; white-space: pre-wrap;\">В случае интенсивной инвазии могут возникать схваткообразные боли, обмороки, субфебрилитет, нарушения функций печени, миокардиодистрофия. У детей отмечается более тяжелое течение гименолепидоза с истощением, судорожными припадками, гепатомегалией, выраженной анемией и гиповитаминозом. Симптомы гименолепидоза, вызываемого крысиным цепнем, аналогичны клиническим проявлениям инвазии карликовым цепнем. Гименолепидоз может утяжелять течение язвенной болезни желудка и двенадцатиперстной кишки, других сопутствующих заболеваний, а также осложняться мезаденитом.</span></p>",
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            },
            "code": "B72",
            "name": "Дракункулез",
            "icd_name": "Дракункулез",
            "gender": 1,
            "age_min": 0,
            "age_max": 100,
            "cause": [
                2,
                5,
                7
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            "periodicity": 1,
            "slug": "b72_drakunkulez",
            "lead": " Гельминтоз из группы нематодозов, вызываемый самками круглых червей Dracunculus medinensis.",
            "description": "Инфекционное заболевание.",
            "etiology": "<p align=\"justify\">Ришта, или Dracunculus medinensis является круглым гельминтом (нематодой) нитевидной формы. Самки червя достигают длины 30-120 см, самцы - 12-30 мм. Взрослые особи паразитируют в организме человека, реже - животных (хищников, обезьян, собак, кошек). Самки ришты отрождают личинок, которые заглатываются пресноводными рачками-циклопами, выступающими промежуточными хозяевами гельминта. В организме циклопов личинки претерпевают линьку и через 4-14 суток достигают инвазивной стадии.</p>\r\n<p align=\"justify\">Заражение человека дракункулезом происходит при заглатывании воды, содержащей инвазированных циклопов</p>",
            "pathogenesis": "<p align=\"justify\">В ЖКТ окончательного хозяина зрелые личинки (микрофилярии) проникают через кишечную стенку и мигрируют по мягким тканям в межмышечное пространство. Здесь через 3-4 месяцев паразиты достигают половой зрелости. После оплодотворения самцы возбудителя дракункулеза погибают (обызвествляются в мягких тканях или рассасываются), а самка продолжает расти и мигрирует в подкожную клетчатку. Достигнув своим головным концом кожного покрова, самка выделяет специальный секрет, под действием которого на коже человека образуется псевдофурункул, а затем происходит разрыв кожи. Через дефект кожных покровов выпячивается матка гельминта; при соприкосновении с водой она разрывается, и наружу выходит тканевой <a title=\"Перейти на страницу симптома Выпот\" href=\"../../../symptom/vypot/\">экссудат</a>, содержащий мелкие личинки паразита. Одна самка выделяет в водную среду до 3 млн. личинок, после чего погибает.</p>\r\n<full></full>\r\n<p align=\"justify\">Длительность паразитирования ришты в теле человека может достигать 18 месяцев. Попав в воду, личинки заглатываются циклопами – так начинается новый цикл развития гельминта. Дракункулезом чаще болеют мужчины различных возрастных групп; женщины и дети младшего возраста заражаются реже. Обычно у человека развивается инвазия одним гельминтом, однако описаны случаи множественного дракункулеза с одновременным паразитированием 17 и даже 50 гельминтов. Заболеваемости дракункулезом способствует антисанитария, отсутствие водопровода и канализации, употребление сырой воды из открытых водоисточников. Загрязнение водоемов личинками гельминта может происходить при заборе воды, купании, ритуальных омовениях. Патогенные эффекты при дракункулезе обусловлены сенсибилизацией организма продуктами жизнедеятельности паразита, механическим травмированием тканей и наслоением вторичной инфекции.</p>",
            "diagnostics": "<p>В эндемичных районах заподозрить дракункулез у больных позволяют характерные кожные проявления, пальпация под кожей шнуровидного образования, обнаружение на месте разрыва волдыря переднего конца ришты. При помещении гельминта в воду происходит выделение личинок, которых можно рассмотреть при микроскопии. Также обнаружить личинки ришты можно в отделяемом из вскрывшихся пузырей. Обызвествленные паразиты могут выявляться при рентгенологическом исследовании тканей. С целью подтверждения диагноза дракункулеза возможно проведение внутрикожной аллергической пробы, серологических исследований (РНИФ). Клинические проявления дракункулеза необходимо отличать от фурункула, абсцесса, флегмоны.</p>",
            "treatment": "",
            "prevention": "<p>После появления гельминта из-под кожи извлечь его можно путем наматывания на марлевый валик или палочку. Во избежание обрыва гельминта делать это следует осторожно и постепенно, прекращая извлечение при появлении ощущения сопротивления. Обычно за один прием удается извлечь 5-10 см нитевидного паразита - в этом случае процедуру повторяют на следующий день. При обрыве гельминта производится его хирургическое удаление. Предварительное проведение курса противогельминтной терапии при дракункулезе препаратами метронидазол, ниридазол, амбильгар, минтезол облегчает извлечение погибшего паразита. Также показано назначение антигистаминных, кортикостероидных, а при бактериальных осложнениях - противомикробных препаратов. Заболевшим дракункулезом вводится противостолбнячный анатоксин.</p>",
            "clinical_picture": "<p align=\"justify\">бычно дракункулез заявляет о себе развитием аллергических реакций (крапивницы, кожного зуда, приступов удушья, одутловатости лица), головокружения, тошноты, рвоты, лихорадки. Отмечаются локальные отеки мягких тканей в месте расположения гельминта, припухание суставов, регионарный лимфаденит.</p>\r\n<full></full>\r\n<p align=\"justify\">Через 8-10 дней после развития общей симптоматики в месте будущего выхода самки ришты на поверхности кожи появляется <a title=\"Перейти на страницу симптома Покраснение кожи\" href=\"../../../symptom/pokrasnenie_kozhi/\">эритема</a> и плотный инфильтрат, из которого затем формируется волдырь. Этот пузырь заполнен желтоватой жидкостью, содержащей лейкоциты, эозинофилы и личинки гельминта. Через несколько дней пузырь вскрывается с отторжением некротических масс и образованием язвы. Из-под кожи появляется головной конец самки паразита. Эти процессы сопровождаются нестерпимым зудом и жгучей болью, которые немного стихают после контакта с холодной водой. Именно этот факт заставляет больных дракункулезом искать спасения от мучительных симптомов, окунаясь в водоемы. При погружении пораженных частей тела в воду происходит массовый выход личинок и обсеменение водоема.</p>\r\n<p align=\"justify\">Излюбленная локализация ришт – подкожная клетчатка нижних конечностей (голеней, лодыжек), реже - верхних конечностей, живота, спины, ягодиц, мошонки, молочных желез и других участков тела. При неосложненной форме дракункулеза язвенный дефект кожи быстро заживает. Если в организме паразитирует единственный гельминт, то через 4-6 недель клинические проявления дракункулеза стихают, и заболевание заканчивается выздоровлением.</p>",
            "image": null,
            "image_alt": null,
            "standard_type": 1,
            "danger": 6,
            "published": 1,
            "parent": null,
            "block_rubric": 17,
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        },
        {
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            },
            "code": "B73",
            "name": "Онхоцеркоз",
            "icd_name": "Онхоцеркоз",
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                2,
                6
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            "lead": "Хронически протекающий филяриатоз, характеризующийся преимущественным поражением кожи, подкожной клетчатки и глаз.",
            "description": "Онхоцеркоз, или «речная слепота», является паразитарной болезнью, вызываемой филярией Onchocerca volvulus., характеризуется образованием подкожных узлов, поражением кожи и глаз.",
            "etiology": "<p>Возбудитель заболевания — Onchocerca volvulus. Гельминт имеет нитевидное, утончающееся к концам тело, длина около 350—500 мм у самок и 19-42 мм у самцов.</p>\r\n<p>Хозяином возбудителя является только человек, а переносчиком — самки мошек Simulium damnosum. Онхоцеркоз часто называют речной слепотой, поскольку переносчики обитают по берегам рек. Взрослые черви поселяются в лимфоузлах человека, где самки производят на свет множество личинок, называемых микрофиляриями. Эти личинки далее мигрируют под эпидермис кожи, откуда могут вновь попасть в организм мошек и, таким образом, продолжить цикл развития паразитов. Часть микрофилярий попадает в глаза человека, проникая во все ткани зрительного органа. В глазном яблоке они вызывают воспаление, кровотечения и другие осложнения, ведущие в конечном итоге к потере зрения.</p>",
            "pathogenesis": "<p>Микрофилярии, паразитируя в коже человека, вызывают ее резкие изменения: утолщение эпидермиса, депигментацию, изъязвления. В глазных яблоках микрофилярии вызывают <a href=\"/symptom/vospalenie/\" title=\"Перейти на страницу симптома воспаление\">воспаление</a>, на конъюнктиве образуются узелки, происходят атрофия пигмента радужной оболочки и другие изменения, которые обусловливают тяжелые расстройства зрения вплоть до слепоты на один или оба глаза.</p>",
            "diagnostics": "<p>К методам диагностики заболевания относят обнаружение онхоцерком в иссеченных подкожных узлах или микрофилярий в тонких срезах или биоптатах кожи. Кроме этого, микрофилярии могут обнаруживаться в роговице, передней и задней камере глаза при проведении биомикроскопии.</p>\r\n<p>В диагностике онхоцеркоза применяются серологические методы (РСК, ИФА, РПГА), внутрикожная аллергическая проба.</p>\r\n<p>Лабораторно-инструментальная диагностика позволяет дифференцировать онхоцеркоз от других филяриатозов, грибковых заболеваний кожи, лепры, гиповитаминозов А и В.</p>",
            "treatment": "<p>При расположении онхоцерком на голове рекомендуется хирургическое лечение, поскольку половозрелые паразиты находятся в непосредственной близости от глаз.</p>\r\n<p>Для лечения онхоцеркоза применяются лекарственные средства с микрофилярицидным (диэтилкарбамазин, ивермектин) и макрофилярицидным (сурамин, тримеларсан) действием. При выраженных аллергических реакциях назначаются антигистаминные препараты, кортикостероиды, плазмаферез.</p>",
            "prevention": "<p>Для уменьшения интенсивности очагов онхоцеркоза применяют ларвициды уничтожающие личинок мошек в местах их выплода. Обработка воды инсектицидами в течение 20-30 мин приводит к гибели личинок на протяжении более 200 км вниз по течению от места их внесения. Обработки повторяются каждые 7 дней. Индивидуальную защиту обеспечивает одежда, которую следует обрабатывать репеллентами.</p>\r\n<p>При необходимости проживания в эндемичных очагах следует избегать пребывания вне населенного пункта или вне жилых помещений в ранние утренние и вечерние часы. Онхоцеркоз можно предотвратить с помощью химиопрофилактики ивермектином 1 раз в 6 месяцев.</p>",
            "clinical_picture": "<p>Длительность инкубационного периода - около 1 года. Заболевание начинается с общего недомогания и лихорадки. Отмечают сухость и шелушение кожи, зудящую узелковую сыпь. Иногда узелки трансформируются в гнойники с образованием язв. При значительной пораженности микрофиляриями кожа приобретает вид лимонной корки, шагреневой или слоновой кожи. С течением времени появляются участки депигментации кожи. </p>\r\n<p>Характерный признак онхоцеркоза — наличие под кожей плотных фиброзных узлов размерами до 7 см. Узлы могут локализоваться как в тазобедренной области, так и около суставов, в области лопаток и редко на голове. </p>\r\n<p>Нередко возникающий дерматит весьма напоминает рожистое воспаление кожи. При этом кожа на пораженных участках становится отечной, темно-красного цвета, температура тела достигает 39-40°, появляется отек губ и ушных раковин. Обострения дерматита длительностью от нескольких дней до нескольких недель постепенно приводят к тому, что кожа на пораженных местах утолщается, становится отечной, отмечается увеличение ушных раковин, они загибаются кпереди.</p>\r\n<p>Поражение лимфоузлов. Часто наблюдается легкое или умеренное увеличение лимфоузлов - особенно поверхностных и глубоких паховых. Под действием силы тяжести в паху образуются свисающие кожные карманы, в которые смещаются увеличенные лимфоузлы. Это повышает риск паховой и бедренной грыж. </p>\r\n<p>Серьёзность прогноза определяет поражение глаз. Паразиты проникают на роговицу с кожи и конъюнктивы, вследствие чего развивается точечный кератит (воспаление роговицы глаза) вокруг гибнущих микрофилярий. Если инвазия продолжается на протяжении многих лет, развивается склерозирующий кератит и иридоциклит, что и ведёт к потере зрения. Кроме того, микрофилярии способны проникать в сетчатку и по сосудам, кровоснабжающим веки. Воспаление сетчатки в области выхода зрительного нерва также способно серьёзно нарушать зрение.</p>\r\n<p>При тяжелой инвазии иногда развивается кахексия с утратой жировой ткани и атрофией мышц. </p>",
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